海马

Low-dosage veratridine may induce runs of PDS like epileptiform activities on rat CA1 pyramidal neurons.

小剂量藜芦碱可在大鼠脑海马CA1区锥体神经元上诱发出阵发性去极化漂移样癫痫活动。

Recertly, the intrinsic nature of PDS has received more attention.OBJECTIVE: To observe the characteristics of epileptiform activities of rat hippocampal CA1 pyramidal neurons induced by low-dosage veratridine and investigate its possible ion mechanism.

近年来，阵发性去极化漂移的内因机制得到进一步关注和重视。；目的：观察小剂量藜芦碱诱发大鼠脑海马CA1区锥体神经元产生癫痫样活动的特征，探讨其可能的离子机制。

RESULTS: After perfusion of 0.5 μmol/L veratridine, the rat pyramidal neurons in CA1 area displayed relatively fixed-mode of runs of PDS bursting, followed by the hyperpolarization of cell membrane.

小剂量藜芦碱可在大鼠脑海马CA1区锥体神经元上诱发出阵发性去极化漂移样癫痫活动。

Methods By means of whole-cell patch clamp technique, the epileptiform discharge model of rats hippocampal CA1 neurons was constructed with extracellular perfusion of 0.5 μmol/L veratridine, and the model should be regarded as successfully estabilshed if bursting discharge emerged within 30 min perfusion.

通过膜片钳全细胞记录技术，胞外灌流0.5μmol/L藜芦碱，制作大鼠海马CA1锥体神经元癫痫样放电模型，如果在灌流30min内出现振荡样放电，即认为造模成功。

Objective To observe the effects of low dose of veratrine on the discharges of rat hippocampus pyramidal neurons, and to elucidate its possible cytological mechanism.

目的 观察小剂量藜芦碱对大鼠海马CA1锥体神经元放电的影响，并对其诱发产生癫痫样放电的离子机制进行初步探讨。

Inducing slow wave epileptoid bursts, the low dose of veratrine can remarkably change the discharge features of CA1 pyramidal neurons.

小剂量Ver可明显改变大鼠海马CA1锥体神经元的放电模式，诱发慢波样癫痫电活动。

Methods The discharge features of hippocampus CA1 pyramidal neurons of 14-day-aged healthy Sprague-Dawley rats induced by low dose (0.3~0.8mol/L) of veratrine were observed by slice patch-clamp technique.

应用脑片膜片钳全细胞记录技术，观察小剂量(0.3~0.8mol/L)Ver灌流后SD大鼠（2~3周龄，共30只）海马CA1锥体神经元的放电特征。

The lateralization of TLE was defined by the absolute volumetry value of HF and the normal range of the deviation of bilateral HF volume.

采用正常人HF体积的正常范围及左右两侧HF体积的差值的正常范围对TLE患者可能存在的海马硬化进行定位定侧。

Aim: To observe the influence s of propofol anesthesia on the release of noradrenaline andα2-NA receptorinhibitor yohimbine in rat hippocampus so as to identify themechanisms of central noradrenaline in propofol anesthesia.

目的：观察大鼠异丙酚麻醉与中枢神经系统海马去甲基肾上腺素的释放以及α2-NA受体抑制剂之间的相互关系，探讨中枢性NA机制在异丙酚麻醉中发生的作用。

The results showed that NOS positive neurons were present in the hippocamp us CA1～CA3） in which NOS strong positive neurons were in CA2; NOS medium positive nucleus in the nuclei nervorum non-cranialium (red nucleus、 su bstantia nigra; NOS positive neurons in the nuclei nervorum cranialium (oculomo tor、 accessory oculomotor nucleus, facial nucleus, abducens nucleus); NOS posit ive nucleus in the pons reticular formation; NOS positive nucleus in the raphe n ucleus and in the plexus chorioideus ventriculi quartic and plexus chorioideus v entriculi tertii.

结果显示：海马(CA1～CA3区)NOS为阳性、其中CA2 为强阳性++）；非脑神经核(红核、黑质为中等阳性；而脑神经核(动眼神经核、动眼神经副核、面神经核、展神经核)NOS为阳性；脑桥网状结构为阳性；缝核群为阳性；第三脑室和第四脑室脉络丛为阳性。

For a big chunk of credit-card losses; the number of filings (and thus charge-off rates) would be rising again, whether

年美国个人破产法的一个改动使得破产登记急速下降，而后引起了信用卡大规模的亏损。

Eph. 4:23 And that you be renewed in the spirit of your mind

弗四23 而在你们心思的灵里得以更新