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Liver function was checked befor e perfusion and 1,3, 5 and 7 days after the perfusion.

分别于灌注前及灌注后1,3,5,7 d检查肝功的动态变化情况。

Results The perfusion images and parameters were obtained successfully in 18 patients.

结果 20例患者有18例成功获得灌注图像和计算出灌注参数。

Methods 20 guinea pigs were randomly divided into the experimental group and control group. By perfusing the antibody to GLAST into tympanic canal in the cochlea of guinea pigs in experimental group and artificial perilymph into the guinea's cochlea in control group, the results of ABR, basilar membrane stretched preparation and transmission electron microscope were observed.

健康豚鼠20只随机分为实验组和对照纸,每组10只实验组于蜗鼓阶内灌注GLAST抗体,对照组灌注入工外淋巴液,观察两组术后3、6、9ABR反应阈、耳蜗基底膜铺片和透射电镜的形态学改变。

METHODS In situ perfusation model was used to evaluate the absorption of Levodropropizine. The phenol red and levodropropizine in the perfusation were detected by HPLC at the same time.

采用大鼠在体小肠循环灌注模型,用HPLC同时测定灌注液中酚红和左羟丙哌嗪的含量。

The rabbits were randomly divided into three groups (n=9 in each): sham operated group, PIR group and PIR+ propofol group.

实验兔27只,随机均分为假手术对照组、肺缺血-再灌注组和肺缺血-再灌注加异丙酚治疗组。

Methods Rats were randomly divided into control group, ischemia-reperfusion group and ischemic preconditioning group.

将大鼠随机分为对照组、缺血再灌注组和缺血预处理后缺血再灌注组。

Methods Rats were randomly divided into control group,ischemiareperfusion group and ischemic preconditioning group.

将大鼠随机分为对照组、缺血再灌注组和缺血预处理后缺血再灌注组。

Prismoid specimen shall adopt horizontal type molding; when pour concrete and tamp down the specimen with hidden steel bar, concrete between steel bar and specimen mould shall be taken particular care so as to keep right condition of pouring and tamping compaction.

棱柱体试件宜采用卧式成型,埋有钢筋的试件在灌注混凝土及捣实时应特别注意钢筋和试模之间的混凝土能保持灌注密实及捣实良好。

Perfusion can be read directly on Pseudocolor images, but the result is coarse.

胰腺彩色灌注图直观,根据颜色可直接读出灌注值,但结果粗略。

Discussion TNF-αis an inflammatory cell cytokine,with the properties of autocrine, produced by cadiocyte at the early age of MIRI,inducing myocardial function abnormal and cadiocyte necrosis in MIRI、pyaemia、chronic cardiac insufficiency、viral myocarditis and the homograft rejection of heart.

讨论 TNF-α是心肌缺血再灌注早期心肌细胞产生的炎性细胞因子,是一种自分泌的作用因子,在缺血再灌注损伤、脓毒症、慢性心功能不全、病毒性心肌炎以及心脏同种移植排斥反应中均能导致心肌功能能障碍和心肌细胞坏死。

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