松弛肽

That means: no animal products, lots of veggies and fruits and of course lots of exercise to raise those endorphins and tone flab.

这意味着：没有动物产品，蔬菜，水果和工作中的许多地段，提高这些内啡肽和语调松弛。

Many literatures focus on preterm prediction study and evaluate every predictor. The predictors include cervical length and the size of some of fetal organs measured by ultrasound; the level of fFN, IGFBP-1, IL investigated in cervicovaginal fluid, serum relaxin, CRP and HbA1c in mother's blood, MMP-8, MCP-1 and ITAC in amniotic fluid which correlated with infection.

早产的预测是研究热点，大量文献分别从超声测量学角度，包括宫颈长度，胎儿某内脏的测量；宫颈阴道分泌物中多项指标，如胎儿纤维结合蛋白、磷酸化及脱磷酸化胰岛素样生长因子连接蛋白-1、白细胞介素的测定；血生化分析，如血清松弛肽、C反应蛋白、糖化血红蛋白含量的变化及羊水化验与感染有关的一些因子，如快速基质金属蛋白酶8、单核细胞趋化蛋白-1、γ干扰素诱导T细胞α趋化因子等，阐述各指标对早产的预测价值。

rutaecarpine pharmacological effects and its related mechanism: rutaecarpine the heart positive inotropic, positive changes in the role and the heart protective effect, which with its promotion of calcitonin gene-related peptide release; rutaecarpine alkali have vasodilative and hypotensive effect, its mechanism mainly related to endothelial cells Ca2 +-NO-cGMP pathways and calcitonin gene-related peptide release; rutaecarpine to relax the anal sphincter, gastrointestinal motility inhibition of stress ulcer and ulcerative colitis have therapeutic effect; rutaecarpine on cerebral ischemia-reperfusion injury has a protective effect, which instead of increasing the concentration of calcitonin gene-related peptide-related; rutaecarpine inhibits platelet aggregation and arterial thrombosis; In addition, rutaecarpine Alkali also anti-inflammatory and analgesic, anti-tumor and other pharmacological effects.

综述了吴茱萸次碱的药理作用及其相关机制：吴茱萸次碱对心脏有正性肌力、正性变时作用和心脏保护作用，这与其促进降钙素基因相关肽释放有关；吴茱萸次碱有扩血管和降压作用，其机制主要涉及内皮细胞Ca2+-NO-cGMP途径和降钙素基因相关肽的释放；吴茱萸次碱能松弛肛门内括约肌，抑制胃肠运动，对应激性溃疡和溃疡性结肠炎也有治疗作用；吴茱萸次碱对脑缺血再灌注损伤有保护作用，这与其增加降钙素基因相关肽浓度有关；吴茱萸次碱能抑制血小板聚集和动脉血栓形成；此外，吴茱萸次碱还有抗炎镇痛、抗肿瘤等药理作用。

It has been suggested that the hormone relaxin, a hormone of pregnancy, causes this joint laxity in pregnant women.

曾有人提出，一种叫松弛肽的孕期激素引起怀孕妇女关节松弛。

Howeer, recent studies hae found that increased peripheral joint laxity in pregnant women was not associated with increased serum relaxin leels or estradiol leels.

然而，最近的研究发现，怀孕妇女外周关节松弛度增加与血清松弛肽或雌二醇水平升高之间没有联系。

We show that the matrix-modifying hormone relaxin increased tumor-associated fibroblast interaction with collagen fibers by stimulating b1-integrin activity, which is necessary for fiber remodeling by matrix metalloproteinases.

我们指出基质修改激素松弛肽通过分泌活性b1-integrin ，从而增加了瘤相关纤维原细胞与胶原纤维的相互作用。这对于基质金属蛋白酶对纤维的重塑很重要。

Because relaxin leels hae been increased in women using oral contracepties, it is also suggested that relaxin may proide the missing key for understanding the association between oral contraceptie use and LBP.

由于使用口服避孕药的妇女其松弛肽水平也会增高，故松弛肽可能是理解口服避孕药使用与LBP之间联系的关键点。

Although the etiology and pathophysiology of LBP during pregnancy are poorly understood, one plausible hypothesis is that causes of LBP in pregnancy are multifactorial (i.e., biomechanical stress on ligaments and joints, muscle fatigue, and the effect of the hormone relaxin on joint laxity), with ≥1 factors dominating in an indiidual case.

尽管对孕期LBP的病因学及病理生理学了解甚少，有一个似乎可信的假说宣称：孕期LBP是多因素引起的（施加在韧带和关节的生物力学上的应力、肌肉疲劳及松弛肽的关节松弛作用），对单个对象来说，有不止一个因素交互作用。

Pregnancy, oral contraceptie use, and use of estrogens during menopause may result in higher estrogen leels, resulting in an increased laxity of joints and ligaments through increased relaxin, resulting in chronic LBP, but not chronic UEP.

既往怀孕史、使用口服避孕药及绝经期使用雌激素等因素可使雌激素水平升高，进一步提高松弛肽水平，使关节和韧带松弛度增加，产生慢性LBP症状，但不会产生慢性UEP症状。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。