死亡率

Objective: Cardiac tamponade has a high mortality rate if aggressive and proper therapeutic measures are not undertaken.

目的：心包膜填塞如果未接受积极而适当的治疗会导致高死亡率。

Microsurgery cerebelli tentorium inciseon and local application of papaverine could take effect on reducing brain

显微手术天幕缘切开、罂粟碱明胶海棉片贴敷能够彻底解除脑干受压、防治血管痉挛进而减轻由此引发的脑干继发性损伤，降低病残率和死亡率。

Accordingly,the cell viability is very low in drier thalli.

酶解后细胞死亡率高。

In this process rectal temperature of the rats were measured every 20 min with the same thermometric probe.

结果制剂组能减缓肛温的上升速率，显著延长大鼠的存活时间，有效地降低死亡率。

Results Among 581 cases, 569 cases were cured, with the recovery rate of 97.4%; 12 cases died, with the death rate of 2.6%, and the main cause in 2 of the 12 deaths was related to injury of heart and large blood vessels, in 2 to thoracoabdominal injury, in 2 to acute respiratory distress syndrome, and in 6 to thorax injury complicated with severe craniocerebral injury.

结果 全组治愈569例，治愈率97.4%，死亡12例，死亡原因包括伤及心脏及大血管2例，胸腹联合伤2例，ARDS 2例，胸外伤合并重度颅脑损伤6例，死亡率2.6%。

Objective To study first aid and nursing method of acute organophosphorus toxicosis,in order to improve the competence of treating and curing and reducing death rate.

目的 研究急性有机磷中毒的急救与护理方法，以提高救治水平，降低死亡率。

Research on four plant extracts showed that tangerine had high stomach and contact toxity to Blattella germanica.

触杀试验结果表明，桔皮油、芥末油和生姜汁对德国小蠊若虫表现出一定程度的触杀作用，其校正死亡率率分别为58.62％、29.31％和15.52％，观察尚未发现大蒜提取物对德国小蠊触杀作用。

In order to verify that free radical damage and pulmonary surfactant deficiency may be two of ARDS major mechanisms, we used ARDS animal model to proceed to a prevention and treatment test of ARDS model by pulmonary delivery, results of animal study showed that the drug preparation can evidently reduced the mortality and pulmonary edema of model animals . Animal tests had showed that the drug preparation has no abnomal toxity.

为了验证自由基损伤和肺泡表面活性剂缺乏的结合可能是ARDS发病的主要因素以及制剂的疗效，通过建立ARDS实验动物模型对该制剂进行肺部给药防治ARDS的模型动物试验，实验结果表明能明显降低模型动物死亡率和肺水肿，同时，动物实验证明该制剂无异常毒性。

The main results and conclusions are as follows: 1. Culrured neurons were damaged by simulated cerebral ischemia and reperfusion in vitro: Results:(1) The activity of the darnaged neurons was decreased, the survival rate was descent.(2) The transudation rate of LDH in cultured medium increased obviously.

体外模拟脑缺血再灌注造成了培养神经元的损伤：表现为：(1)受损神经元的活性降低，细胞存活率下降；(2)细胞培养液LDH的漏出率明显提高；(3)细胞死亡率显著上升；(4)NOS染色强阳性细胞数在缺血期和再灌注早期明显增多。

Kangdai Ⅰ has protective function to the damaged neurons and astrocytes: Main results:(1) It has direct protective function to the damaged neurons. It can increase the activity and survival rate, decrease the mortality and the transudation rate of LDH in cultured medium and the strong positive cell count of NOS expression of injured neurons.(2) It has also directly protective function to the damaged astrocytes. It can increase the activity and survival rate and protein content in conditioned medium.(3) It can strengthen the ability of BDNF, GDNF, bFGF, HSP and IL-6expression in damaged astrocytes.(4) It can also strengthen obviously the expressions of NSE, bFGF-receptor and bc1-2, lower the expression of bax and caspase-3.(5) It can indirectly protect and restore the damaged neurons by astrocytes. Because the effect of ACMK (ACM interfered by Kangdai Ⅰ) is stronger than ACM+K (ACM associated with Kangdai Ⅰ).

抗呆Ⅰ号对受损的神经元和星形胶质细胞均具有保护作用：主要表现为：(1)对受损神经元具有直接的保护作用，可提高受损神经元的活性和存活率，降低细胞培养液LDH的漏出率、细胞死亡率和NOS染色强阳性细胞的表达量；(2)对受损的星形胶质细胞也有直接的保护作用，可提高其活性、存活率以及培养液蛋白质的含量；(3)能增强受损星形胶质细胞分泌BDNF、GDNF、bFGF、HSP和IL-6的能力；(4)可明显增强受损神经元对NSE、bFGF的受体和bc1-2的表达，降低受损神经元对bax和caspase-3的表达；(5)抗呆Ⅰ号可通过星形胶质细胞间接地保护和修复受损的神经元，因为在多数实验组中经抗呆Ⅰ号作用的ACM的作用远大于ACM与抗呆Ⅰ号联合应用的作用，统计学上具有显著性差异。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。