灌注

Histological observation was performed. Results The peak of serum CINC concentrations occurred 3 h after reperfusion with the difference between the groups with and without ONO-5046 being significant. Expression peak of CINC mRNA in the pancreatic graft occurred 3 h after reperfusion with the expression level in the ONO-5046 treated group significantly lower than in the ONO-5046 untreated group. The activity of MPO in the treated group was obviously decreased as compared with in the untreated group.

结果 CINC于再灌注后3小时出现峰值，治疗组为(7.24±1.46)μg/L，对照组为(28.4±1.85)μg/L，差异显著；移植物中CINC mRNA于再灌注后3小时表达最强，治疗组的表达水平明显低于对照组；MPO的活性，治疗组显著低于对照组；治疗组的胰小叶间质水肿及胰小叶间质和胰小叶内的中性粒细胞浸润较轻。

Methods Twenty-four mature male rabbits were randomly divided into sham-operated group, control group, sodium ferulate group with 8 in each group. The later groups were subjected to 240 minutes of left circumflex coronary artery occlusion followed by 120 minutes of reperfusion.

随机将24只成熟雄性新西兰大白兔分为假手术组、对照组、阿魏酸钠组3组，每组8只，分别建立AMI缺血再灌注模型，于缺血前5 min、结扎后240 min，再灌注后120 min取静脉血。

METHODS: Eighteen piglets were randomly divided into control group, IR group and nonpulsatile continuous perfusion group, with 6 in each group.

18只乳猪随机分为对照组，缺血再灌注组和非搏动持续灌注组，每组6只。

The overexpression of HO-1 induced by CoPP can inhibit death of the myocardial cells and subsequently alleviate myocardial ischemia/reperfusion injury,whose major mechanism may be related with anti-oxygen free radicals.

CoPP诱导的HO-1过表达可以抑制心肌缺血再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

The upregulation of HO1 expression induced by CoPP can inhibit the necrosis of the myocardial cells and subsequently alleviate the myocardial ischemia/reperfusion injury. One of its main mechanisms may be related with antioxygen free radicals.

CoPP诱导的HO1过表达可以抑制心肌缺血-再灌注损伤后的细胞坏死，从而减轻心肌的再灌注损伤，其主要机制与抗氧自由基有关。

Result HO-1 expression was upregulated following pretreatment of CoPP before reperfusion,resulting in both an increase in SOD activity and a decrease in MDA content and thus in a reduction in death of the myocardial cells after ischemia/reperfusion injury.

结果：再灌注前使用CoPP进行预处理，可以诱导HO-1蛋白的表达上调；HO-1蛋白表达上调可以减少缺血再灌注后的心肌细胞坏死，提高心肌组织中SOD含量并降低MDA的含量。

SHEN-FU Injection can accelerate the resume of myocardial function during ischemia-reperfusion, countermine arrhythmias after reperfusion, reduce the myocardial infarct size.

可以促进缺血再灌注心肌收缩和舒张功能的恢复，降低再灌注心律失常的发生率，缩小心肌梗死范围，减轻心肌组织的超微结构损伤性改变。

Malignant tumor after 1 to 2 weeks to the beginning of reperfusion, the partial cystectomy From the beginning of reperfusion after 4 weeks.

恶性肿瘤术后1～2周即可开始灌注，行膀胱部分切除术者术后4周开始灌注。

Activity of succinct dehydrogenate, superoxide dismutase glutathione peroxides and levels of glutathione malanldehyde and nitric oxide were measured.

结果 bFGF和SM联合预处理组心肌中SDH、SOD、GSHpx活性明显高于缺血再灌注组，抗氧化物GSH和NO的含量明显高于缺血再灌注组，心肌病理改变明显好于未用bFGF和SM组。

Material and methods Biodegradable and nonantigenic in vivo albumin was selected as a carrier, and encapsulated the anticancer agent cisplatin to treat 18 patients with terminal carcinoma of tongue by infusion through lingual artery with cisplatin-loaded albumin microspheres 56.3μm in diametre, to observe and analyze the curative effect.

利用平均φ56.3μm顺铂-白蛋白微球100mg(含CDDP 13.6mg)经舌动脉灌注，治疗晚期舌癌患者18例，观察分析治疗效果。结果顺铂-白蛋白微球CDDP-AMS经舌动脉灌注治疗舌癌18例，近期疗效达特效者100%，配合其它治疗后，经5年观察，原发灶无复发。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。