染色质的

It is also controlled by specific changes in chromatin structure in the vicinity of lineage-specific genes.

同时这一结果还受特定谱系基因附近的染色质结构的具体变化的控制。

In this study, we have explored the regulatory function of SATBl toβ-globin gene cluster at chromatin level.

我们的研究就要探讨SATB1在染色质水平上是如何调控β珠蛋白基因的表达的。

It is this that is visible as the typical looped threadlike chromatin material of the nondividing chromosome.

这就是在不分裂染色体中可见的典型的松弛的线状染色质。

Chromatin structure is a key control point in eukaryotic gene expression and DNA replication and DNA repair.

在真核细胞进DNA复制与修的过程中，染色质结构的改变扮演著重要的角色。

The results that the chromatin structure in ANF gene region for 0.5 month-old rats are in loose form while it is in close-packed form for 6 month-rats.

本项研究发现并肯定了发育及老化过程中ANF基因表达规律，老化时染色质构象的变化是ANF基因表达衰退的重要原因，发现ANF基因中三个CCGG序列完全甲基化可能是该基因表达"关闭"的原因。

Transmission electron microscope showed the evident damage of hypoxic cardiocyte ultrastructure after blocking HSP90: caryotheca crenation, chromatin margination and mitochondria vacuole.

(3)透射电镜显示阻断HSP90后的缺氧心肌细胞超微结构不但出现细胞肿胀，肌丝排列紊乱等细胞受损的表现，还可以看到核膜皱缩，染色质边集，线粒体出现空灶化等细胞凋亡的特征。

For TEM, there were classical chatacteristics of cell apoptosis, which was bridge structure disappeared, cell withered , chromosome aggregated and broken and there was bubbles in cell.

透 射电镜下可见桥粒结构消失，细胞萎缩，胞内出现大量的空泡，染色质边集，断裂等，出现凋亡的典型特征。

Results: Compared with control group, apoptosis cells increased from 0.5%to 10%(some even to 15%) after 24,48 and 72 h action of -300,-500 and -1 000 V electrets. After action of -500 V PTFE electrets for 48-72 h, fibroblast cells showed characteristic morphological features of apoptosis. These features included chromatin aggregation, nuclear and cytoplasmic condensation and partition of cytoplasm and nucleus into membrane bound-vesicles.

结果：-300、-500和-1 000 V驻极体作用成纤维细胞24、48和72 h以后，与对照组相比，成纤维细胞的凋亡量从0.5％增至10％（部分可达15％）；驻极体作用成纤维细胞48～72 h，出现细胞凋亡特有的形态学特征，即：细胞异染色质边集，细胞裂解，可见凋亡小体。

Covalent modification of histones on chromatin is a dynamic mechanism by which various nuclear processes are regulated.

染色质上组蛋白的共价修饰是一个动态的机制，多种细胞核过程通过该机制被调控。

Observations underelectron microscopy showed that YSV destroyed the structure of chromatin,damaged cell organelles of tumor cells, and induced apoptosis and necrosis intumor cells.

超微结构研究显示，YSV(320μg／kg／d)诱导人肝癌BEL-7402细胞的染色质溶解、固缩，细胞器的普遍崩解，细胞出现凋亡和坏死。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。