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OBJECTIVE: To investigate the effect of IPC on contractile function of skeletal muscle during ischemia-reperfusion.

目的:探讨缺血预处理对骨骼肌缺血再灌注期间收缩功能的影响。

Objective To study the influence of cardioplegic preconditioning on the contractile function of isolated inchemic/repperfused rat heart.

目的研究停跳预处理对大鼠缺血,再灌注心肌收缩功能的影响。

Master the principle, image analysis of myocardial perfusion imaging, indication and contraindication of intervene test.

掌握心肌灌注显像的原理、图像分析和临床应用,介入试验的适应症和禁忌症。

Region of interestanalysis was performed in white matter and gray matter of ischemic side and contralateral side.

并对27例患者FAIR灌注图的患侧及对侧灰白质作感兴趣区分析。

All patients were performed with coronary angiography in the 2 weeks.

目的 评价腺苷负荷心肌灌注显像对冠心病诊断的临床应用价值。

ABSTRACT Objective: To observe the protective effects of Corydalis ambailis migo total alkaloids on cerebral ischemia/reperfusion injury in rats and to investigate its mechanism.

摘要] 目的:观察夏天无总碱(Corydalis ambailis migo total alkaloids, COAMTA)对脑缺血/再灌注损伤的保护作用并对其机制作初步探讨。

Ischemia-reperfusion of the cremaster could induce the apoptosis of the endothelial cells and might play an important role in microcirculation disorder.

大白鼠提睾肌缺血再灌注可引起血管内皮细胞凋亡,并可能在微循环障碍中起重要作用。

METHODS Expression of Bcl-2,Bax and P53in endothelial cells of cremaster ischemia-reperfusion of the rat was investi-gated by means of immunohistochemical SP method.

以免疫组织化学SP法对大白鼠提睾肌缺血再灌注后血管内皮细胞Bcl-2,Bax及p53的表达进行观察。

Immunoprecipitation and cresyl fast violet staining were employed to detect the expression of relevant message proteins, activation and the death of the hippocampal neurons.results it was found that hpk1 was expressed in rat hippocampus neurons.conclusions administration of hpk1 antisense oligodeoxynucleotides can significantly protect hippocampus neurons from apoptosis without dose-dependence.

使用免疫沉淀和焦油紫染色法等技术检测相关信号蛋白的表达、活化水平及神经元细胞的死亡。结果在大鼠海马神经元中有hpk1的表达,脑室注射as-odns大鼠海马ca1区存活锥体细胞明显增多。结论 hpk1反义寡核苷酸对缺血/再灌注引起的海马神经元凋亡具有保护作用。

To investigate the protective effect of curcumin on myocardial ischemia and reperfusion injury and the possible mechanism.

目的 探讨姜黄素抗心肌缺血再灌注损伤的效果及其可能的作用机制。

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