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梗塞

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Objective: To study the relationship between the type of acute infraction and sleep apnea hypopnea syndrome.

目的 :探讨急性脑梗塞患者的梗塞类型与睡眠呼吸暂停低通气综合症之间的关系。

Cerebral infarction is one of main complications of diabetes, and one of main lethal and maimed reasons impatient...

梗塞是糖尿病的主要并发症之一,也是糖尿病致死致残的主要原因之一,同年龄糖尿病患者发生脑梗塞的机率明显高于非糖尿病患者。

At 6h after ischemia both MRI were found to indicate definite infarct foci in the caudate nucleus and the lentiform nucleus,mass effect was seen too.Histopathologic findings:At 2h after ischemia,electro microscope revealed the changes of ischemia and edema.At 4h after ischemia,the photomicroscope demonstrated the changes of ischemia edema and the electromicroscope indicated the damage to blood brain barrier.

结果 梗塞2h MRI T2加权可见尾状核头部、豆状核信号增高;6h MRI可见尾状核、豆状核形成明确梗塞,并出现占位征象。2h电镜下已有缺血水肿改变,4h光镜下出现脑缺血水肿改变,电镜下见到血脑屏障受损。

Results The clinical manifestation of brain stem hemorrhage secondary to cerebral infarction included sudden onset,evelution abruptly,coma,quadriplegia,higher fever and respiratory failure. CT usually do not identify the high dense location of brain stem. Large infarction of cerebral hemisphere may lead to shift the brain laterally and downward to produce herniation with subsequent brain stem compression,shift and distortion. Secondary brain stem hemorrhage occured usually in midderline area of midbrain or pontine.

结果 脑梗塞继发脑干出血的临床特点是发病急、进展快、昏迷、四肢瘫、过高热及呼吸衰竭,CT一般不能发现脑干高密度病灶,病理结果均为半球大病灶脑梗塞,严重海马回疝致脑干严重受压、变形和移位,继发脑干出血以中脑为主,可累及桥脑,主要位于中线部位。

Above resalts preved protection edffects on ischemic brain tissue by scalp pomt through point acupuncture with EA acering acate stase and reperfusion stage.

由此说明在脑梗塞的急性期及再灌注期,配合针刺方法对急性脑组织缺血具有保护作用,为针刺治疗急性脑梗塞提供了科学的依据。

The results demonstrate that hypertensive cerebral arteriolosclerosis is the pathologic base which cerebral infarction is easy induced by hypotension, and thereis a close relationship between the occur of cerebral infarction and hypertensive degrees and duration in hypotension condition.

表明高血压性脑内小血管病变是低血压容易诱发脑梗塞的病理基础,说明低血压时,脑梗塞的产生与患者高血压程度的高低及持续时间的长短有密切的关系。

It suggests that hypertensive left ventricular hypertrophy and hypertensive arteriolosclerosis in heart and brain may be the same pathological base which causes myocardial infarction complicating with cerebral infarction, and that hypotension is an important inducing factor causing myocardial infarction complicating with cerebral infarction.

提示高血压所致的左心室肥大和心脑小血管的动脉硬化改变可能是心脑梗塞并发的共同病理基础,而低血压则是引起心脑梗塞并发的一个重要诱因。

This result demonstrates that the complicative rate between cerebral infarction and myocardic infarction in RHRSP is relatively high.

本实验发现在38只脑梗塞的RHRSP中有20只合并心肌梗塞,并发率高达52.6%,表明RHRSP的脑梗塞和心肌梗塞的并发率相当高。

It is concluded that biomolecular structure and chemical components are markedly changed in the infarcted cerebral cortex.

结果显示:脑梗塞后大鼠大脑皮质的生物大分子的结构及化学成分均出现明显的变化,与脑梗塞的病理变化结果密切相关。

Results:Infarcted area could not be differentiated from non-infarcted area on plain MR images.

结果:MRI平扫不能区别梗塞心肌和非梗塞心肌。

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