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Results : After intervention of MRS, the treated group;the structure of free villi was clear, intra-axial capillary was not congistid, vascular wall was intact, villi area and intervillous space area compared with control group, the difference was significant(p.01).interspace area was smaller than blank group, but without significance.

结果:经黄芪丹参复方成分干预后,治疗组游离绒毛结构清晰,中轴内毛细血管未见充血,管壁较完整,绒毛面积及绒毛间隙面积与模型组相比,差异显著(P.01),间隙面积比空白组略小,但无统计学意义;底蜕膜层结构清晰,血管数目较多。

60 SD rats were divided in to 3 groups randomly: sham operation group, ischemia reperfusion injury group, and leonurus japonicus injection treating group.

60只 SD大鼠被随机分为3组:假手术组、缺血再灌注损伤组、益母草注射液治疗组

Result more Xi Luowei orders cure troop is calorific drop and pharynx clip is phlogistic improve, the time that liver narrows after lienal lymph node enlargement all relatively interferon contrasts the group is apparent shorten (P · 05), GCV group shows efficiency interferon of apparent prep above group (P · 05 or 0 · 01), short-term use more dosage control is in Xi Luowei 8~10mg/, did not see apparently harmful response happens.

结果更昔洛韦治疗组发热下降和咽夹炎好转,肝脾淋巴结肿大后缩小的时间均较干扰素对照组明显缩短(P·05),GCV组显效率明显高于干扰素组(P·05或0·01),短期使用更昔洛韦剂量控制在8~10mg/,未见明显不良反应发生。

INTERVENTION: The models of ischemia/reperfusion in SD rats were established by middle cerebral artery occlusion with a nylon monofilament suture. The rats were randomly divided into the treatment groups 32cases and the control group 32 cases. Each group was divided into eight subgroups, which consisted of 4 rats at 2 hours, 6 hours, 12 hours, 24 hours, 2days , 3 days, 7 days and 14 days after reperfusion of MACO.

干预措施:成年健康雌性SD大鼠68只,应用线栓法建立SD大鼠大脑中动脉阻塞再灌注模型,随机分为治疗组32只和对照组32只,每组再随机分为缺血1.5 h再灌流2,6,12,24 h,2,3,7,14 d组(n=4),另外4只作假手术组。

Methods:One hundred and sixtyeight patients meeting the diagnostic criteria were randomly divided into 3 groups: Moutan powder with Aspirin group, Moutan powder alone group and Aspirin alone group, 56 patients in each group.

采用分组对照的方法,对符合诊断标准的168例患者随机分为三组,每组56例。治疗组给予牡丹皮散加阿司匹林,中药对照组给予牡丹皮散,西药对照组给予阿司匹林。

Thirty minutes later intubation of pancreaticoduodenal artery was done and 3.6 ml of 0.5% CaNa2 EDTA were infused.

动脉灌注磷酯酶A2抑制剂质量浓度为0.5%的CaNa2 EDTA 3.6 ml,并与不治疗组、区域灌注生理盐水组及外周用质量浓度为0.5%的CaNa2 EDTA 3.6 ml 3组对照。

Forty rats were randomly divided into ALR therapy group and saline control group, which were injected peritoneally ALR gene (200μg/kg) and saline per 12h respectively 4h after CCl4 injection.

在染毒后4h,将大鼠随机分为治疗组和生理盐水组,每组20只,每12h分别腹腔注射ALR基因(200 μg/kg)和生理盐水,存活超过96h计为存活。

Rats in the Danshen treatment group and the normal saline group were peritoneally injected with Danshen injection (3.0, 10.0, 15.0 g/kg) and normal saline respectively at 30 minutes before ischemia to establish the global cerebral ischemia models by modified 4-vessel occlusion method.

丹参治疗组和生理盐水组大鼠在缺血前30min腹腔分别注射丹参注射液(3.0,10.0,15.0g/kg)和生理盐水再进行4血管阻断建立全脑缺血模型;缺血再灌组模型建立同前,但不注射丹参和生理盐水。

Patients in treatment group (n=70) were treated by combination of Jieheling and phthisical.

结果 治疗组和对照组在强化期痰菌阴转率分别为82.86%和67.14%,2组比较差异有统计学意义(P<0.05);2组强化期病灶吸收及空洞闭合情况比较差异有统计学意义(P<0.05)。

Results:⑴Animals made into models turned to polyuric, overeating, polydipsic, and appeared dejected.

4中药治疗组和培哚普利组血液粘度和纤维蛋白原在2周和4周时都低于模型组(P﹤0.05,P﹤0.01)。

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