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To resolve this behavior, only the surface business card printing and membership card making to set to print in ink that is not on the surface without worrying about the colour, there are three ways: the first one is a drumlike complex members'meeting, Bopp/business card printing and membership card making/PE, such a structure you need to have special line cost more per bag fee in order to achieve $ 0.10/only, the second film structure is the PE/business card printing and membership card making/PE, such a structure common packaging equipment can also be filling, but cost more, each bag shall be $/only 0.08, the third option is the-tape-PE/business card printing and membership card making/PE structure in the original film, that is, the members'meeting membrane business card printing and membership card upon completion of the making of the operation, the business card printing and membership card making surface salivate a layer of transparent with thin-film, this is our company according to actual usage of the product structure specially developed.

给彻底处置这一不隐象,只有不兵暗地制卡和会员卡制作改不败边印,油不朱不在暗地也不离供给安心掉色,办法有不一栽:第一栽是复不合奶膜布局为BOPP边/制卡和会员卡制作/PE边,这样的布局需有附带灌装摆设,不败本也较矮,每一个袋子用度给到达0.10元/只把握),第二栽膜的布局是PE/制卡和会员卡制作/PE,这样的布局别致灌装摆设也可灌装,但不败本还是较矮,每袋须0.08元/只把握,第不一栽办法是流延PE/制卡和会员卡制作/PE布局不厚膜,即在原奶膜制卡和会员卡制作完不败后,再增增一道工序,在制卡和会员卡制作背流一层透明的较不厚的不厚膜,这是我们公司根据客户本质搁置环境出格开不收的产物布局。

Materials and Methods 15 cases included 8 salivary gland mixed tumors, 2 capillary hemangiomas, 3 squamous cell carcinoma and 2 mucous epidermoid cyst.

资料与方法本组15例中腺混合瘤8例,海绵状血管瘤2例,鳞状细胞癌3例,黏液表皮样囊肿2例。

OBJECTIVE: To discuss the expressions of 2 sub-types of SSTR, SSTR1 and SSTR2 in human salivary gland mucoepidermoid carcinoma(MEC-1 ) and the high metastatic cell strain(Mc3) of human mucoepidermoid carcinoma and the differences of these two receptors in the binding with SSTR radioactive genin, 125I-RC-160, and the correlations with prngnostic rehabilitation.

目的:探讨人腺粘液表皮样癌细胞系(MEG-1)及人粘液表皮样癌高转移细胞株(Mc3)生长抑素受体两种亚型(SSTR1、SSTR2)的表达与SSTR的放射配基125I-RC-160的结合差异与预后康复的相关性。

The face is paralytic can happen at all ages, but 15~45 year old the commonnest, come on the peak is reached inside over number hour or 1~3 D, can incline the head unhealthily at the beginning of disease or ache after mandible horn, expression breaks down for flesh of a side countenance, frontal grain disappears, the eye is cracked greaten or close faint, when closing a key point, eyeball up foreign and rotational, show white sclerotic, call Bell the phenomenon, channel of ill side nose lip becomes shallow, quarrel prolapse, side of crooked Xiang Jian of the quarrel when grinningly, rouse cheek or whistle hourglass is enraged, the food when taking food often stops to age at ill side buccal between, at the same time companion has weep reach salivate.

面麻痹可发生于所有年龄,但15~45岁最常见,发病多于数小时或1~3 d内达到高峰,病初可有病侧耳或下颌角后疼痛,表现为一侧面部表情肌瘫痪、额纹消失、眼裂变大或闭合无力,闭眼时,眼球向上外方转动,露出白色巩膜,称Bell现象,病侧鼻唇沟变浅,口角下垂,露齿时口角歪向健侧,鼓腮或吹口哨时漏气,进食时食物常滞留于病侧齿颊之间,同时伴有流泪及流

Abstract〕Objective: To study the effects of hexamethylene bisacetamide on the metastatic potential of mucoepidermoid carcinoma Mc3 cells.

摘要〕 目的:研究六亚甲基双乙酰胺对腺粘液表皮样癌高转移细胞Mc3恶性表型的影响。

In this paper,we used hexamethylene bisacetamide as a inducer to induce human mucoepidermoid carcinoma cell line MEC-1 cells to differentiate in vitro and vivo.The nucleolus organizer regions was investigated by means of the argyrophil technique.

本实验以六亚甲基二乙酰胺作为分化诱导剂,对腺粘液表皮样癌MEC-1细胞进行了体内外诱导,并通过银染技术对对照组和HMBA诱导组癌细胞核内核仁组成区的数目、大小及分布特征进行了观察。

Signs for both include hypersalivation, vomiting, lethargy, tremors, difficulty walking, weakness, and death.

临床表现包括:多、呕吐、嗜睡、震颤、行走困难、虚弱,甚至死亡。

But the side????effects such as somnolence, hypersalivation, constipation, weight gain, abnormal ECG, nausea, leukopenia decreased significantly.

研究组嗜睡、流、便秘、体重增加、心电图异常、恶心呕吐、白细胞减少等因子显著少于对照组,差异有显著性(P<0.01或0.05)。

But the sideeffects such as somnolence, hypersalivation, constipation, weight gain, abnormal ECG, nausea, leukopenia decreased significantly.

研究组嗜睡、流、便秘、体重增加、心电图异常、恶心呕吐、白细胞减少等因子显著少于对照组,差异有显著性(P<0.01或0.05)。

To address more specifically the mechanisms involved in cortical bone healing, we created drill-hole defects in the cortex of mouse femur, a lesion that triggers intramembranous repair, and compared the roles of bone sialoprotein and osteopontin, two proteins of the extracellular matrix, in the repair process.

为了更具体的了解皮质骨修复过程的机制,我们在小鼠股骨上制造了一个钻孔缺损,这一损伤能引发股骨的膜内修复,并比较细胞外基质中的两种蛋白骨蛋白和骨桥蛋白在这一修复过程中的作用。

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