漏

Methods DNA aneuploid and TA were detected respectively in 165 cases of malignant pleural effusion and 127 cases of benign dropsy of chest (benign group, including tuberculous,other effusion and transudate of chest) by FCM and TRAPHybELISA.Carcinoembryonic antigen was also detected in the two groups.The difference of DNA aneuploid and TA between these two groups was analyzed.

恶性积液165份、良性积液127份(包括结核性、其他渗出液、漏出液)，应用流式细胞术检测DNA异倍体、端粒末端重复序列扩增—杂交—酶联免疫分析技术检测TA，同时检测临床常用的癌胚抗原，比较各类积液中上述3项指标检测结果的差异。

Therefore, accumulates based on SEAG the chest cavity the fluid to divide into the high gradient (SEAG≥11 g/l) or the low gradient (SEAG1 g/l), namely the transudate or the percolate, accumulate the fluidity archery target diagnosis to the chest cavity to have the important clinical value.objective Studies on the effects of psychological therapy on HIV/AIDS prisoner in improving their psychological problems.

因此，依据SEAG将胸腔积液分为高梯度性(SEAG≥11 g/l)或低梯度性(SEAG1 g/l)，即漏出液或渗出液，对胸腔积液性质的诊断有重要的临床价值。目的研究心理疗法对解决艾滋病病毒感染者/艾滋病病人罪犯的心理问题、帮助HIV/AIDS罪犯重塑健全人格的作用。

Test of NO, TNFa, LPS in the serum and ascites of the patients with liver cirrhosis ascites can contribute to distinguishing between exudative ascites and transudate ascites and distinguishing between spontaneity peritonitis is infected in Gram-positivebacteria and Gram-negative-bacteria so as to instruct clinical use of drugs.

肝硬化腹水患者血清及腹水中测定NO、TNFa、LPS，有助於鉴别渗出性腹水和漏出性腹水；有助於鉴别自发性腹膜炎是革兰阴性菌或革兰阳性菌感染引起，指导临床用药。

Methods Ascites of the patients with liver cirrhosis were carried out routine test to distinguish between exudative ascites and transudate ascites. All patients with exudative ascites had bacterium cultured and drug-effective tested in their ascites. At the same time, nitric oxide, tumor necrosis factor-a, LPS were tested in the serum and ascites of all patients with liver cirrhosis ascites.

肝硬化腹水患者常规作腹水检查，以区别渗出性腹水或漏出性腹水，对渗出性腹水患者均作细菌培养＋药敏，所有肝硬化腹水患者均同时作血清及腹水一氧化氮、肿瘤坏死因子、内毒素水平检测。

The main results and conclusions are as follows: 1. Culrured neurons were damaged by simulated cerebral ischemia and reperfusion in vitro: Results:(1) The activity of the darnaged neurons was decreased, the survival rate was descent.(2) The transudation rate of LDH in cultured medium increased obviously.

体外模拟脑缺血再灌注造成了培养神经元的损伤：表现为：(1)受损神经元的活性降低，细胞存活率下降；(2)细胞培养液LDH的漏出率明显提高；(3)细胞死亡率显著上升；(4)NOS染色强阳性细胞数在缺血期和再灌注早期明显增多。

Early hypotheses of atherosclerosis included the "incrustation" hypothesis of Rokitansky (778), suggesting that intimal thickening was due to arterial fibrin deposition, and the "lipid transudation" hypothesis of Virchow (995) according to which lipid complexes with mucopolysaccharides caused atherosclerosis.

早期的动脉粥样硬化症假说包括：指出内膜增厚是因为动脉纤维蛋白存积所致的Rokitansky(778)「结痂」假说、以及含有黏多醣之脂质合成物会造成动脉粥样硬化症的Virchow(995)「脂漏」假说。

Kangdai Ⅰ has protective function to the damaged neurons and astrocytes: Main results:(1) It has direct protective function to the damaged neurons. It can increase the activity and survival rate, decrease the mortality and the transudation rate of LDH in cultured medium and the strong positive cell count of NOS expression of injured neurons.(2) It has also directly protective function to the damaged astrocytes. It can increase the activity and survival rate and protein content in conditioned medium.(3) It can strengthen the ability of BDNF, GDNF, bFGF, HSP and IL-6expression in damaged astrocytes.(4) It can also strengthen obviously the expressions of NSE, bFGF-receptor and bc1-2, lower the expression of bax and caspase-3.(5) It can indirectly protect and restore the damaged neurons by astrocytes. Because the effect of ACMK (ACM interfered by Kangdai Ⅰ) is stronger than ACM+K (ACM associated with Kangdai Ⅰ).

抗呆Ⅰ号对受损的神经元和星形胶质细胞均具有保护作用：主要表现为：(1)对受损神经元具有直接的保护作用，可提高受损神经元的活性和存活率，降低细胞培养液LDH的漏出率、细胞死亡率和NOS染色强阳性细胞的表达量；(2)对受损的星形胶质细胞也有直接的保护作用，可提高其活性、存活率以及培养液蛋白质的含量；(3)能增强受损星形胶质细胞分泌BDNF、GDNF、bFGF、HSP和IL-6的能力；(4)可明显增强受损神经元对NSE、bFGF的受体和bc1-2的表达，降低受损神经元对bax和caspase-3的表达；(5)抗呆Ⅰ号可通过星形胶质细胞间接地保护和修复受损的神经元，因为在多数实验组中经抗呆Ⅰ号作用的ACM的作用远大于ACM与抗呆Ⅰ号联合应用的作用，统计学上具有显著性差异。

Cytomorphology changes occurred obviously in cultured neurons such as cell edema, cell atrophy, etc."Jiu Nao Ning"Injection could improve the activity, decrease the transudation rate of LDH and the apoptotic rate. It could also increase the contents of DNA, RNA and improve the pathologic changes in cultured neurons.

救脑宁注射液高、中、低剂量组，在各时间点表现出不同程度地提高细胞代谢率、降低LDH漏出率和凋亡率的作用，与模型组比较p＜0.05；同时也显示出改善细胞形态学异常变化、增加细胞内DNA、RNA含量，保护神经细胞功能与结构免遭损伤等作用，以中剂量效果最好，其与清开灵的药效作用相比，要么相似要么更佳。

The optical density value of cells was detected by MTT colorimetric method, content of MDA and NO in the supernatant fluid of cultured cells was detected by spectrophotometer, and the transudatory content of LDH was detected by automatic biochemistry analyzer.

用MTT法检测细胞吸光度值，利用分光光度计分别测量细胞上清液中的MDA和NO含量。用自动生化分析仪测定培养液中LDH漏出量。

The protection of AF－1 was observed with the measurement of transudatory rate of LDH ,the breaking rate of DNA and contents of LPO and SOD,Results:The intotoxincation of glutamate was decreased by AF－1 which reduced the transudatory rate of LDH,the breaking rate of DNA and the content of LPO,and increased the content of SOD in culture significantly.

通过测定乳酸脱氢酶漏出率、DNA断裂率、超氧化物歧化酶和脂质过氧化物，观察AF－1药物的保护作用。结果：高浓度的谷氨酸可增加培养神经细胞的LDH释放率和DNA断裂率，并诱导自由基产生。AF－1明显降低谷氨酸的神经细胞毒性，使LDH释放率及DNA断裂率明显降低，LPO含量明显下降，SOD含量明显升高。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。