海马后的

Results: The expression level of Brn-4 mRNA started to increase on the 3rd day after fimbria fornix transaction. The peak appeared on the 14th day , and then the expression decreased slowly to pre-transection level on the 28th day.

结果：海马内Brn-4 mRNA的表达量在切割后第3 d开始升高，14 d达到最高水平，随后下降，28 d左右恢复至正常水平。

Results: The expression level of Brn-4 mRNA started to increase on the 3rd day after fimbria fornix transection.The peak appeared on the 14th day,and then the expression decreased slowly to pre-transection level on the 28th day.

结果：海马内Brn-4 mRNA的表达量在切割后第3 d开始升高，14 d达到最高水平，随后下降，28 d左右恢复至正常水平。

Results: the expression level of lhx8 mrna started to increase from the 3rd day after fimbria fornix transection. the peak appeared on the 7th day, and the expression decreased slowly to pre-transection level on the 28th day.

结果：海马内lhx8 mrna的表达量在切割后第3 d开始升高，7 d时达到最高水平，以后逐渐下降，于28 d时恢复至正常水平。

The influence on PDE activity was assayed in hippocampus homogenate in vitro.

体外给予G-Rg1后，检测海马匀浆液中PDE活性的变化。

Methods: The rats bearing cerebro-ventricle intubations, were inflicted with 30% of total body scope area of the animals, full thickness burn injury. The pathological changes, the conten ts of brain water, the contents of LDH and NO in hippocampus were observed in th e rats.

SD大鼠侧脑室埋管，制成30％体表面积Ⅲ度烫伤模型；伤后3 d，测定脑组织含水量，海马乳酸脱氢酶和一氧化氮的含量；做病理切片，尼氏染色。

Methods AD model was established by injecting β amyloid protein into the hippocampus of rat. The rats were killed ten days after injection. The hippocampus sections were made coronally on a freezing microtome.

采用海马注射A淀粉蛋白的方法建立AD模型。10d后对大鼠进行灌注，取脑，冰冻切片，用NGF、BNDF和NT3抗体行免疫组织化学染色。

However, effects of the short-term SD on learning and neurogenesis were controversy and still not clear.

本研究之主要目的在观察Long-Evans雄性大鼠进行12小时的短期睡眠剥夺后，对其海马回神经新生与空间学习之影响。

The results indicated that EMP can promote necrosis and apoptosis of hippocampal neurons,induce morphological changes in blood vessels, neuroglia cells and neurons at early stage, which may be the possible injury mechanism in rat brain induced by EMP.

表明大鼠大脑受电磁脉冲辐照后早期海马区可发生神经细胞坏死和凋亡，以及各组织成分的病理形态学改变，上述变化可能与电磁脉冲致细胞DNA损伤有关。

Objective To investigate the long term protectingly effect ofdiazepam interferential treatment on hippocampal formation in immaturerats with status epilepticus.

目的 探讨地西泮干预治疗幼鼠早期癫痈持续状态对成鼠后海马结构损伤的远期保护作用。

Methods The mRNA and protein expression levels of NCAM were detected by reversed transcriptive polymerase chain reaction and Western blot after the rat hippocampal neurons were incubated with 20, 40 and 80μg/ml sodium fluoride for 24 hours in vitro.

原代培养海马细胞暴露于20、40、80μg/ml氟化钠24 h后，用RT-PCR和Western blot方法分别检测细胞内NCAM mRNA和NCAM各蛋白亚型的表达。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。