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Results Intraocular pressure recovered to normal in 15 cases with in 3 days and 1 case with in 3 weeks, 2 cases remained under normal after the surgery. 16 cases experienced improvement of vision after surgery. cystoid macular edemaoccurred to two cases.

结果 15例在术后3 d内眼压恢复正常及短暂增高后恢复正常,1例在术后3周内眼压逐渐恢复正常,2例手术后眼压仍然低。16例术后视力不同程度提高。2例出现黄斑囊样水肿

Many diseases may cause cystoid ma- cular edema,which suggests that CME is not an independent disease,but a clinical sign and result caused by many diseases.

许多眼病可产生黄斑囊样水肿,它不是一个独立的疾病,而是各种眼病所产生的临床症状和后果。

Objective To evaluate the clinical effects of krypton yellow laser photocoagulation for cystoid macular edema in nonproliferative diabetic retinopathy.

目的 评价氪黄激光治疗非增殖性糖尿病性视网膜病变(nonproliferative diabetic retinopathy, NPDR)所致的黄斑囊样水肿(cystoid macular edema, CME)的临床效果。

Methods A total of 430 eyes in 251 cases of diabetic focal macular edema, diffuse edema and cystoid edema were treated with krypton yellow laser photocoagulation.

目前激光光凝术已成为治疗糖尿病性黄斑水肿的常用方法,但激光参数的选择是临床治疗十分重要而又难统一的问题。

There was statistically significant difference (P<0.01).CONCLUSION: Intravitreal injection of TA is a safe and effective method for the treatment of cystoid macular edema after cataract surgery.

玻璃体腔注射TA是一种安全有效的治疗白内障术后黄斑囊样水肿的方法。

Methods RTA produced by Tilia company was used to analyze the patients who were suspected to have get macular hole or cystoid macular edema.

采用Tilia公司的视网膜厚度分析仪对怀疑黄斑裂孔及黄斑囊样水肿的患者进行分析。

AIM: To observe therapeutic effect of intravitreous injection of triamcinolone acetonide for cystoid macular edema after cataract surgery.

目的:观察玻璃体腔注射曲安奈德(triamcinoloneacetonide,TA)治疗白内障术后黄斑囊样水肿(cystoid macular edema, CME)的疗效。

Results Among the 105 patients,the postoperative hypopsia were caused by keratopathy in 37,optic nerve atrophy in 21,macularpathy of high myopia in 15, diabetic retinopathy in 11,senile macular degeneration in 8,cystoid macular edema in 7,idiopathic macular hole in 4 and idiopathic macular epiretinal membrane in 2 respectively.

结果 本组低视力病例中37例为角膜病变,21例为视神经萎缩,高度近视黄斑病变15例,糖尿病性视网膜病变11例,老年性黄斑变性8例,黄斑囊样水肿7例,特发性黄斑裂孔4例,特发性黄斑前膜2例。

The sensitivity of RTA was high. Macular hole and cystoid macular edema can be diagnosed definitely with RTA.

视网膜厚度分析仪灵敏度高,利用RIA可以确诊黄斑裂孔及黄斑囊样水肿

Objective To investigate the clinical features, causes of blindness and diagnosis of Vogt Koyanagi Harada syndrome Methods The data of 157 patients with VKH syndrome were reviewed and analyzed Patients were carefully examined with slit lamp, ophthalmoscope, three mirror lens, fundus fluorescein angiography, indocyanine green angiography and HLA typing Results Headache was noted in 73 5% of these patients Simultaneous involvement of both eyes occurred in 80 8% of these patients Chroiditis,papilledema and edema of the retina adjacent to the optic nerve were noted in 100% of these patients in the posterior uveitis stage, whereas recurrent granulomatous anterior uveitis (98 4%),"sunset glow" fundus (95 8%) and Dalen Fuchs nodules (71 2%) were the common ocular findings in the recurrent anterior uveitis stage The common causes of blindness were papillitis, exudative retinal detachment and complicated cataract in the posterior uveitis stage, anterior uveal involvement stage and its recurrent stage Poliosis (36 3%) and alopecia (35 0%) were the most common extraocular findings Early irregular patches of fluorescence, followed by localized hyperfluorescent spots were the typical findings of FFA Dilation of choroidal vessels and leakage of ICG from the choroidal vessels were the common ICGA findings The prevalence of HLA DR4 and HLA DRw53 in patients (54 9% and 71 8% respectively) was significantly higher than that in controls (14 7% and 38 2% respectively) Conclusions VKH syndrome is characterized by chroiditis, papillitis or neuroretinitis in the posterior uveitis stage, followed by a generalized uveitis with a typical recurrent granulomatous anterior uveitis Extraocular findings and relevant examinations including FFA, ICGA and HLA typing are helpful to the diagnosis of VKH syndrome

目的探讨Vogt-Koyanagi-Harada综合征患者的临床特征、盲目原因及诊断等有关问题。方法对在1996年1月至2000年12月间就诊资料完整的157例VKH综合征患者进行回顾性分析,并对裂隙灯、眼底镜、三面镜、荧光素眼底血管造影(fundus fluorescein angiography,FFA)、吲哚青绿血管造影(indocyanine green angiography,ICGA)及人类白细胞抗原分型等检查结果进行分析。结果 VKH综合征最常见的前驱症状为头痛(102例,73.5%),双眼同时患病118例(80.8%);后葡萄膜炎期眼部主要表现为脉络膜炎、视乳头及附近视网膜水肿(100.0%);前葡萄膜炎反复发作期眼部表现为复发性肉芽肿性前葡萄膜炎(128例,98.4%)、晚霞状眼底改变(95.8%)及Dalen-Fuchs结节(71.2%);后葡萄膜炎期、前葡萄膜受累期及前葡萄膜炎反复发作期导致盲目的主要原因分别为视乳头炎、视网膜脱离及并发性白内障;毛发变白(36.3%)及脱发(35.0%)是最常见的眼外表现;炎症活动期FFA典型表现为斑驳状高荧光,ICGA发现脉络膜血管扩张、通透性增高等改变;VKH综合征患者HLA-DR4及HLA-DRw53的阳性率(54.9%及71.8%)显著高于正常对照组(14.7%及38.2%)。结论 VKH综合征患者在后葡萄膜炎期眼部典型表现为双侧脉络膜炎、视乳头炎或神经视网膜炎,随后出现以反复发作的肉芽肿性前葡萄膜炎为特征的全葡萄膜炎。眼外症状及相关的辅助检查包括FFA、ICGA 及HLA分型等有助于VKH综合征的诊断。

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