海马后的

Results The results showed that:(1) Only fluoxetine could decrease the 5-HT immunoreaction in cornu ammonis and thalamas in rat brain in the acute group;(2) Four antidepressant drugs could decrease 5-HT immunoreaction in six regions of rats brain in the chronic group.

结果 ①一次给药后，仅氟西汀降低大鼠海马和丘脑5-HT免疫反应性；②长期给药组中，4种抗抑郁药均能降低大鼠额叶、海马、丘脑、脑干和小脑的5-HT免疫反应性。

the increase of lhx8 mrna expression after fimbria fornix transected might be related to the regeneration mechanism of differentiation of neural stem cells into cholinergic neurons in hippocampus.

切割穹窿海马伞后海马中lhx8 mrna表达的增高可能与神经干细胞向胆碱能神经元分化的再生机制有关。

Objective To study the distribution of substance P in the rat hippocampal formation and the change of SP in the rat hippocampal formation after hepatocirrhosis.

目的 研究P物质在大鼠海马结构中的分布特点及肝硬化后对大鼠海马结构中SP的影响。

Results: Neonatal rats in hypoxic-ischemicgroup behaved restlessness, cyanosis, deep and rapid breath, astasia, lethargy, irritation and spasm; There were local pyknosis, fragmentation, dissymmetry, blur or disappearance of nucleoli, raritas of ground substance in left cerebral cortex; but there was no statistic significance in chinese traditional medicine group and sham group. The expression of HIF-1α mRNA in brain tissue of experimental group and chinese traditional medicine group were higher than that in sham group, especially chinese traditional medicine group at 3d; HIF-1α positive cells were found in cerebral cortex and hippocampus. There were more HIF-1α positive cells in experimental group and chinese traditional medicine group and mainly in vascular endothelial cells.

结果 缺氧缺血后大鼠相继表现为烦躁不安、全身发给、呼吸加深加快、站立不稳、嗜睡、激惹或间断发作的痉挛和抽搐；HE染色显示实验组大鼠左侧大脑皮层出现局灶性神经元核固缩，核碎裂，核仁偏位，不清或消失，基质疏松；中药组大鼠脑组织结构基本接近假手术组，无明显水肿和坏死表现；实验组和中药组大鼠脑组织HIF-1α mRNA表达较假手术组明显增加，尤其是中药组实验第3日者；免疫组化显示实验组和中药组各时间点大鼠大脑皮层和海马区均可见不同程度的HIF-1α表达，明显高于假手术组，阳性表达主要在血管内皮细胞，海马和皮层的锥体细胞亦有HIF-1α阳性细胞分布。

Yinao Jieyu Prescription and fluoxetine can increase the expression of bFGF and decrease the level of GFAP in the area of hippocampus CA3 and cingulated cortex of model rats.

颐脑解郁方、盐酸氟西汀能增加脑出血后抑郁模型大鼠海马CA3区、扣带皮质bFGF的表达，减少海马CA3区、扣带皮质GFAP的表达。

Results: Remarkable pathological changes were found in h ippocampus: Nissl bodies decreased or disappeared, cyton swelled. The water cont ents and the level of LDH and NO were increased in scalded rats. CNTF could rema rkably improve the pathological changes and reduce the increase of LDH, NO a nd the water contents.

结果：大鼠严重烫伤后，脑组织含水量增加，海马出现怎么明显的病理变化，尼氏小体减少或消失，胞体肿胀，LDH和NO的含量增加；给予CNTF后，脑水肿、海马的病理变化有明显改善，并能降低 LDH和NO的含量。

The cells expressing nAchR clearly decrease in the areas of CA1 and cortex of rats when their fimbria/fornix is transected, but the cell number increases significanly if ectogenic estrogen is added. The change in nAchR is closely relative with AD whose pathological course can be influenced by the intervention of estrogen.

穹隆海马伞切断组大鼠脑内海马CA1区、皮层区nAchR的阳性细胞显著减少，补充外源性雌激素治疗后，上述各脑区内的nAchR表达显著增加。nAchR的变化与AD的病理变化过程密切相关，补充雌激素可以干预这种病理过程。

The cell suspensions were injected into the hippocampus of rat which had received unilateral transection of fimbria-fornix before one week. 5,7,14,30,60,90,150 and 180 days after grafting, the rats were perfused and postfixed, cut section. A series of every second and third sections were used for NOS enzyme histochemistry and Nissl histochemistry.

将大鼠14～16d胚胎的基底前脑移植到单侧穹隆海马伞切断的成年大鼠海马内，动物在移植后存活5、7、14、30、60、90、150和180d分别取脑，经NADPH-d法和尼氏染色观察。

The machanism may be that 7-NI inhibits activation of nNOS and decreases the number of overproducing neuronal NO in hippocampal during anoxia,so the ability of hippicampal slices against anoxic injury is increased and neurotoxic role of neuronal NO during anoxia is definite.

作用机理可能是7-NI抑制nNOS活性降低缺氧后海马神经原性NO的过多形成，从而提高海马脑片抗缺氧损伤的能力，更加明确了缺氧早期神经原性NO的神经毒性作用。

Three hours following seizures there was an evident expression of c-fos proteins in the hippocampus (CA1 area), dentate gyrus, piriform cortex, dorsal part of entorhinal cortex, and amygdaloid nucleus, and there was a dramatic increase of c-fos proteins in CA3 area and the areas mentioned above except the CA1 area where c-fos proteins apparently decreased after electroacupuncture treatment.

在海马青霉素致痫模型上观察了致痫及致痫加电针后脑内c-fos蛋白的变化，结果表明致痫时海马〓区、齿状回、梨状皮层、背侧内嗅皮层、杏仁核见到较多的c-fos表达；电针后c-fos在〓区明显减少，在〓区及上述其余各区则显著增加。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。