梗塞

ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

结果脑康灵胶囊可降低大鼠脑缺血/再灌注后神经病学评分及梗塞范围，降低脑组织MDA，NOS含量及升高SOD活性。

Methods The rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

采用大鼠大脑中动脉缺血2 h/再灌注22 h模型，神经病学评分，脑梗塞范围及脑组织水含量变化，观察脑康灵胶囊抗脑缺血/再灌注损伤的效应；通过测定大鼠脑组织中一氧化氮合酶（nitric oxide synthase，NOS），超氧化物歧化酶（superoxide disumtase，SOD）活性及丙二醛（malondialdehyde，MDA）含量的变化以探讨药物作用的机制。

MethodsThe rat model of middle cerebral artery ischemia 2h/reperfusion 22h was established.The neurological scale, cerebral infracted volume, cerebral water content, activities of NOS and SOD were measured. ResultsThe average neurological scote,cerebral infracted volume,cerebral water content,activity of NOS,content of MDA in Naokangling Capsule group significantly decreased.

方法采用大鼠大脑中动脉缺血2 h/再灌注22 h模型，神经病学评分，脑梗塞范围及脑组织水含量变化，观察脑康灵胶囊抗脑缺血/再灌注损伤的效应；通过测定大鼠脑组织中一氧化氮合酶（nitric oxide synthase，NOS），超氧化物歧化酶（superoxide disumtase，SOD）活性及丙二醛（malondialdehyde，MDA）含量的变化以探讨药物作用的机制。

Infarction it have shve sheet wedge shape anomalous low density screenage.

梗塞型：表现为脑实质内片状低密度区，呈楔形或不规则形。

Results All of the ischemic lesions were detected on superacute DWI scans.

结果 DWI对识别超急性期脑梗塞缺血损伤达 10 0 %。

Objective To confirm the diagnostic value of DWI in superacute cerebral infarction and analyze the prognostic value of the volume of ischemic lesions on superacute DWI scans.

目的 明确弥散加权成像对超急性期脑梗塞的诊断价值，探讨DWI缺血损伤体积与预后的关系。

However, cysts in the suprasellar cistern, cerebellopontine angle, and sylvian cistern may cause cerebral arteritis with subsequent infarction of the middle cerebral or posterior cerebral distribution.

而位于鞍上池、桥小脑角、以及大脑外侧裂池的囊虫病可以引起大脑动脉炎伴随着继发性的大脑中动脉和后动脉梗塞。

The complications included one greater trochanteric fracture,3 cases with pneumonitis,2 urinary infection,1 cerebral infarction,1 dysfunction in central nerve system and 1 case with deep venous thrombosis.

术后X线假体稍内翻2例，术后合并肺部感染3例，泌尿系感染2例，脑梗塞1例，中枢神经系统功能障碍1例，合并下肢深静脉血栓形成1例。

After administration of L-N〓-nitro-Arginine, a specific antagonist of NOS, it was found that the basal and stimulated NO release of blood vessels (aorta, internal carotid, renal and mensenteric artery) was decreased further. The increase of coronary blood flow stimulated by Ach was also reduced further, while the platelet aggregation degree and erythrocyte aggregation index increased significantly. Pathological examination indicated that aortic endothelium was destroyed, the blood vessels in cerebral, cardiac and renal tissues were hypertrophied further and some were almost occluded. The above fin dings resulted in SHR feeded with L-NNA having high incidence of stroke rate and high degree of renal cirrhosis when compared to control SHR. The infarct and hemorrhagic focuci in cerebral tissues, infarct focuci in cardiac tissues, atrophy and hyalinedegeneration of renal glomeruli and degeneration even necrosis of renal tubuli in renal tissues were al so found. The increase ratio of brain and heart to body weight and decrease ratio of kidney to body wei ght further demonstrated that the cerebral, cardiac and renal injuries were aggravated.

在上述结果的基础上进一步给予SHR NOS特异性拮抗剂左旋亚硝基精氨酸后，血管(胸主、颈内、肾及肠系膜动脉)NO基础释放及激动剂刺激释放进一步减少，Ach刺激冠脉流量的增加和胸主动脉内膜NOS活性也进一步减少，血小板聚集程度及红细胞聚集指数显著上升，病理切片显示胸主动脉内皮细胞破坏明显，脑心肾组织内的血管进一步增厚甚至几乎阻塞，导致饲L-NNA的SHR缺血性及出血性脑卒中发生率显著高于对照SHR，脑体重比显著升高；心肌内出现明显的缺血梗塞疤痕灶，心体重比显著升高；肾小球明显萎缩、玻璃样变性，肾小管也变性甚至坏死，使肾硬化程度显著增加呈颗粒性固缩肾，肾体重比也显著减少。

Baekgroud: Spinal cord ischemia reperfusion injury is an serious secondary spinal cord injury. Trauma, arteriosclerosis, vasal abnormality, inflammation, vasal infarct, decompression after the diseases that can bring spinal cord compression, lessening the blood stream of the spinal cord temporarily ,because of clamping the aorta during the operation of aorta, all of which can engender different extent spinal cord ischemia.

背景：脊髓缺血再灌注损伤是严重的脊髓继发性损伤，创伤、动脉硬化、血管畸形、炎症、血管梗塞、各种产生脊髓压迫的疾病减压后以及在主动脉手术中，钳夹主动脉导致脊髓血液供应暂时性减少都可造成不同程度的脊髓缺血再灌注损伤。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。