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Changes in false operation group, rehabilitation group and the intracerebral synaptic structure parameter of the model group.

为此本实验采用右侧大脑中动脉缺血大鼠模型应用滚笼网状训练、平衡训练以及Morris水密宫训练后,观察假手术组、经康复训练的康复组和未经训练的模型组大鼠在Y-迷宫分辨学习能力和一次性被动回避反应记忆保持能力的行为模式以及海马CA3区突触结构参数测定、在体LTP记录和NMDA受体通道电流记录等方法研究康复训练对梗死大鼠学习记忆的影响及其机制。研究内容包括: 1。

Our parent company has nearly 20 years of history and employs a lot of excellent engineers.

韩国世纪海马产业公司拥有近二十年的生产经验及优秀的工程设计及技术人员。

Second we use excitability medicine to provocat the nerve,s causality discharage,after 1 to 2 minutes,perfuse the brain slice with a certain concentration of puerarin to be abaissement,in result,we find that some kinds concentration(such as 1:1000) of puerarin have prominen depressant effect to the provocated discharage of the hippocampal nerves.

之后,通过在电活性较好的脑片上先进行兴奋性药物的诱发,待诱发性放电出现后1-2分钟再灌注一定浓度的葛根素进行实验观察,结果表明,一定浓度的葛根素(如1:1000)对海马神经元的诱发放电活动有显著的抑制作用。

Objective: Investigation of the Kangnaoling granulation"s protective effect on cerebral vascular injured rat"s hippocampus neural cell of pasting cerebral pia mater with FeCl_3 solution.

目的:观察康脑灵颗粒剂对FeCI_3溶液贴敷软脑膜所致脑血管损伤模型大鼠海马神经细胞的保护作用影响。

Right hemisphere became obviously swelling and pale, the sign ofhemorrhage under cerebral pia mater is manifest, and the other side of the brain isnormal. The colors of whole brains were stained into rose pink with TTC.

电镜超微结构形态学观察:小脑皮质最先在HIBD 1h出现凋亡征象,其次为大脑皮质在HIBD 12h、海马CA1区在HIBD 24h出现凋亡细胞超微结构明显改变,随损伤时程进展各部位细胞超微结构改变明显,细胞器损伤加重。

Methods Synaptosomes was made from hippocampus and incubated with artificial cerebrospinal fluid. 30μmol/L propofol, 50μmol/L picrotoxin or both was added into aCSF. Intralipid was administered as solvent control and any drugs was applied in control. To observe Ca~(2+)-dependent release of glutamate and GABA, dihydrokainic acid and nipectic acid were added into aCSF.

制备大鼠海马突触体,随机分为5组:PTX组加入印防己毒素使其终浓度为50μmol/L,Pro30+PTX组加入终浓度为30μmol/L的丙泊酚和50μmol/L的PTX,Pro30组加入终浓度为30μmol/L的丙泊酚,溶剂对照组加入脂肪乳,空白对照组不加入上述药物。

By using radioactive ligand receptor binding assay and observing animal activities, this paper reports the effects of convulsants of picrotoxin, penicillin and kainic acid on the 3H GABA A binding in the cerebral cortex, hippocampus and cerebellum of mice.

采用放射配体受体结合分析法和动物行为观察,研究了致惊药印防己毒素(picrotoxin ,Pic)、青霉素(Penicillin ,PG)、红藻氨酸(kainicacid ,KA)对小白鼠大脑皮层、海马、小脑三脑区3 H GABA与GABAA 受体结合的影响。

Methods Rats were injected with Lithium-chloride,Pilocarpine intraperitoneally to establish TLE model.

轴突出芽是颞叶癫突出的病理学特点,海马兴奋性异常增高是颞叶癫的电生理学特点。

Next the authors attempted to activate the latent stem cells in vivo, using pilocarpine to invoke seizures, and thus hippocampal synaptic activity, in mice.

这些研究者下一步尝试了体内研究潜在干细胞群,他们用毛果芸香碱作用于小鼠引发小鼠癫痫和海马突触活动。

Second, I exercise is "the pineal gland,""pituitary" and "brain hippocampus" near the muscles.

二是我锻炼的是"松果体"、"脑垂体"和"大脑海马"附近的肌群。

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