灌注

30 male Wistar mice were divided into normal control group and groups treated by bleomycin on days 3,7,14,21,30.Each group included 5 mice, and the mice were treated by single intratracheal instillation with sterile saline solution or with 5 mg /kg body weight of bleomycin.

30只大鼠随机分为：(1)正常对照组 5只，经气管内一次性灌注生理盐水；(2)灌注博莱霉素分为博莱霉素3、7、14、21、30天组，每组各5只。

Objectives To study the expressions of STAT1 and PDGF in lung tissue of pulmonary fibrosis model induced by bleomycin in rats, and discuss the possible mechanism.Methods Thirty Wistar rats were randomly divided into two groups (15 in each group), they were bleomycin group and control group.①The BLM group was intratracheally instilled with belomycin (5mg/kg);② NS group was intratracheally instilled with saline (5mg/kg).

目的 观察肺纤维化鼠肺组织信号转导和转录活化因子1（STAT1）和血小板源性生长因子表达的变化，探讨其在肺纤维化的作用机制方法 30只Wistar大鼠随机分博莱霉素组和对照组，①BLM组：气管内灌注BLM（5mg/kg）诱导肺纤维化；②NS组：气管内灌注NS。

Methods Thirty Wistar rats were randomly divided into two groups (15 in each group), they were bleomycin group and control group.①The BLM group was intratracheally instilled with belomycin (5mg/kg);② NS group was intratracheally instilled with saline (5mg/kg).

30只Wistar大鼠随机分为博莱霉素组和对照组，①BLM组：气管内灌注BLM（5mg/kg）诱导肺纤维化；②NS组：气管内灌注NS。

Objective To evaluate the early effect of intravesical instillation of hydroxycamptothecinfor prevention of postoperative recurrence of superficial bladder cancer.

目的 评价羟基喜树碱膀胱内灌注预防浅表性膀胱癌术后复发的近期疗效方法对36例浅表性膀胱癌患者术后应用HCPT20mg/30ml作膀胱内灌注，每周1次，共12次。

METHODS: The middle cerebral artery occlusion model described by Zea longa was used.

采用改良Longa法复制大鼠局灶脑缺血再灌注模型。56只实验大鼠随机分为正常对照组、假手术组、缺血再灌注组及银杏叶制剂预处理组。

BACKGROUND: Recent studies have demonstrated that phenolic alkaloids from Menispermum dauricum can protect the heart and brain from ischemia/reperfusion injury, and promote neuron survival by inhibiting neuronal Bax and upregulating Bcl-2 expression following ische-mia/reperfusion.

背景：近来实验证明，蝙蝠葛酚性碱有保护心脑缺血再灌注损伤的作用，能通过抑制缺血再灌注后神经元bax和上调Bcl-2表达促进神经元存活。

Methods 100 cases were randomized into treatment group in which 50 cases were treated with Safflower injection via carotid artery infusion, added Safflower injection 20ml into 0.9% sodium chloride injection 60ml with the 4ml/min velocity, once a day; control group in which 50 cases were treated with venous drip of Safflower injection, added Safflower injection 20ml into 0.9% sodium chloride injection 500ml. After therapy, the curative effect、 myodynamia and hemorrheology were compared between the two groups.

将100例脑梗死患者随机分为红花注射液颈动脉灌注组50例和红花注射液静脉滴注组50例，治疗组：于0.9%氯化钠注射液60ml中加入红花注射液20ml，以4ml/min的滴速经颈动脉灌注；对照组：以红花注射液20ml加入0.9%氯化钠注射液500ml中静滴，20天后对两组疗效进行分析并观察治疗前后肢体肌力及血液流变学变化。

Methods:The artificial defects of 20 rabbits were repaired with frozen allogeneic mandible and fresh syngeneic mandible to study the revascularization's dynamic process of mandible transplantation by the inkgelation vascular perfusion,vascular corroisioncast,Nuclein scintigraphy;the 85 cases received frozen allogeneic mandible transplantation and the measures to improve revascularization;The defects were implanted with bFGF and allogeneic bone,the defects were only repaired with allogeneic bone as control group.

通过冷冻异体下颌骨和新鲜自体骨移植修复20例家兔下颌骨缺损的动物实验，经墨汁明胶血管灌注法、血管铸型法、放射性核素骨显像研究了异体下颌骨移植再血管化及骨改建的动态过程；同时另20例采用bFGF和异体骨修复作为实验组，单纯异体骨修复作为对照组，植入后1个月、3个月行墨汁明胶血管灌注透明法及组织学观察。

There are ultrastructural changes in the brain tissue with hepatic ischemia-reperfusion. Oxidization stress may be one of the mechanisms of the change.

肝脏缺血再灌注后可以导致脑组织超微结构的改变，氧化应激可能是肝脏缺血再灌注所致脑损伤机制之一。

Thirty minutes later intubation of pancreaticoduodenal artery was done and 3.6 ml of 0.5% CaNa2 EDTA were infused.

动脉灌注磷酯酶A2抑制剂质量浓度为0.5%的CaNa2 EDTA 3.6 ml，并与不治疗组、区域灌注生理盐水组及外周用质量浓度为0.5%的CaNa2 EDTA 3.6 ml 3组对照。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。