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甲状旁腺素

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The increased serum phosphate tends to drive serum calcium down, which in turn drives the parathyroids to secrete more parathormone .

血清中磷酸盐含量增高会使血清中钙盐含量降低,促使甲状旁腺分泌更多的甲状旁腺素

Its features consist of biochemical disturbances - hypocalcemia, hyperphosphoremia and hyperparathyroidiemia and lack of response to the parathormone of target organs. The patients usually manifest tetany seizures and parathyroid gland hypertrophy accompanied by hereditary osteodystrophy.

其临床特点是手足搐搦,低血钙、高血磷、高甲状旁腺素血症,周围靶组织对PTH生物活性不敏感,甲状旁腺可以增生肥大,常伴先天遗传性骨发育畸形。

Human blood calcium level is stable, because of parathyroid hormone and calcitonin regulate.

人体血液中钙水平是稳定的,因为有甲状旁腺素和降钙素进行调节。

The pleiotropic ef-fects of PTHrP/PTH and the receptor, which become the focuses of research on...

下面就甲状旁腺素相关蛋白和甲状旁腺素及其Ⅰ型受体在髁突发育中的研究进展作一综述。

AOPA, by "multiple relevant convergence reaction", adjusts and affects systematically the endocrinal function of bone metabolism,improves the levels of estrogen and CT/PTH, reverses the declining tendency of pituitary-thyroid axis function and improves thyroid function so as to weaken osteocyte in the bony absorption, enhance osteoblast in osteogenesis and increase bone density.

AOAP可以通过&多位点相关集合效应&综合调控影响骨代谢的内分泌功能,提高雌激素、降钙素/甲状旁腺素水平,扭转垂体-甲状腺轴功能减退的趋势,改善甲状腺的功能,使破骨细胞的骨吸收减弱,成骨细胞的骨形成增强,增加骨密度。

METHODS: Osteoporosis model was prepared with injection of dexamethasonc. Antagonism osteoporosis acupoint paste on acupoints and non-neridian places was administrated to compare with oral recipe for kidney tonification in the treatment of osteoporosis. It took the changes in serum estrogen, bone density, calcitonin/parathyroid hormone, thyrotropic hormone, free T3 and free T4 as the measuring indexes. Immunocyto-chemistry stain and morphological measurement of TSH cell,follicular adenoma of thyroid gland and thyroid globulin were applied in pituitary gland and thyroid gland to observe the therapeutic results.

采用注射地塞米松建立骨质疏松模型,用抗骨松穴位贴剂(antagonism osteoporosis acupoint paste,AOAP)外贴穴位、非经非穴位、并与口服补肾方药比较治疗骨质疏松,以血清雌激素、骨密度、降钙素/甲状旁腺素、促甲状腺激素、游离T3、游离T4的变化为衡量指标,取垂体、甲状腺作促甲状腺激素细胞、C细胞和Tg等免疫细胞化学染色,并形态计量,观察其治疗效果。

In this setting the parathyroid is unable to secrete hormone, and renal and skeletal responses to PTH are blunted .

在这种情况下,甲状旁腺无法分泌激素,同时肾脏和骨骼对甲状旁腺素的反应也愈加迟钝。

To overcome this potential problem in some cases of hyperparathyroidism, the assay must be carried out in multiple dilutions of the serum sample to fully assess the PTH level.

为了克服这个问题,在部分甲状旁腺功能亢进的病人中,血样必须先经过稀释,才能测定甲状旁腺素

Hyperphosphatemia stimulates parathyroid gland hypertrophy and stimulates increased production and secretion of PTH.

高磷血症刺激甲状旁腺增生,刺激提高生产和甲状旁腺素分泌。

Hypocalcemia stimulates the parathyroid gland to excrete PTH, a process termed secondary hyperparathyroidism.

低血钙刺激甲状旁腺分泌甲状旁腺素的,这个过程被称为继发甲状旁腺功能亢进。

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