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Neuroanatomical bases of spatial memory[J].

大鼠海马 CA3区突触素的增龄性变化[J]。

The therapy groups displayed significant improvements in neuromotor functions and survivals of neuron and oligodendrocyte compared with control groups. These results demonstrated the neuroprotect roles of GGF2, and cationic liposome-mediated GGF2 transgene therapy has potential possibility.

结果:1、大鼠LFP致伤后3—7天,GGF2 mRNA在海马CAl区、CA2区、CA3区、DG及皮层表达明显增加。2、阳离子脂质体介导GGF2转基因治疗可促进大鼠伤后行为学的恢复,可促进损伤区神经元及少突胶质细胞的存活。3、原核表达方法得到GGF2重组蛋白。

Results The neuronic apoptosis in cortex and hippocampus appeared at 6 hoursafter HIBD, peaked at 24 hours, and then decreased gradually, There was a remarkable lower quantity in melatonin treated groups compared with HIBD group at 6h,12h,24h,48h and 72h (p0.05), but there were significant differences between theirs and after HIBD2 hours groupp

结果:TUNEL法显示皮质和海马神经元的凋亡于缺氧缺血后6小时开始出现,24小时达高峰,然后逐渐下降,MT治疗组组间存在差异,但各组在6小时、12小时、24小时、48小时、72小时均显著低于HIBD组(p0.05),但它们与HIBD后2小时组在各项指标的变化上均有显著性差异p

In vitro electrical neurophysiological and behavioural studies have shown that diabetes mellitus negatively affects hippocampal function.

离体电生理和行为研究表明,糖尿病会对海马功能造成负面影响。

We now show that cognitive and behavioral deficits and impaired neurophysiological function accompany early hippocampal spongiform pathology.

本研究显示认知和行为缺陷及受损的神经生理功能与早期海马海绵样病变有关。

Objective To explore the expression of brain-derived neurotrophic factor in the hippocampus after ovariectomy in mice.

目的:探讨小鼠卵巢切除后的不同时期脑源性神经营养因子在海马表达的变化。

Objective: To explore the effect of exercise training on concentration of hippocampus brain-derived neurotrophic factor in mental stress rats.

目的:探讨适宜运动训练对心理应激大鼠海马脑源性神经营养因子水平的影响。

AIM Aim to study the protein expression of neurotrophic factor in rat brain.

目的 观察拟除虫菊酯对正常大鼠大脑皮层和海马的甲状腺激素反应基因-神经营养因子蛋白表达的影响。

Objective To investigate the effect of 8 Hz infrasound on the expression of brain derived neurotrophic factor in the rat hippocampus.

目的研究频率为8 Hz,声压级分别为90,100和130 dB的次声作用对大鼠海马脑源性神经营养因子表达水平的影响。

This effect may be closely related to the up-regulation of the BDNF signal pathways of hippocampus, therefore promotion of the neural neurotrophy and plasticity.

CTN-T在大鼠慢性应激模型上具有抗抑郁作用,其作用机制可能与上调海马BDNF相关的信号传导通路活性,改善神经营养和神经可塑性有关。

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