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炎症

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Results In diabetes anabrosis raw surface, collagenoblast and inflammatory cell appeared lately, inflammation endurance was long.

结果 糖尿病溃疡创面成纤维细胞、炎症细胞出现晚;炎症持续时间长,各种细胞器不丰富,且功能不活跃。

RhA can effectively prevent the information of postoperative ankylenteron by inhibiting the expression of inflammatory cytokines and reducing the inflammatory response.

大黄酸精氨酸可通过抑制炎症因子的过度表达,减轻炎症反应而有效预防术后肠粘连的形成。

Aside from pancreatic enzymes activiation and autodigestive process, recent investigations have established that the upregulation of inflammatory mediator are believed to be the critical steps in the progression of mild pancreatitis to severe pancreatitis. Tumor necrosis factor-, ThyomboxaneA2 ,ProstaglandinsI2 are all important inflammatory mediator. TNF-( is one of cytokines. It is important to the over-production of ICAM-1,VCAM-1, TXA2 andPGI2. The ICAM-1 and VCAM-1 mediate both leukocytes adhesion and migration through the endothelium into tissues to connect with injuried target cells. The TXA2 and PGI2 are the production of arachidonic acid. They are responsible for the tissue ischaemia.

肿瘤坏死因子(Tumor necrosis factor-, TNF-、血栓素A2(ThyomboxaneA2,TXA2),前列环素(ProstaglandinsI2,PGI2)均是炎症反应中重要的炎性介质,其中肿瘤坏死因子-是非常重要的细胞因子之一,它由活化的单核细胞产生,在细胞间黏附分子(Intercellular adhesion molecule-1,ICAM-1)及血管细胞黏附分子(Vascular cell adhesion molecule-1,VCAM-1的表达、血栓素A2及前列环素过量生成方面起着重要的作用,前两者是介导白细胞与内皮细胞黏附,迁移于组织并与受损的靶细胞相结合的主要媒介,是炎症反应中的重要介质,后两者是花生四烯酸的代谢产物,亦是重要的炎性介质,参与构成血液循环障碍。

Objective: Bletilla Striata polysaccharide was used to stimulate the whole blood of the ICR mice. The mRNA and protein levels of all the inflammatory factors and coagulation factors in the whole blood were determined at different time intervals in order to investigate the molecular mechanism of BSP on the pathway of inflammation and coagulation. Methods:The whole blood was obtained from the ICR mice and stimulated by BSP.

目的:用白芨多糖刺激全血,检测不同刺激时间全血中各种炎症因子和凝血因子在mRNA水平和蛋白质水平的表达量,探讨白芨多糖作用于炎症和凝血通路的分子机制。

Results showed:(1) The airway responsiveness of smoke control group was higher than that of normal control group, the airway responsiveness of smoke + exercise group was lower than that of smoke control group;(2) Plasma level of cortisol determined immediately after exercise was higher than that determined before exercise, no significant difference occurs between that determined next day morning after all exercise finished in contrast to that determined before exercise;(3) HE staining showed, there was severe chronic pulmonary inflammatory response in smoke control group, which was reduced in the smoke + exercise group;(4) chronic cigarette smoke downregulated the protein and mRNA expression of BKca in smooth muscles of bronchi and bronchioli, exercise increased the mRNA expression of BKca;(5) chronic cigarette smoke downregulated the protein and mRNA expression of Kv1. 5 in smooth muscles of bronchi and bronchioli, exercise increased both the protein and mRNA expression of Kv1. 5 in contrast to smoke control group in bronchioli and did not effect the expression of Kv1. 5 in bronchi;(6) chronic cigarette smoke induced upregulation of FIZZ1/RELMα in bronchial smooth muscle cells, exercise decreased these effect.

结果:(1)吸烟对照组的气道反应性明显增高,吸烟加运动应激可明显降低大鼠的气道反应性;(2)吸烟运动组运动后血浆皮质醇浓度明显高于运动前,但全部运动结束后次日晨测定的血浆皮质醇浓度与运动前无明显差异;(3)HE染色显示,吸烟对照组肺组织出现明显的慢性炎症反应,吸烟加运动应激组比吸烟对照组炎症反应轻;(4)慢性吸烟可降低大鼠大气道和小气道BKca mRNA和蛋白表达,而吸烟加运动应激可使BKca mRNA表达上调,但对小气道的蛋白表达无影响;(5)慢性吸烟可降低大鼠大气道和小气道Kv1.5蛋白和mRNA表达,在小气道吸烟加运动应激可减轻这种作用,大气道则无作用;(6)吸烟使支气管平滑肌的FIZZ1/RELMα蛋白表达明显增强,而吸烟加运动组的RELMα蛋白及mRNA均明显减弱。

The present agents may be administered in an anti-bronchoconstriction and/or anti-inflammation effective amount to alleviate the bronchoconstriction and inflammation afflicting a subject.

本发明药剂可以以抗支气管狭窄和/或抗炎症的有效量给药,以减轻个体的支气管狭窄和炎症

CT display: In the small intestine:The boiler of the small intestine was thicker and the appearance was broadened;In the sinonasal cavity:The appearance of the middle and inferior turbinates were enlarged and the infiltrateive bone destruction,the affected paranasal sinuses were filled with soft tissues and sinus walls demonstrated bone infiltration; In the lungs:The repeatedly pneumonia happened in the same part for a long time and had a slowly speeding and it had normal bronchorrhagia.

小肠黏膜相关淋巴瘤表现为肠壁增厚、肠腔扩大;鼻腔黏膜相关淋巴瘤表现为鼻甲增大、鼻甲骨质虫蚀状破坏,鼻窦骨质破坏以及窦腔内软组织肿块;肺黏膜相关淋巴瘤表现为同一部位不易消散的、缓慢进展的节段性炎症炎症区域支气管气像正常。

These include inflammatory arthritides such as rheumatoid arthritis, ankylosing spondylitis and reactive arthritis,99 101 synovial impingement,meniscoid entrapment, chondromalacia facetae, pseudogout,synovial inflammation, villonodular synovitis, and acute and chronic infection.102 106 Intrafacetal synovial cysts can be a source of pain because of distension and pressure on adjacent pain-generating structures, calcification,and asymmetrical facet hypertrophy.107 110 In a retrospective review of MRI scans in 303 consecutive patients with LBP, Doyle and Merrilees111 found that9.5% had facet joint synovial cysts, the large majority of which were located posteriorly.

包括炎症性关节炎如类风湿性关节炎,强直性脊柱炎及反应性关节炎等,还有滑液撞击,关节软骨版卡压,关节面软骨软化,假性痛风,滑液炎症,绒毛结节性滑膜炎和急慢性感染。小关节内滑囊囊肿可由于拉伸和压缩临近的疼痛发生结构,骨化及不对称关节增生等原因引起疼痛。在一项303例连续患者的MRI研究中,Doyle 和 Merrilees发现其中9.5%存在小关节囊肿,大部分发生于关节囊后部。

Anterior uveitis - inflammation of the iris or inflammation of the iris and the ciliary body.

前葡萄膜炎-炎症的虹膜或炎症的虹膜和睫状体。

The patient of glucoside of strong to applying at the same time heart and diuretic should notice compensatory sylvite. 1.2 cause or aggravating infection candy is coriaceous of hormone fight phlogistic action, its are main the mechanism is to restrain inflammation to promote factor, restrain antigen antibody reaction, reduce blood capillary to connect appear a gender, reduce the toxin harm to airframe, relieve the symptom that inflammation reacts thereby.

对同时应用强心甙和利尿剂的患者应注重补充钾盐。1.2诱发或加重感染糖皮质激素的抗炎功能,其主要机制是抑制炎症促进因子、抑制抗原抗体反应、降低毛细血管通透性,减轻毒素对机体的损害,从而减轻炎症反应的症状。

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