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The equipment is applicable for a variety of fabrics in pre-and post-treatment and dyeing process at a constant temperature.

本机适用于各种织物在常温条件下的前后处理及进行染色等工艺。

The curve of absorption is mirror symmetry of that of emission.

对荧光染料染色样进行了一系列的实验和测定。

Acridine orange dyeing was used to observe the proliferation of cell in collagen sponge at 20 and 35 days of culture.

培养20,35 d,对共培养物进行AO染色,观察细胞在胶原海绵中的增殖情况。

An increased glomerular staining with Peanut agglutinin lectin, indicative of removal of neuraminic acid, was noted.

实验中所观察到的肾小球花生凝集素染色增强标志着神经氨酸被清除。

As you are walking, you come across some tiles on the floor. They read "AHOY DYE FOOT."

行进间,你偶然发现地上的瓷砖组成了"喂,染色脚",多么奇怪。

ARID (AT-rich interaction domain) protein is a transcription factor family in higher eukaryotes that regulates cell proliferation, development, and differentiation. Specificity of DNA binding ability in this family prefers AT-rich sequences, but some ARID family proteins are not sequence-specific DNA-binding proteins or they do not bind AT-rich sequences. We found two genes that encode ARID in Giardia lamblia genome database, garid1 and garid2. We analyzed the function of garid1 first. AU1-tagged gARID1 was found to localize to nuclei. During encystation, gARID1 mRNA level decreased emphatically, but protein level increased. We also found that gARID1 can bind AT-rich initiator of the cwp1 promoter by EMSA. Mutation analysis revealed gARID1 binding sequence was AGATC and AATAAAATA. We used ChIP to demonstrate that gARID1 can bind cwp1 gene promoter in vivo.

ARID(AT-rich interaction domain)蛋白质家族是真核生物的一种转因子,在许多同种的真核生物有它的同源基因,这个家族的蛋白质通常与调控细胞的生长、发育和分化的作用有关,而这个家族的蛋白质和DNA的结合能,各种ARID蛋白质的专一性尽相同,过大致上偏好於和AT-rich的序结合;我们已经在形鞭毛虫的基因组中找到个含有ARID 的基因,分别是garid1和garid2,我们首先对於garid1做分析;将AU1标记接到gARID1转染形鞭毛虫,用免疫萤光染色可发现gARID1存在於细胞核中。gARID1的讯息RNA在囊体化后会明显下,过其阳性染色和蛋白质表现有明显增加;EMSA实验中也发现gARID1会明显的与cwp1基因启动子之AT-rich initiator结合,经由突变序分析,也显示gARID1的结合序为AGATC和AATAAAATA,随后我们也用ChIP证明gARID1在细胞内也的确会和cwp1基因的启动子结合。

Following studies had been performed:(1)repetitive WLT and real-time ultrasonography had been performed to 18 HS and 40 FD toassess the reproducibility and influencing factors.(2) correlations of WLT with symptomscore and electrogastrography had been analysed.(3) differences ofWLT between HS and FD had been compared.(4) methophenolane andimmunohistochemistry stain of gastric fundus mucosa biopsy from 40 FD patients hadbeen performed to analyse the correlations of symptom score and WLT with count andactivity of mast cells, 5-HT level, and H.Pylori infectious status.(5) the effect of oralsumatriptan to proximal stomach function in 10 HS and 10 FD had been assessed, so as toestablish the possible regulating mechanism of fundic relaxation, explore the therapeuticpotential and feasibility of sumatriptan on FD.Results:I. Evaluation of WLT and its application in FD management.

我们主要进行以下几个方面的研究:(1)对18名健康人及40名FD患者重复进行水负荷试验并结合B超声对近端胃的实时监测,评价水负荷试验结果的可靠性并分析其影响因素;(2)观察水负荷试验结果与症状积分及体表胃电结果的相关性;(3)比较FD患者和健康成人水负荷试验结果的差别;(4)对40名FD患者胃底粘膜活检组织进行免疫组化染色和甲苯胺蓝染色,观察FD患者的症状及水负荷试验结果与胃底粘膜肥大细胞、5-HT表达情况及H.pylori感染情况的相关性:(5)观察口服舒马曲坦对10名健康人及10名FD患者近端胃功能的影响,并由此推测胃底舒张运动调节的生理机制,探讨口服舒马曲坦治疗FD的可行性。

Cells were collected to undergo HE staining and immunocytochemistry reaction against Thy-1 to identify RGCs. Axons length of retinal neurons and RGCs were measured by microruler under microscope respectively to evaluate the neurotropic effects of B vitamins in improving the regeneration and growth of axons for retinal neurons and RGCs. Finally, we observed the reaction of retinal neurons when exposed to the insult of glucose deprivation. The neuroprotection of B vitamins were also evaluated at the same time.

对B族维生素的神经营养和保护作用的研究过程分为三个阶段,首先确定维生素B族对视网膜神经元是否有神经营养作用,并根据细胞活力找出最佳有效作用浓度;然后在接种细胞的即刻用各种维生素的最佳有效作用浓度作用于视网膜神经元,并在不同时间终止培养,收集细胞,HE染色和RGCs的免疫细胞化学染色,分别测量视网膜神经元和RGCs的轴突长度,评价维生素B族是否有促进视网膜神经元和RGCs轴突再生伸长的神经营养作用;最后,观察视网膜神经元对低糖损伤的反应,及维生素B族对受损的细胞是否有保护作用。

Objetive:To investigate the histopathological features and detect the mutation of JAK2V617F in PV、ET and IMF;to analyze the effects of the stages of myelofibrosis and the mutation on the number of leucocyte in PV and ET patients;Methods: the morphological features of 69 patients who dignosed in the first time were studied by HGF and Gomori stains ; the mutation of JAK2V617F was detected in 38 blood or marrow samples of PV、ET and IMF patients by real-time PCR;the effects of the stages of myelofibrosis and the mutation on leucocytes were analyzed;Results: Different stages of reticulin were observed in 15 PV and 15 ET patiens'marrow sections,and the cytosis、the pleomorphism and the bone trabecula side location of megakaryocytes were observed in all PV、ET and IMF patients;the means of leucocyte in patients with and without JAK2V617F were 20.4×109/Land 11.4×109/L respectively;the P value was 0.367 as comparing the leucocytes among different stages of reticulin increasing.

目的:探讨PV、ET、IMF患者的骨髓病理特点并且检测JAK2V617F突变的发生率,分析骨髓中纤维增生程度以及JAK2V617F对PV、ET患者白细胞数的影响;方法:69例初诊患者的骨髓切片经HGF染色以及Gomori染色后进行形态学研究;并利用real-time PCR法测38例患者的骨髓或者外周血的JAK2V617F突变;分析骨髓切片中纤维组织增生程度以及JAK2突变对患者白细胞数的影响;结果:15例PV和15例ET患者的骨髓切片中可以观察到不同程度的纤维组织增生,所有PV、ET、IMF患者骨髓病理中可见到巨核细胞增多、巨核细胞多形性改变以及巨核细胞定位于骨小梁旁;12例PV、4例ET、4例IMF中检测到JAK2V617F;有突变和无突变患者发病时白细胞数均值分别为20.4×109/L和11.4×109/L,比较不同纤维增生程度的PV、ET患者的白细胞数均值,P值为0.367。

The Dac model is easily described:choose a graph at random according to bond percolation,and then paint randomly and independently the different clusters,each cluster being monochromatic.We generalize the Dac model in the sense that the independent condition is weakened to uncorrelate.

第二章研究了Z~d上Bernoulli边渗流开簇的随机着色模型:按照Z~d上的边渗流机制随机的选择一个子图,然后给每个开簇上的点随机的染色,要保证这种不同的开簇上的染色行为是互不相关的,而且同一开簇上的点被染的颜色是相同的。

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The labia have now been sutured together almost completely.The drains and the Foley catheter come out at the top.

此刻阴唇已经几乎完全的缝在一起了,排除多余淤血体液的管子和Foley导管从顶端冒出来。

To get the business done, I suggest we split the difference in price.

为了做成这笔生意,我建议我们在价格上大家各让一半。

After an hour and no pup, look for continued contractions and arching of the back with no pup as a sign of trouble.

一个小时后,并没有任何的PUP ,寻找继续收缩和拱的背面没有任何的PUP作为一个注册的麻烦。