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Connective tissue disease is a group and the human immune response of multiple organ of the multi-system inflammatory connective tissue disease, the main lesion is myxedema, fiber-like degeneration and necrosis vasculitis.

结缔组织病(connective tissue disease, CTD)是一组与免疫反应有关的人体多器官多系统结缔组织的炎症性疾病,其主要病变为粘液性水肿、纤维蛋白样变性及坏死性血管炎。

The patient of glucoside of strong to applying at the same time heart and diuretic should notice compensatory sylvite. 1.2 cause or aggravating infection candy is coriaceous of hormone fight phlogistic action, its are main the mechanism is to restrain inflammation to promote factor, restrain antigen antibody reaction, reduce blood capillary to connect appear a gender, reduce the toxin harm to airframe, relieve the symptom that inflammation reacts thereby.

对同时应用强心甙和利尿剂的患者应注重补充钾盐。1.2诱发或加重感染糖皮质激素的抗炎功能,其主要机制是抑制炎症促进因子、抑制抗原抗体反应、降低毛细血管通透性,减轻毒素对机体的损害,从而减轻炎症反应的症状。

PSC is a kind of rare and unknown disease due to stenosis cholangitis.Its main symptoms is diffuse fibrotic incrassation and duct stenosis in intrahepatic and extrahepatic bile duct system..

原发性硬化性胆管炎原发性硬化性胆管炎(primary sclerosing cholangitisPSC)是一种少见的、原因未明的胆管炎症性狭窄性疾病。

Thus, a western dietary pattern may be highly relevant to activation of the innate immune response, which drives a neutrophilic pattern of airway inflammation, which is increasingly recognised in asthma.

因此,西方饮食习惯可能与先天免疫应答的激活高度相关,后者会导致气道中性粒细胞性的炎症反应,这种炎症反应在哮喘中的作用逐渐得到认识。

Many years ago, it have been noticed that, not all injurise of heart cell were directly proportional to the inflammatary reaction, and this kind of noninflammatory damage may be apoptosis. Kawano has verified first time in 1994 that, the onset and development of VMC accompanied the apoptosis, and the apoptosis probably are significant mechanism from VMC to the dilated cardiomyopathy.

早有人注意到,VMC时心肌损伤并不都与炎症反应成正比,其存在的非炎症性损伤很可能是细胞凋亡。1994年Kawano等首次证实心肌炎伴有心肌细胞凋亡,后者可能是心肌炎发展成扩张型心肌病的重要机制。

The stimulations of dexamethasone, lipopolysaccharide, erythromycin and hypertonic saline can significantly change the AQP1 expression in pleura, which indicate that AQP1 may contribute to the accumulation and clearance of pleuritic fluids. Aquaporin-1;Pleura;Pleural effusion;Dexamethasone;Lipopolysaceharide;Erythromycin;Hypertonic saline

地塞米松、内毒素、红霉素和高渗盐水刺激下胸膜组织AQP1表达的改变,提示存在炎症因子和渗透梯度下调控AQP1的途径,胸膜组织上AQP1可能参与了炎症性胸腔积液的分泌和吸收。

Thus, in polyarthritic rat, a model of chronic inflammatory pain, we demonstrated that herpes simplex irus ector mediated oerexpression of proenkephalin A in primary sensory neurons at the lumbar leel elicited both antihyperalgesic and anti-inflammatory actiities.

在一个慢性炎症性疼痛模型-多关节炎鼠上,我们证明单纯疱疹病毒媒介介导的前脑啡肽在腰水平初级神经原的过度表达引起了抗痛觉高敏和抗炎症活动。

The pathogenesis of asthma can be divided into three stages in immunology. The initial stage is sensibilization, which is closely associated with the activation of T cells. B7/CD28 and CD69 also play a significant role in this process. The second stage is chronic allergic inflammation. The immune dysfunction resulting from the disturbance of TH1/TH2 is a major cause of chronic allergic inflammation, and dominates asthma attack. And the third stage is airway remodeling due to chronic inflammation. Airway remodeling is one of the major causes of refractory asthma, which also contributes to the death from asthma.

哮喘的发病从免疫学角度可以分为三期:初期为致敏阶段,与T细胞激活关系密切,B7/CD28和CD69在这一过程也扮演重要角色;第二期为慢性过敏性炎症阶段,TH1/TH2失衡导致免疫功能紊乱是此阶段的主要原因,在哮喘发作过程中起主要作用;第三期为慢性炎症引起的气道结构重构阶段,是难治性支气管哮喘的重要发病和死亡原因。

Crohn's Disease is a form of Inflammatory Bowel Disease which occurs when the immune system overreacts to an agent of the digestive system, sends excessive white blood cells to the area, and causes inflammation as a result.

克罗恩病是一种形式的炎症性肠病发生时,免疫系统overreacts以代理人的消化系统,发送过多的白血细胞在该地区,并导致炎症的结果。

Especially compared with Western medicine,there are obvious advantages in reducing the side effect,and preventing from recuring.

本文概述了环境香烟烟雾对人体的危害,阐述了现代医学对香烟烟雾引起气道炎症的作用机制的认识,并对穴位敷贴疗法机理及其治疗气道炎症性疾病的临床研究和机理研究作了全面地总结。

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