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The ratio of PMNs activity was distinguishingly higher in patients with positive blood culture,and also increased in patients with positive results of midstream urine culture and sputum culture.

血培养阳性的全身感染PMNs活化率最高,中段尿培养及痰培养阳性者PMNs活化率与正常对照相比也增高,但低于血培养阳性者。4 2例感染组患儿PMNs活化增加占78 。6 %,C反应蛋白增高仅占31%。

Based jellification dynamics theory that the larger the jellification activation energy, the greater the lubricant's jelling tendency, it is advisable to analysis the whole jellification process in attempt to find out maximum jellification activation energy Emax and its ambient temperature Tmax, as a ground for judging the lubricant's pumpability.

根据凝胶动力学理论,凝胶活化能越大,润滑油的凝固趋势越大,因此对润滑油应分析其凝胶全过程,从中找出最大凝胶活化能Emax以及开始出现最大活化能的温度Tmax,作为润滑油低温可泵性判据。

The results of activity energy calculation were abserved as that, compared with that of the control, cineol and menthanol decreased activity energy of model drug permeation, menthone didn't change it, while bornel, camphor and bisaborlol increased activity energy.Two systems of microemulsion components were selected according to the results of solubility research of model drug in oil phase mixture, and the pseudo ternary phase diagram of them were draw.

活化能实验结果表明,桉树脑和薄荷醇显著减小模型药物透皮活化能,薄荷醇与对照组比较没有显著差异,冰片、樟脑和红没药醇存在下,药物的透皮活化能显著增加。

The sulfation ratio of the unreacted CaO in the spent sorbent increases with the hydration temperature or the reaction time decreasing. The overmany hydration water reduces the sulfation ratio of the unreacted CaO and there exists an optimal rate.

增湿活化温度越低,高温脱硫时间越短,越有利于低温脱硫反应;随着活化水量的增加,低温脱硫反应的钙利用率先提高后降低,活化水量存在一个最佳值。

Experiment conclusions ①The expression and activation of MARCKS and p-MARCKS were elevated in brain under the condition of acute ischemia, suggesting that over expression of MARCKS transudation pathway might play an important role in brain ischemia.②Treatment with Qingnao Pilular could inhibit the over expression and activation of MARCKS; MARCKS signal transduction pathway could be novel target for the treatment of acute ischemia.

2实验研究结论①在缺血易损区MARCKS及p-MARCKS表达与活化明显增强,提示MARCKS蛋白过表达和活化与脑缺血损害的发生密切相关;②清脑滴丸可以调节脑缺血易损区MARCKS的过表达与活化,MARCKS信号转导通路可能是脑缺血保护治疗的新靶点,为熄风清热、化痰活血法治疗急性缺血性脑血管病提供了一定的理论和实验基础。

Our results indicate that 1 NSC606985, at nanomolar level, can effectively induce apoptosis in AML cells NB4 and U937 and significantly inhibit the proliferation without cell death in breakpoint cluster region–Abelson murine leukemia kinase-carrying leukemic K562 cells; 2 At such low concentrations, this agent also significantly inhibits the clonogenic activity of hematopoietic progenitors from patients with AML; 3 For apoptosis induction, NSC606985 rapidly induces the proteolytic activation of protein kinase Cδ with loss of mitochondrial transmembrane potential and caspase-3 activation; 4 Co-treatment with rottlerin, a PKCδ-specific inhibitor, completely blocks NSC606985-induced mitochondrial m loss and caspase-3 activation, while the

结果显示:(1)纳摩尔浓度的NSC606985即可有效诱导AML细胞系NB4和U937细胞凋亡并显著抑制含有bcr-abl融合蛋白激酶的K562细胞的增殖;(2)低浓度的NSC606985也显著抑制来自AML病人骨髓的新鲜白血病细胞的克隆形成能力;(3)除了迅速诱导线粒体跨膜电位的崩塌及Caspase-3的活化外,NSC60698也导致蛋白激酶Cδ的水解激活;(4)PKCδ特异的抑制剂rottlerin能够完全抑制NSC606985诱导的线粒体跨膜电位的崩塌和Caspase-3的活化,而Caspase-3特异的抑制剂z-DEVD-fmk仅能部分削弱PKCδ的活化及细胞的凋亡;(5)以移植PML-RARα转基因小鼠产生的白血病细胞建立的模型研究了该化合物对白血病可能的治疗作用。

According to the data of chemical composition analysis, XRD and TGA, the rule of carbonate and sulphation reaction in middle temperature range is found, and the feasible temperature range is determined for SO〓 removal with the carbonate reaction occurring at the same time.

为了进一步发展中温烟气脱硫技术和蒸汽活化技术,今后还将研究床内的物料量和循环倍率对脱硫效率的影响,同时利用本文单纯石灰脱硫和蒸汽活化所得到的结果和机理,研究粉煤灰存在的情况下,蒸汽活化对脱硫剂钙利用率的影响。

The results of the determinating the activity of the CA in vitro showed that the CA of the water—fed individuals were in activing statics after eclosion. Because of the apperence of the Vg in haemolymph and the deposition of the vitellin in oocytes in the unfed females, it was shown that the CA in such individuals were in activiting statics, too. Again because in the water—fed females which did not lack the feeding action the activity of the CA showed dropping tendency gradually still, it could be said that the activiting of the CA was possibly effected by the nutrition but it was never related to the adult feeding action. In another respect, the brain of the female adult had not significant effect on the activiting CA. The oocytes could depositing vitellin in individuals removed brain tissue on the day of eclosion, which showed that the activting of CA was not concerned with brain.

CA活性的体外测定说明饲水个体的CA同样具有活性,不取食个体血淋巴中Vg的存在以及卵母细胞中卵黄的沉积表明,在不取食的个体中CA同样具有活性,所以CA的活化与成虫期的取食动作无关,与成虫是否取食花蜜也无太多的联系,但是CA活性的维持可能受成虫期营养的影响,如果以卵母细胞发育和CA体积大小作为CA活性的指标,则饲水个体CA的活性在后期受到了影响,说明饲水不能替代取食蜂蜜来维持成虫期CA的活性;成虫期脑对CA的活化无明显作用,羽化当日摘除脑的个体,其卵母细胞仍然可以沉积卵黄,说明CA的活化与脑无关,而是受某种不确定的因子的影响;用抗JH物质—早熟素—Ⅱ以10ug/虫的剂量在颈部点滴处理雌虫,对Vg的发生和卵巢的发育具有一定的迟缓作用,其原因有待于进一步的研究。

The research show that from anthracite, lean coal to bituminous, reaction rate become more and more large and ignition activation energy become more and more small; ignition activation energy falls and reaction rate increases as the volatile content increases;With the carbon content of dry ash-free basis increasing, ignition activation energy increases and reaction rate falls.

结果表明:从无烟煤,贫煤到烟煤,其反应速率越来越大,频率因子越来越小,着火活化能越来越低;挥发分增加,活化能减小,反应速率变大;干燥无灰基固定碳含量增加,着火活化能增加,反应速率减小。

RESULTS ① TLSFJM could induce apoptosis of PHA plus IL-2-activated mouse thymo cytes;② TLSFJM could induce apoptosis of alloantigen activated human pe ripheral blood mononuclear cells and hold the cell cycle at G1 phase;③ TLSFJM neither influenced the CD25 expression nor induced apoptosis of PMA activated human PBMC .

结果 TLSFJM 在体外可诱导PHA/IL-2活化的小鼠胸腺细胞的凋亡。 TLSFJM 在体外可诱导alloantigen活化的人外周血单个核细胞的凋亡,且把细胞周期阻遏在G1期。 TLSFJM 对PMA诱导的人PBMC活化抗原CD25的表达无明显影响,也不引起凋亡。

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随着死亡的吉他手Schuldiner接受主唱的职务,乐队在现实中树立了重要的影响。

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