梗塞的

Angiostenosis,hypertension,diabetes, lipid and fibrinogen are risk factors for progressing stroke .

血管狭窄、高血压、糖尿病、高血脂及高纤维蛋白原血症是进展性脑梗塞患者的危险因素。

Angiostenosis,hypertension,diabetes, lipid and fibrinogen are risk factors for progressing stroke .

血管狭窄、高血压、糖尿并高血脂及高纤维蛋白原血症是进展性脑梗塞患者的危险因素。

In clinical and experimental studies, it has been found that cerebral ischemia and cerebral infarction were easy induced by hypotension in elder, hypertensive cerebral arteriolosclerosis, and stenosis or occlusion in carotid artery or in major cerbral arteries, butnow the effect machanisms still remain unclear.

从临床研究和动物实验也发现，在年老、高血压脑动脉硬化和颈动脉或大的脑动脉狭窄或闭塞时，低血压易诱发脑缺血和脑梗塞，但是目前其作用机制还未完全阐明。

They weren′t only the pathologic basis of arthrosclerosis, but the important factors in prethrombosis. So they promoted the occurrence of cardiac and cerebral infarction.

心肌梗塞和脑梗塞时除TG和apoB增高，HDL-C减低外，Lp、PLT及M-PKC增高，在血栓早期起重要作用，对心梗和脑梗的发病起促进作用。

Results Asymptomatic brain infarcts were present in 145 persons (7.2%).

结果：无症状的脑梗塞有145名(7.2%)。

Curative effect is distinct and avirulent deputy reaction, it is safety of block of head of a kind of cure, effective remedy, worth while and clinical promotion is used.

疗效显著且无毒副反应，是一种治疗脑梗塞平安，有效的药物，值得临床推广使用。

Summary: OBJECTIVE: to study the mechanism of cognitive function decreased after cerebellar infarction .

目的：分析小脑梗塞出现认知功能障碍的机制。

Free radical reaction was enhanced and the enzyme system as scanner was consumpted because of acute ischemia and reperfusion injury after CI.

脑梗塞由于急性缺血缺氧和再灌流损伤，使体内自由基产生连锁反应而进一步增加，清除自由基的酶系统进一步消耗。

RESULTS: The results of immunohistochemistry showed that ephrin-A5 distributed in the region of V2MM, V2ML, V2L, Au1, AuV in damaged cerebral cortex, and located in the body of neuron, cytodendrite, and somewhat in axon.

结果：免疫组化结果显示：大脑中动脉皮层支梗塞后ephrin-A5主要表达于梗死灶侧皮层的V2MM、V2ML、V2L、Au1、AuV，分布在神经元胞体、树突中，轴突亦有少量分布。

The immunohistochemistry method was used to detect the distribution and location of ephrin-A5, and the change of expression was detected at 1 week, 2 weeks and 4 weeks after operation.RESULTS: The results of immunohistochemistry showed that ephrin-A5 distributed in the region of V2MM, V2ML, V2L, Au1, AuV in damaged cerebral cortex, and located in the body of neuron, cytodendrite, and somewhat in axon.

结果： 免疫组化结果显示：大脑中动脉皮层支梗塞后ephrin-A5主要表达于梗死灶侧皮层的V2MM、V2ML（第二视皮质内侧外区、V2L、Au1、AuV，分布在神经元胞体、树突中，轴突亦有少量分布。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。