灌注

Thereby it demonstrated that resisting the damage of oxygen free radial. antigoniz- ing the overloading of calcium, relieving cerebral edema and regulating the imbalamce between 6—Keto—〓 and 〓 are the key link of NMTI in Curing ischemia/reperfu- sion injury and inhibiting the progress of the disease. The mechanism of NMTI at brain protecting also lies there. The results Provided strong basement on which NMTI was studied furtherly and widely used for curing certbral ischamia and reirrigate injury in clinical practice.

从而表明抗自由基损伤，拮抗钙超载，减轻脑水肿，调节前列腺素与血栓素失衡状态是脑脉通注射液防治脑缺血再灌注损伤，阻止病程进展的主要作用环节，也是脑脉通注射液发挥脑保护作用的重要机制所在，为脑缺血再灌注损伤的治疗和脑脉通注射液的进一步研究及临床推广应用提供了一定的依据。

In group E, which had just experienced 30-minute cold storage without rewarm ischemia, hepatic function could well be recovered in a short time during reperfusion. On the contrary, in group F、G and H, which had experienced rewarm ischemia for 15, 30 and 60 minutes respectively following 30-minute cold storage, hepatic function suffered the more severe damage with the more prolonged rewarm ischemia during reperfusion.

在对供肝复温缺血-再灌注损伤的研究中发现，供肝于再灌注期间，仅低温保存30分钟而无复温缺血的E组供肝功能于短时间内即可得到很好的恢复，但低温保存30分钟后又分别经历15、30、60分钟复温缺血的F、G、H三组供肝，其肝脏功能却遭到了明显的损伤，且复温缺血时间越长者，损伤程度越重。

Methods 28 rabbits were randomly divided into three groups:control group,ischemia/reperfusion group and Safflor injection group.

方法28只家兔随机分为假手术对照组、缺血/再灌注组和红花注射液组，分别在缺血前，缺血30min，再灌注30、60、120min取静脉血，测定血清IL-8浓度，实验结束取脑组织观察脑超微结构的变化。

Methods The rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

采用大鼠大脑中动脉缺血2 h/再灌注22 h模型，神经病学评分，脑梗塞范围及脑组织水含量变化，观察脑康灵胶囊抗脑缺血/再灌注损伤的效应；通过测定大鼠脑组织中一氧化氮合酶（nitric oxide synthase，NOS），超氧化物歧化酶（superoxide disumtase，SOD）活性及丙二醛（malondialdehyde，MDA）含量的变化以探讨药物作用的机制。

MethodsThe rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

方法采用大鼠大脑中动脉缺血2 h/再灌注22 h模型，神经病学评分，脑梗塞范围及脑组织水含量变化，观察脑康灵胶囊抗脑缺血/再灌注损伤的效应；通过测定大鼠脑组织中一氧化氮合酶（nitric oxide synthase，NOS），超氧化物歧化酶（superoxide disumtase，SOD）活性及丙二醛（malondialdehyde，MDA）含量的变化以探讨药物作用的机制。

Batroxobin can decrease PAF level in plasm after cerebral ischemic reperfusion and may have some effects on PAF-R mRNA expression in semidark band cortex after cerebral ischemic reperfusion, which provides experimental data for prophylactic intervention.

巴曲酶可降低脑缺血再灌注后血浆中PAF水平，并且可能对脑缺血再灌注缺血半暗带皮质组织PAF-RmRNA表达有影响，以期为预防性干预提供试验数据。

Systolic perfusion or diastolic perfusion was calculated bydividing the subendocardial intensity at systole or diastole,respectively,for each segmentby the mean value across all segments for each patient.T-test was used for the statistics.

DSCT冠脉成像作为心肌灌注的定性评估方法，在兼顾冠脉成像和心功能分析的基础上，能初步评价心肌首过灌注及其时相和节段的变化，为进一步评价缺血性心脏病提供参考。

AIM and METHODS:We adapted the none circulating method perfused rat heart and high performance liquid chromatography technique and compared with the control group, the ischemia reperfusion group and the Acanthopanox Senticosus group, studied the effect of Cordyceps Sinensis succi on the content of adenine nucleotides and MDA during ischemia reperfusion.

目的和方法：观察冬虫夏草对缺血再灌注过程心肌保护作用。采用Langendorff非循环式离体心脏灌流方法，与正常对照组、缺血再灌组、刺五加注射液用药组相对比，研究了冬虫夏草醇提取物对大鼠心肌缺血-再灌注过程脂质过氧化产物和腺嘌呤核苷酸含量的影响。

Methods The specimens were obtained from an in vivo rabbit model of the ischemiac reperfused lung, and were studied by use of transmissional electromicroscopy technique.

取用新西兰兔I-R模型的肺组织(正常组、缺血1h、再灌注0.5h和再灌注2h，共4组)制作常规电镜标本，在透射电镜下进行观察。

The main results and conclusions are as follows: 1. Culrured neurons were damaged by simulated cerebral ischemia and reperfusion in vitro: Results:(1) The activity of the darnaged neurons was decreased, the survival rate was descent.(2) The transudation rate of LDH in cultured medium increased obviously.

体外模拟脑缺血再灌注造成了培养神经元的损伤：表现为：(1)受损神经元的活性降低，细胞存活率下降；(2)细胞培养液LDH的漏出率明显提高；(3)细胞死亡率显著上升；(4)NOS染色强阳性细胞数在缺血期和再灌注早期明显增多。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。