炎性的

TNF α protein and mRNA strongly expressed in the area of bronchiolar epithelium and the inflammatory cells in the lymph follicle near the bronchia and the alveolus cavity and pulmonary interstise in the model group.

此外，慢支大鼠TNFα蛋白及其mRNA在支气管周围的淋巴滤泡中的炎性细胞和肺泡间质浸润的炎性细胞中有强阳性表达，而经艾麻口服液治疗后，其表达减弱。

Ravan Napas can reduce the expression of ICAM 1 and HO 1 on broncho epithelial cells and thus inhibits the aggregation of the inflammatory cells and the release of the inflammatory media, as a result leads to less inflammation of bronchia. Such inhibitory way might be one of the possible mechanisms of Ravan Napas in treating asthma.

纳气平喘颗粒通过调节支气管上皮细胞ICAM 1和HO 1的表达，可抑制炎性细胞在气道内聚集及其炎性介质的释放，减少气道变应性炎症，这可能是纳气平喘颗粒治疗哮喘病的部分机制。

EGCG was reported to inhibit leukocyte migration in response to chemokines such as IL-8, CINC and MIP-2, to trap reactive oxygen species, to inhibit neutrophil degranulation and inhibit inflammatory mediator release.

EGCG的抗炎作用涉及多个环节，包括抑制趋化因子如白介素-8(IL-8、细胞因子诱导的中性粒细胞趋化因子-1(CINC-1)、巨噬细胞炎性蛋白-2(MIP-2)等诱导的白细胞的定向迁移、清除氧自由基、抑制嗜中性粒细胞脱颗粒及炎性介质的释放等。

The reasons of RA focus on disbalance of immunity net, the effect of so many cytokines and MMPs , and the tumor-like proliferation of fibroblast-like synovicytes in joints, though the cause of developing disease is unknow.

RA的发病过程中，免疫网络失衡，加上成纤维样滑膜细胞(fibroblast-like synoviocytes，FLS)的类肿瘤样生长及所分泌的大量促炎性细胞因子、趋化因子和金属蛋白酶，是导致关节内炎性细胞聚集，滑膜细胞增生和向周围组织侵蚀及基质降解和破坏的主要因素。

Clinical diagnostic considerations ranged from benign entities such as rhabdomyoma, intramuscular lipoma, fibromatosis, myositis ossificans, proliferative myositis, inflammatory myofibroblastic tumor, and inflammatory myopathy to malignant entities such as rhabdomyosarcoma, leiomyosarcoma, liposarcoma, and lymphoma.

这些病例的临床考虑多样，有良性的横纹肌瘤、肌内脂肪瘤、纤维瘤病、骨化性肌炎、增生性肌炎、炎性肌纤维母细胞瘤和炎性肌病，也有恶性的横纹肌肉瘤、平滑肌肉瘤、脂肪肉瘤和淋巴瘤。

A single systemic injection of resiniferatoxin , an ultrapotent analog of capsaicin, produced a long-lasting, reversible blockade of adjuvant-induced thermal hyperalgesia and reduction of early edema in the adult rat. Depletion of mast cell granules in peripheral connective tissues with secretagogue compound 48/80 also reduced the hyperalgesia and early edema to some extent. Co-pretreatment with RTX and compound48/80 had the similar effects as that of RTX treated alone. After unilateral surgical sympathectomy, the thermal hyperalgasia and edema were both significantly enhanced.

成年鼠一次性注射辣椒素类似物resiniferatoxin后，长时程、可逆性阻断了CFA所致热痛过敏的发展并削弱了早期炎性水肿；用促脱颗粒剂compound48／80慢性耗竭外周结缔组织肥大细胞内分泌颗粒后，也部分削弱了热痛过敏及早期水肿的形成；RTX与compound48／80共同处理对热痛过敏及水肿有与RTX单独处理相近程度及时程的影响；单侧交感链切除显著易化了炎性热痛过敏及水肿。

Achyranthes root could significantly reduce the swelling induced by the injection of adjuvant in the paw of rats and reduce the content of PGE2 in the inflammatory exudate,which suggest that it could inhibit the biosynthesis of PGE2 in the inflammatory tissues.

牛膝散对注射佐剂引起的大鼠足跖肿胀有显著抑制作用，减少炎性渗出物中PGE2的含量，说明能抑制炎性组织中PGE2的生物合成。

The Western blot results were consistent with those of immunohistochemical method. CONCLUSION: p38 MAPK expression increases in alveolar and airway epithelial cells in LPS induced acute lung injury rat models. The activation of p38 MAPK is found in most lung tissues, suggesting that p38 MAPK participates in the signal transduction in inflammatory and noninflammatory cells.

LPS诱发的大鼠急性肺损伤模型中，磷酸化p38 MAPK在气道和肺组织内表达增加，p38 MAPK的激活见于肺组织内多数细胞，提示肺内炎性和非炎性细胞均有p38 MAPK信号分子的激活。

to use spinal lateral approach under the guidance of the c-arm x-ray machine, the needle position will be part of the disc, after successful percutaneous insert the plasma sword to melt herniated nucleus pulpous with the power, and then enter the moderate ozone to further expand the scope of ablation, and strengthen the inter-disk degradation and inhibition of the release of inflammatory mediators to eliminate protrusion of nerve roots caused by the oppression and inflammatory stimulation.

采用脊柱后外侧入路，在c型臂x线机引导下，将穿刺针定位于椎间盘部位，经皮穿刺成功后先插入等离子刀，通电后消融髓核使髓核皱缩，再打入适量臭氧，进一步扩大消融范围，加强降解间盘和抑制炎性介质的释放，来消除因间盘突出引起的对神经根的压迫和炎性刺激。

P＜0.01. 28d, q=4. 0452, p＜0.05 . Corresponding pathologic result shows that the corneal stromal of height edema increase thickness to have infiltration of inflammation cells with hyperplasia of fibroblast cell and large vessel. The collagen fiber in stromatic layer is dissolved and destroyed seriously, suggesting that in addition to HSV-1's direct destructuring effect, inflammatory cells that might play an important role in the pathology of HSK induce the immune-pathological reaction.

相应的组织病理学研究结果显示（UV-B 14天）：病毒对照组角膜基质层高度水肿、增厚，并有大量中性粒细胞等炎性细胞浸润、成纤维细胞和新生血管增生，胶原纤维肿胀、分解等，说明除HSV-1对角膜组织直接破坏外，炎性细胞介导的免疫病理反应在复发性HSK发病机理是重要的因素。

By the time of its fall, most of the prisoners were writers who had written against the corruptions of the government.

到它被攻陷的时候，里面多数的犯人是写了反对政府贪污文章的作家。

The most obvious variation to ovum morphological character was that the color was changed from light green to sepiaceous in embryonic development, and all the ovums were almost hatched after 96h.

在胚胎发育过程中卵的形态特征最明显的变化是颜色从淡绿到深褐色，卵在发育96h后卵基本全部孵化。