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CySeCD exhibits better GPX activity than 6-AnSeCD in the reduction of H2O2 and cumenyl hydroperoxide by glutathione, respectively.

采用双酶偶联法测定GPX的活力结果显示,6A,6A'-二环己胺-6B,6B'-二硒桥联-β-环糊精(6-CySeCD)催化谷胱甘肽还原H2O2和枯烯H2O2的活力均比6-AnSeCD的高。

In present study, levels of dopamine 〓 and 〓 receptors and expression of the protein mRNAs in striatum were exarmined by the methods of immuno- histochemistry and in situ hybridization, respectively. It was found that mice treated by paraquat taken orally for four months displayed a marked hypoactive behavior.

本研究以口服给予小鼠百草枯的PD模型,采用免疫组织化学、原位杂交结合计算机图像处理分析技术,观察百草枯对纹状体区多巴胺〓受体、多巴胺〓受体蛋白含量和基因表达的影响。

Paraquat, bearing structural simility to the known dopaminergic neurotoxicant MPTP, has been suggested as a potential etiologic factor in PD.

除草剂—百草枯(paraquat,PQ)与多巴胺能神经毒物MPTP结构相似,具有与MPTP相似的神经毒性作用。

The bioassay indicated that some of them possess certain fungicidal activity against Botrytis cinerea, Gibberlla zeae, Rhizoctonia solani, Venturia nashicola at a dose of 50 μg/mL. The EC50 value of title compounds B-8、B-9、B-10、B-12 was reached 1.0, 0.2, 1.2 and 0.4 μg/mL against B. cinerea, the effect was approximated or better than pyrimethail and EC50 value of title compounds B-8, B-10, B-11 was reached 0.07, 0.2 and 0.05 μg/mL against G. zeae, the effect was better than carbendazim.

在50μg/mL浓度时,该系列化合物对番茄灰霉病菌Botrytis cinerea、小麦赤霉病菌Gibberlla zeae、水稻纹枯病菌Rhizoctoniasolani、梨黑星病菌Venturia nashicola菌丝生长均具有很好的抑制活性,其中化合物B-8、B-9、B-10、B-12对番茄灰霉病菌的EC50值分别为1.0、0.2、1.2、0.4μg/mL,接近或优于对照药剂嘧霉胺,B-8、B-10、B-11对小麦赤霉病菌的EC50值分别为0.07、0.2、0.05μg/mL,优于对照药剂多菌灵。

Objective To study the possible mechanism of glutamine to manipulate the immune function of rat Kupffer cell in vitr o.

目的 探讨谷氨酰胺调节大鼠枯否细胞免疫功能的可能机制。

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This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。

There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.

双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。