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The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死,从而减轻心肌的再灌注损伤,其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果:再灌注前使用CoPP进行预处理,可以诱导HO-1蛋白的表达上调;HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死,提高心肌组织中SOD含量并降低MDA的含量。

SHEN-FU Injection can accelerate the resume of myocardial function during ischemia-reperfusion, countermine arrhythmias after reperfusion, reduce the myocardial infarct size.

可以促进缺血再灌注心肌收缩和舒张功能的恢复,降低再灌注心律失常的发生率,缩小心肌梗死范围,减轻心肌组织的超微结构损伤性改变。

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After testing for normal distribution, data were tested by t test and the one - way AN OVA.

缺血再灌注中高切变的出现是暂时的,在再灌注后 60分钟时则降到正常的 50%以下。

The myocytes were continuously perfused with various experimental solutions at room temperature and paeonol applied in the perfusate.

在室温的情况下,我们用不同的灌注液和带有芍药醇的灌注液来对心肌灌流。

In patients with clinical evidence of hypotension associated with hypoperfusion and obvious evidence of elevated cardiac filling pressures (e.g., elevated jugular venous pressure; elevated pulmonary artery wedge pressure), intravenous inotropic or vasopressor drugs should be administered to maintain systemic perfusion and preserve end-organ performance while more definitive therapy is considered.

对伴有临床证据的因低血压而导致低灌注和有明确证据的心脏充盈压增高(如颈静脉压力增高、PCWP增高)的患者,应该应用静脉用强心剂或血管加压素维持组织灌注和靶器官功能,以进一步决定最终治疗方案。

CT perfusion images can visually show different traits of Hemodynamics of hepatic Benign and Malign pathology; No matter what background, various parameters of hepatic tissue around malign lesions had significant difference with far-end liver tissue, while various parameters of hepatic tissue around benign lesions had no significant difference with far-end liver tissue; Allopatric efficiency of hepatic Malign pathology distinctly went up with hepatocirrhosis background;Various parameters of hepatic Benign and Malign pathology had no significant difference and differential significance were no significant.

灌注参数图像能直观显示肝脏良、恶性占位性病变血流动力学的不同特点;无论在何种背景下,恶性病变灶周组织与远端肝组织各同种参数间有显著差异,而良性病变灶周组织与远端肝组织各同种参数间无显著差异,以上特点可以鉴别肝脏占位性病变的良、恶性;肝硬化背景下,恶性病变的发生率明显增高;正常肝组织背景下,良、恶性占位病变组织灌注参数间无显著差异,鉴别诊断意义不大。

Objective To evaluate the therapeutic effects of aminoguanidine on cerebral ischemia-reperfusion damage in rats.

目的 探讨氨基胍对大鼠脑缺血再灌注后的血脑屏障的影响及对再灌注损伤的保护作用。

The contrast material was bolus injected through antecubital vein ,while four layers of dynamic scans were performed for 50 seconds.These slice dynamic images were processed with the CT perfusion software package.Cerebral blood flow,cerebral blood volume,and mean transit time were measured within the specific regions of interest of the brain,and quantitative analysis was performed.

在常规轴面CT扫描后选取基底节及其相邻层面,经肘静脉团注对比剂,同时开始连续50 s的四层动态扫描,重建的198幅动态图像使用CT脑灌注软件包进行处理,获得灌注图像,测量所选脑内感兴趣区的血流量、血容量及平均通过时间,并对其进行定量分析。

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The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

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这种做法不仅鼓励了更多的反应,而且减少跟风的可能性。

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