正常体

The abnormal release of DA from ischemic striatums were significantly inhibited by L-NNA.

结果：大鼠半球缺血30分钟时，纹体DA的释放形成高峰，其后逐渐下降，但都高于缺血前释放量。L-NNA能明显抑制缺血时纹体DA的不正常释放。

However,all of the DA releases during 60,90,120min global ischemia were significantly higher than that before ischemia. The abnormal release of DA from ischemic striatums were significantly inhibited by L-NNA.

结果：大鼠半球缺血30分钟时，纹体DA的释放形成高峰，其后逐渐下降，但都高于缺血前释放量。L-NNA能明显抑制缺血时纹体 DA的不正常释放。

Dense cytoplasm with abundant mitochondria, endoplasmic reticulums, multivesicular bodies, vesicles and plastids were observed in normal light intensity.

在正常光照下的伴胞具有致密的细胞质，内含丰富的线粒体、内质网、多泡体、囊泡和质体，而弱光下的伴胞明显液泡化，同时含有少量的线粒体和内质网。

Thus, we can conclude that the persistent expression of actin during late stage of baculoviral infection inhibited the polyhedrin expression, and then the formation of polyhedra.

本研究的结果表明，晚期持续表达的外源肌动蛋白抑制了多角体基因的转录和表达，从而导致多角体不能正常产生。

It can be concluded that the persistently expression of actin during late stage of baculovirus infection inhibit the polyhedrin expression, and then the formation of polyhedra.

结果表明，晚期表达的外源肌动蛋白抑制了多角体基因的转录和表达，从而导致多角体不能正常产生。

GFAP overexpessed but NOS neuron decreased, distorted and pale. The glia in SM-DB complex of ONR hypertrophied, but NOS neuron no degeneration. The increased GFAP immunoreactivity and the morphometric and cytological changes in SM-DB complex astrocytes may reflect a sustained upregulation of cellular activity with the insults of neurons, resulting in hypertrophy of glial perikarya and cell processes.

老年减退组星形胶质细胞的细胞体密度较老年正常组和青年组明显增加（P 在GFAP免疫细胞化学和NOS组织化学双重染色的切片上，老年减退大鼠SM-DB星形胶质细胞明显肥大，突起粗壮弯曲，GFAP超量表达，而NOS阳性神经元数量减少，胞体变形，突起减少，染色深度下降。

When cultured on the medium with different auxin , root segments could elongate and branch, and the addition of 1mg/l IBA into the medium was the most suitable for long-term root culture. Calli could be induced from root explants in the presence of 2, 4-D and when transferred onto E medium, they transformed further into embryonic calli which could develop into normal plants through the stages of globular-embryo, heart-shaped embryo, torpedo-shaped embryo, cotyledonary embryo.

培养基中添加生长激素（IAA，IBA，NAA）能够诱导无菌苗根切段的生长和分枝形成；而1mg／l IBA的存在，最适合于根的长期离体培养。2，4-D的存在能够诱导根外植体脱分化形成愈伤组织，愈伤组织转移至E型分化培养基能够转变成胚性愈伤组织，并进一步经球形胚、心形胚、鱼雷形胚、子叶形胚发育成正常植株。

Results ①Of 82% OSAHS patients' sleep indices were normal after the oral appliance therapy;②Compared to the control group,the patients with OSAHS had narrower upper-posterior airway volume and shorter distance from tip of the uvula to the posterior pharyngeal wall and lower positioned hyoid bone and shorter tongue before treatment;③Compared to before treatment,the OSAHS patients after therapy had lower positioned and shorter and flatter tongue,the upper hyoid bone,more anterior and downward mandibular and wider upper-posterior airway volume.

结果 ①82%患者治疗后睡眠指标达正常标准；②与配对组相比OSAHS患者上气道后间隙及悬雍垂尖至咽后壁距较窄，舌骨至下颌平面距较大，舌较短；③戴入口腔矫治器后患者舌体下降，舌变短而平坦，舌骨上移，下颌骨前下方移位，上气道后间隙扩大，与配对组比较，除舌体下移外其它测量项目值差异无显著性。

Results ①Of 82% OSAHS patients' sleep indices were normal after the oral appliance therapy;②Compared to the control group,the patients with OSAHS had narrower upper-posterior airway volume and shorter distance from tip of the uvula to the posterior pharyngeal wall and lower positioned hyoid bone and shorter tongue before treatment;③Compared to before treatment,the OSAHS patients after therapy had lower positioned and shorter and flatter tongue,the upper hyoid bone,more anterior and downward mandibular and wider upper-posterior airway volume.In comparison to control subjects,the indices were almost the same except for the lower positioned tongue.

结果 ①82%患者治疗后睡眠指标达正常标准；②与配对组相比OSAHS患者上气道后间隙及悬雍垂尖至咽后壁距较窄，舌骨至下颌平面距较大，舌较短；③戴入口腔矫治器后患者舌体下降，舌变短而平坦，舌骨上移，下颌骨前下方移位，上气道后间隙扩大，与配对组比较，除舌体下移外其它测量项目值差异无显著性。

The animal model of this method system have, the characteristics of the high fat, high leptin, high Ins, and match with the characteristic of clinical and the pathologic of simple obesity. This studies showed: The acupuncture can obviously reduce the vaule of intaking food and water of the fat rats, and lower the body weight and fat; The acupuncture can urge the diameter, area, physical volume of white fat cell contract, Brown fat tissue of fat rat group surroundings vascular containing large quantity of white fat cell, the acupuncture group was less. this showed: The acupuncture enhanced the white fat cell metabolism, and promotesed the white fat cell to convert to brown fat cell; The frequency of spontaneous electric of nerve cell of pvn of the fat rat is high, the acupuncture counld depress the spontaneous electric of nerve cell of pvn, This showed: the acupuncture can depress the appetite passing to adjust function of pvn nerve; The monoaminergic neurotransmitter of inside of the fat rat brain is disfunction, the acupuncture can rectify this disfunction, and adjust level of 5-HT, and affect appetite, and improve metabolism; The level of leptin and Ins of fat rat increased high, The level of it of acupuncture group decreased, compared with the fat group p.001, This showed: the fat rats have the defeat of leptin and Ins in the body, The acupuncture can lowering this defeat, The level of leptin and Ins of the fat rat in the brain is less than the normal group, The level of leptin of the acupuncture group is high than the fat group, This showed: the acupuncture can increase the transportation of leptin from blood to brain.

本方法所制肥胖大鼠模型具有高体重、高脂、高leptin和高Ins血症的特点，符合人类单纯性肥胖病的临床和病理特征；实验研究显示：针灸能明显减少肥胖大鼠的摄水、摄食量及降低其体重、体脂；针灸能促使白色脂肪细胞直径、面积、体积缩小，减少肥胖大鼠棕色脂肪组织血管周围白色脂肪细胞数量，显示：针灸增强了白色脂肪细胞的代谢，促进了白色脂肪细胞向棕色脂肪细胞的转化：肥胖大鼠PVN神经细胞自发放电频率增加，脑内单胺递质紊乱，针灸通过抑制PVN亢奋的自发放电，调整5-HT水平，达到影响食欲，改善代谢的作用；肥胖大鼠血中leptin及Ins水平增高，与正常组大鼠比有明显差异，提示大鼠体内存在leptin及Ins抵抗，针灸后肥胖大鼠随着体重体脂的下降，血中leptin、Ins水平降低，与肥胖组比均有显著性差异，而针灸后大鼠脑内leptin增加，与肥胖组比P.05，提示：针灸具有改善leptin、Ins抵抗及增加leptin脑内转运的作用。

They weren't aggressive, but I yelled and threw a rock in their direction to get them off the trail and away from me, just in case.

他们没有侵略性，但我大喊，并在他们的方向扔石头让他们过的线索，远离我，以防万一。

In slot 2 in your bag put wrapping paper, quantity does not matter in this case.

在你的书包里槽2把包装纸、数量无关紧要。