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炎症

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Results: There were 14 cases of solitary bronchioloalveolar carcinoma, the distance of pleura to the centre of pulmonary node was smaller than 3.1cm, node diameter from 1.4cm to 3.5cm, the CT morphologic features of nodes were margin slick 2 cases, irregular margin 6 cases, short barb 5 cases, pleural retraction 11 cases, bronchogram 6 cases, vacuole 12 cases. 5 cases of consolidation bronchioloalveolar carcinoma, the main features were multisegment or lobes inflammatory consolidation, bronchogram phase 4 cases, and honeycomb 3 cases. 6 cases of diffused bronchioloalveolar carcinoma, the main features were widespread diffusion nodes, inequality of size, anisodistribute.

结果:孤立结节型14例,结节中心距胸膜均小于3.1cm,病灶直径为1.4~3.5cm,CT表现有边缘光滑2例,呈分叶征6例,短毛刺征5例,胸膜凹陷征11例,含气支气管征6例,空泡征12例;炎症型或实变型5例,病变呈多个肺段或肺叶的炎症样实变,见支气管气相4例,以及蜂房状含气腔3例;弥漫结节型6例,弥漫分布粟粒状与结节状致密影,大小不等,分布不均。

However, higher concentration of CRP may increase the expression of protein and mRNA of inflammatory factor in endothelial cells; 2 Higher concentration of CRP may participate in the development of inflammatory process in endothelium by the mechanism that CRP induce cells apoptosis and activate signal pathway involved in cell proliferation. 3 CRP is the member of pentraxins family, in which two faces of pentamer form a special bi-phase structure: one face is ligand recognition phase; the other face is effector phase.

总结: 1CRP是一种保守蛋白,在正常生理水平下对内皮细胞没有明显的诱导炎症发生的作用,仅在高浓度条件下有可能诱导细胞炎症相关蛋白及mRNA的表达; 2高于生理水平的CRP对内皮细胞的致炎作用主要通过诱导细胞凋亡及激活细胞增殖相关蛋白信号通路的表达; 3CRP是一种双面五聚体蛋白,结构具有特殊的二相性,能够分别结合内皮细胞和E-LDL。

After 3 years, most patients end up have an almost normal liver in terms of inflammation.

接受长期治疗的患者坏死性炎症有显著改善,3年后,大部分患者曾存在坏死性炎症的肝脏几乎都恢复正常了。

Results 甀ntrahepatic vascular diseases, liver tissues diseases and the expressions of VEGF were different in different time of experiment induced by CC14. In earlier periods (2 to Swk) of rats? liver tissues induced by CC14, the inflammatory was significant. Fatting degeneration was seen in hepatocytes around central veins and part of small vesells.

早、中期(2~5wk),肝组织炎症反应显著,中央静脉及部分小血管周围肝细胞脂肪变,VEGF阳性肝细胞主要分布在脂肪变区域;晚期(8~12wk)则肝组织炎症反应轻微,但肝细胞病变显著,血管破坏、血管及成肌纤维细胞增生更趋明显,并见有局部纤维瘢痕形成,假小叶内肝组织肝窦扩张或毛细血管化,其VEGF示阳性。

Whereas restricted inflammation is beneficial, excessive or persistent inflammation incites tissue destruction and disease.

适度的炎症反应对人体是有益的,而过度的或持续的炎症反应却会引起组织损伤和诱发疾病。

The NPC tissue was all EBV-positive except for one sample, which was identified as keratinizing carcinoma, but the control samples were all negative for EBV infection, which was correlated with CD25 expression.

对照组选取鼻咽慢性炎症组织。结果 CD25淋巴细胞在鼻咽癌组织中比在鼻咽慢性炎症组织中的表达明显升高,差异具有显著性意义(P.001),且在未分化癌中表达高于角化型癌和非角化型癌(P.05)。原位杂交显示EB病毒与CD25淋巴细胞高表达有关。

Crohn's Disease is a form of Inflammatory Bowel Disease which occurs when the immune system overreacts to an agent of the digestive system, sends excessive white blood cells to the area, and causes inflammation as a result.

克罗恩病是一种形式的炎症性肠病发生时,免疫系统overreacts以代理人的消化系统,发送过多的白血细胞在该地区,并导致炎症的结果。

Much progress has been made in recent years through studying septic shock,however brain injury is rarely involved.This study takes the models of endotoxin-shock rats as research object to simulate septic shock.The study aims are to explore anomalous signal transduction in brain injury caused by septic shock and the effect of Sini Decoction in the process;define the molecular and cellular mechanisms of reviving yang for resuscitation method used to prevent and treat brain injury in septic shock and establish the scientific foundation for modernizing traditional Chinese medicine.

研究目的:1。明确内毒素休克后脑损伤组织中炎症介质水平,中性粒细胞和脑血管内皮细胞等细胞表面黏附分子表达情况,脑血管平滑肌细胞、神经胶质细胞和神经细胞对细胞外炎症介质刺激产生的功能代谢变化以及结构改变等三者之间的规律性关系。2。

Aim To investigate the effects of carbachol on inflammatory cytokine releases from rat peritoneal macrophages induced by lipopolysaccharide and its receptor.

目的在体外脂多糖(lipopolysaccharide,LPS)刺激大鼠腹腔巨噬细胞炎症模型中观察拟胆碱药卡巴胆碱对炎症细胞因子释放的影响,并研究其受体途径。

These features of oxidative stress, inflammation, and problems with detoxification invite us to borrow knowledge across fences that have previously divided the landscape of heart disease, cancer, dementias, autoimmune, allergic, and other chronic inflammatory problems.

这些功能的氧化应激,炎症,和问题,戒毒邀请我们借用知识,栅栏了先前划分的风景心脏病,癌症,痴呆,自身免疫性,过敏和其他慢性炎症的问题。

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