毒毒血症

One hand mechanical obstruct led to the increase of veinous resistance and the obstacle of microcirculation, the other hand the adhesive PMN was activated in excess, the white blood cells released a lot of enzymes, in which PMN-elastase can decompose the components of cell and many albumens, inclusive of immunoglobulin、alexin and fibrication. These components induced the injury of the pancreatic capillary vessels and cell and lysosome enzy made the tissue protein hydrolyze and produced unsaturated fatty acids, which destroyed the structure and function of cellar membrane. The inflammatory cellar factors activate other immunocytes to produce the injury and necrosis of tissue, which aggravated the pathological injury and led to shock、pyaemia and MODS. So ICAM-1 and LFA-1 played an important role in SAP. Frossard found that the expression of ICAM-1 in the rat model, especially in serum、pancreas and lung. All these showed ICAM-1 is an important factor in AP and concomitant lung injury.

胰腺小叶组织局部血管EC首先被激活，ICAM-1表达升高，与被激活的PMN表面表达的LFA-1相结合，"PMN-EC"相互作用加剧，一方面机械性阻塞毛细血管导致静脉阻力增加、微循环障碍；另一方面粘附的PMN过度吞噬或激活，当白细胞吞噬的颗粒不能被封闭隔离，连同细胞内的酶被释放出来，其中的PMN-elastase能够降解细胞基质中各种成分，水解多种蛋白，加重胰腺的毛细血管内皮细胞和腺泡的损伤；释放的溶酶体酶使组织蛋白水解，产生的不饱和脂肪酸引发脂质过氧化方应，破坏细胞膜的结构和功能；释放的炎性细胞因子，激发其他的免疫细胞的功能，导致进一步的组织损伤和坏死，加重SAP的病理损伤，最终导致休克、脓毒血症及多器官功能障碍等严重后果。

A long dike will collapse because of an ant-hole in it; a tall Building will be Burned down by a spark form a chimney's chink

在猪出现虚脱和突然死亡的个别急性病例中，毒血症可能是一种原因。

A condition in which the blood contains toxins produced by body cells at a local source of infection or derived from the growth of microorganisms.

毒血症一种血中含有毒素的状况，毒素是由传染病灶部的人体细胞产生的，或来自于微生物的生长

Infection positions are inclined to lead to abscess, then invade into organs and blood stream and results in organic abscess for marion, pyemia is another clinical character of Chromobacterium violaceum infection.

可引起感染部位脓肿，并可侵入体内各脏器及血液引发脏器的脓肿，脓毒血症是该菌感染的临床特症，重者可引起各脏器功能衰退，中毒性休克甚至死亡。

Endplate potentials, acetylcholine potentials, and electrotonic potentials were recorded from the motor endplates of isolated diaphragms from acute late septic and nonseptic rats.

记录急性迟发性脓毒血症和非脓毒血症大鼠的离体横膈运动终板上的终板电位、酰胆碱电位和电紧张电位。

And they often are considered as "prime criminals" of triggering systemic inflaming reaction such as sepsis or bacteremia and so on.

革兰氏阴性菌细胞外壁中的脂多糖结构即内毒素，经常是引发脓毒症、菌血症等系统性炎症反应的&元凶&。

Serum and urinary methylmalonic acid levels were found to be significantly decreased after LDLT, whereas serum and urinary MMA levels did not return to normal in any patient. One patient died of sepsis 44 days after LDLT.

血和尿中甲基丙二酸的水平在LDLT后明显下降，虽然任何一名患者血和尿中MMA水平均为降至正常。1名患者在LDLT后因脓毒血症而死亡。

A prospective, observational study of critically ill patients with septic and nonseptic AKI was performed from September 2000 to December 2001 at 54 hospitals in 23 countries.

为此，我们从2000年9月到2001年12月在23个国家54家医院针对脓毒血症性和非脓毒血症性急性肾损伤的危重患者进行了一项前瞻性观察。

Piglet edema were first discovered in Britain, is hemolytic E. coli intestinal toxemia, so capillaries or systemic damage to the small blood vessels, increasing permeability and fluid extravasation too much water, to the head, eyelids, Equality ear edema, acute ataxia and death as the main feature of acute infectious diseases, also known as gastrointestinal edema pigs or pig intestinal toxemia E. coli, a highly lethal disease.

仔猪水肿病最早发现于英国，是由溶血性大肠杆菌引起肠毒血症，而使全身毛细血管或小血管受到破坏，通透性增大，水液外渗过多，造成的以头部、眼睑、耳部等处水肿、共济失调和急性死亡为主要特征的急性传染病，又称猪胃肠水肿或猪大肠杆菌肠毒血症，有高度致死性疾病。

It is concluded that latencies of P wave and N wave of CSEP prolong obviously with the development of sepsis.

脓毒血症过程中随病情的发展大鼠CSEP正、负波潜伏期有逐渐延长的趋势，为脓毒血症的基础与临床研究提供了依据。

They weren't aggressive, but I yelled and threw a rock in their direction to get them off the trail and away from me, just in case.

他们没有侵略性，但我大喊，并在他们的方向扔石头让他们过的线索，远离我，以防万一。

In slot 2 in your bag put wrapping paper, quantity does not matter in this case.

在你的书包里槽2把包装纸、数量无关紧要。