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He Study on the Role of Toll-Like Receptor 2, 4 in Acute Edematous Pancreatitis

oll样受体2,4在大鼠急性水肿性胰腺炎发病中的作用研究

Methods: Acute edematous pancreatitis and acute necrotizing pancreatitis were induced on male Wistar rats.

使用雄性Wistar大鼠建立急性水肿性胰腺炎与急性坏死性胰腺炎动物模型。

Methods we examined the serum level of TNF and TNFR75 in 22 patients with acute edematous pancreatitis,and 10 patients with acute necrosis pancreatitis on the first,third,fifth and seventh day.

对急性充血水肿性胰腺炎组22例和急性出血坏死性胰腺炎组10例病人外周血液TNF及可溶性s-TNFR75浓度在起病第1、3、5、7天进行检测。

Myxedema syndrome: obvious sex ,growth and development retardation, myxedema,mental disability,T4↓ TSH↑.25 percents, goiter

粘液水肿性综合征:以显著的生长发育和性发育落后、粘液水肿、智能低下为特征,血T4↓TSH↑。25%有甲状腺肿大

T4↓TSH↑.Myxedema syndrome: obvious sex ,growth and development retardation, myxedema,mental disability,T4↓ TSH↑.

粘液水肿性综合征:以显著的生长发育和性发育落后、粘液水肿、智能低下为特征,血

Methods The acute edematous pancreatitis and hemorrhagic necrotizing pancreatitis model of rats were induced by cerulein and dextran-110 and endothelin-1 was administered on AEP rats via intravenous injection. Serum amylase, plasma ET and 6-Keto-PGF1α, pancreas hostologic observation were determined.

应用蛙皮素及高分子右旋糖酐造成大鼠急性水肿性和出血坏死性胰腺炎模型,观察血浆内皮素水平及其它相关指标,同时观察外源性内皮素对水肿性胰腺炎大鼠血清淀粉酶、6-Keto-PGF1α及胰腺组织病理形态等的影响。

Tender, erythematous, edematous plaques that may centrally vesiculate on the upper part of the body associated with fever and leukocytosis characterize Sweet's syndrome.

身体上部压痛性水肿性红斑块伴发热和白细胞增多为Sweet综合征的特征,这可能是骨髓性白血病的早期征象。

Tender, erythematous, edematous plaques that may centrally vesiculate on the upper part of the body associated with fever and leukocytosis characterize Sweet's syndrome.

身体上部触痛、红斑块、水肿性斑块伴发热和白细胞增多为 Sweet 综合征的特征,这也可能是骨髓性白血病的早期征象。

American Atlanta conference edited in September 1992 pancreatitis of serious illness acute definition is: AP companion has local complication (cyst of pancreatic and necrotic, false sex, pancreatic abscess) or the organ fails (function of shock, lung not complete, kidney declines, gastrorrhagia, sow medicinal powder the cruor inside sexual blood-vessel and serious metabolization obstacle), or both holds concurrently.

概要: 急性胰腺炎为临床常见急腹症,是由于胰液和溶蛋白酶漏入到胰腺实质和胰腺四周组织而引起。1963年,法国马赛会议以病理形态学为基础,将AP分为水肿性胰腺炎和出血、坏死性胰腺炎。1992年9月美国亚特兰大会议修订重症急性胰腺炎定义为:AP伴有局部并发症(胰腺坏死、假性囊肿、胰腺脓肿)或器官衰竭(休克、肺功能不全、肾衰、胃出血、播散性血管内凝血和严重的代谢障碍),或二者兼有。

The number of BrdU labeling in the nucleus was remarked increasing onbFGF-treated pancreatitis rats than pancreatitis rats without bFGF treatment. And there was a significant difference.(P.01)ConclusionThe result of the study has demonstrated the effective therapeutic effects on acute edematous pancreatitis rats, which induces proliferation of immature acinar cells, facilitates the repair of damaged pancreatic tissues and reconstitutes tissue integrity.

兔疫组化结果显示bFGF治疗组大鼠具有BrdU标记的细胞核胰腺细胞数明显多于非治疗对照组大鼠,两者在统计学上具有显著性差异Oeq.of人结论实验结果表明bFGF对急性水肿性胰腺炎大鼠具有明显的治疗作用,它的治疗作用具体表现在诱导非成熟的胰腺细胞分化增殖,促进受损胰腺组织修复,重建组织完整性。

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