海马

Meanwhile, in the CA3 area of hippocampus in NS group, a small amount of N-Cadherin positive cells were visible and were slightly stained. Compared with NS group, in PTZ seizure group, N-Cadherin positive cells had a marked increase in area CA3 and dentate hilus (P.01), but they were strongly inhibited in PDTC group (P.05). There was no correlation between the expressions of N-cadherin, Timm-stained granules and between the age of rats with seizure.

同时，NS组14天和28天大鼠海马CA3区可见少量的N-Cadherin阳性细胞，着色不深；PTZ致惊组海马CA3和齿状回门区的N-Cadherin阳性细胞与NS组相比明显增多（P＜0.01），PDTC预处理后相同区域内N-Cadherin阳性细胞较PTZ致惊组明显减少（P＜0.05）。N-Cadherin与Timm染色颗粒的表达结果与惊厥鼠的日龄并无关联性。

Results The learning and memory ability of experimental group was better than that in saline control group (P＜0.01), but there was no significant difference between experimental group and normal control group (P＞0.05).There was significant difference in cytochrome C of hippocamp and frontal lobe between saline control group and the other groups (P＜0.01).The expression of cytochrome C in hippocamp was higher than that in frontal lobe of experimental groups.

结果 NSCs移植后AD模型大鼠的学习记忆能力明显改善，与生理盐水对照组相比差异显著（P＜0.01），与正常组相比无显著差异（P＞0.05）；海马与额叶的CytC表达显示生理盐水对照组与其他各组之间有显著差异（P＜0.01）；各实验组海马内的CytC表达均高于额叶。

The data showed that GST-RI significantly decreased level of MDA and obviously increased activity of SOD, GSH-Px in the hippocamp of brain that underwent ischemia following reperfusion in mice.

结果： 融合蛋白GST-RI能显著降低脑缺血小鼠皮质及海马组织中MDA的含量，能显著提高脑缺血小鼠脑皮质及海马组织中SOD、GSH-PX的活力。

It implies that electroacupuncture Ren and DuMeridians can restrain the excess proliferation of hippocamp horizontal cells after ischemiawhile enhance the cell differentiation.

提示电针任督脉使缺血海马GFAP／NSE双标细胞增加的同时，可以调节脑缺血后缺血海马GFAP的表达，这两方面作用优于单独电针督脉组。

Results: Compared with sham operation group, the ability of vascular dementia model mouse for learning and memory reduced, which showed extending the time for swimming the whole process, and increasing times of mistakes, and the activity of SOD in hippocamp was decreased and the content of MDA was increased(P.01). Compared with model group, Danggui Shaoyao-San could make the time for swimming the whole process shorten and times of mistakes decreased, and increase the activity of SOD and decrease the content of MDA in hippocamp, which were excellent than that of contrast medicine.

结果：与假手术组比较，模型小鼠学习与记忆能力下降，表现为游全程时间延长，错误次数增加(P.01)，脑海马SOD活性明显降低，MDA含量明显升高(P.01)；与模型组比较，当归芍药散组可使游全程时间缩短，错误次数减少，脑海马SOD活性明显升高，MDA含量明显降低(P.01)，并优于对照药物(P.05)。

Resultsthe ability of vascular dementia model mouse for learning and memory reduced, which showed extending the time for swimming the whole process, and increasing times of mistakes; the content of cgrp and ss in hippocamp reduced. dangguishaoyao-san could make the time for swimming the whole process shorten and times of mistakes decreased, and increase the content of cgrp and ss in hippocamp, which were excellent than that of contrast medicine.

结果模型小鼠学习与记忆能力下降，表现为游全程时间延长，错误次数增加，脑海马cgrp和ss含量下降；当归芍药散可使模型小鼠游全程时间缩短，错误次数减少，脑海马cgrp和ss含量升高，优于对照药物。

The Sham rats were only performed the incision on cervix but without any treatment on the arteries. Apoptosis neurons in hippocamp CA1 subregion were observed at 72 hours after global brain ischemia.

在脑缺血再灌注后72小时，杀死大鼠，取海马组织依POD法作凋亡细胞检测，计数各组大鼠海马CAI区凋亡细胞数。

Regions of limbic systemincluding hippocamp,parahippocamp,insular lobes showed increased positivefunctional connectivity with the bilateral STR.

海马、海马旁回以及岛叶等边缘系统与双侧STR正连接增强。

OBJECTIVE: To establish a detailed model of hippocampal CA3 function according to CA3 structure.

目的：根据海马CA3区的结构，建立一个海马CA3区功能的详细模型。

In spite of having 50% volume loss bilaterally in his hippocampi, retrieval in Jon was associated with increased activation of the hippocampi.

尽管他双侧海马已经各失去50%的容量，Jon的记忆提取伴随著海马激发的增加。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。