松弛

In chapter three, on the basis of non-steady tire theoretical model, the definition equation of relaxation length is get by expanding E function in series of s and truncating the higher order.

第三章，在轮胎非稳态侧偏特性理论模型的基础上，通过对E函数进行低阶近似，得出了侧偏松弛长度的表达式，给出了侧偏松弛长度随侧偏角变化关系。

OBJECTIVE: To compare the rheological properties of stress relaxation in normal and atherosclerotic animal model of ascending aorta, and to identify the effects of atherosclerosis on the stress relaxation properties of ascending aorta.

目的：比较正常和动脉粥样硬化动物模型升主动脉的应力松弛流变特性，确定动脉粥样硬化对升主动脉应力松弛特性的影响。

This research is to develop a set of mechanical model which can simulate heel pad effectively, and perform the stress relaxation experiment to get the coefficients of constitutive law using the optimization method.

本研究主要建立一组可以有效模拟出后足部软组织之机械特性的数学模型，并且藉由体外量测后足部的应力松弛实验，经由最佳化的方式求出与应力松弛实验结果相符的组成律系数值。

Stress relaxation of delignified and hemicellulose-removed wood was determined during periodic temperature change, and found that after 4-hour relaxation at 25℃ the stress curves hardly level off, at this time when increase temperature the stress immediately sharply fall and whereafter the decrease of temperature make stress rapidly recover again.

通过木材在温度周期变化过程中的应力松弛研究，发现，未处理木材和化学处理木材在25℃松弛4小时后，应力几乎水平下来，升温后，应力立即急剧下降，温度下降时，应力又急剧回复。

Pregnant bodies produce a hormone, relaxin, which softens tissues around the pelvis to prepare it for birth.

怀孕的身体产生荷尔蒙、松弛素，松弛素使骨盆周围的组织变软，使骨盆为生产做好准备。

It has been suggested that the hormone relaxin, a hormone of pregnancy, causes this joint laxity in pregnant women.

曾有人提出，一种叫松弛肽的孕期激素引起怀孕妇女关节松弛。

Howeer, recent studies hae found that increased peripheral joint laxity in pregnant women was not associated with increased serum relaxin leels or estradiol leels.

然而，最近的研究发现，怀孕妇女外周关节松弛度增加与血清松弛肽或雌二醇水平升高之间没有联系。

Because relaxin leels hae been increased in women using oral contracepties, it is also suggested that relaxin may proide the missing key for understanding the association between oral contraceptie use and LBP.

由于使用口服避孕药的妇女其松弛肽水平也会增高，故松弛肽可能是理解口服避孕药使用与LBP之间联系的关键点。

Although the etiology and pathophysiology of LBP during pregnancy are poorly understood, one plausible hypothesis is that causes of LBP in pregnancy are multifactorial (i.e., biomechanical stress on ligaments and joints, muscle fatigue, and the effect of the hormone relaxin on joint laxity), with ≥1 factors dominating in an indiidual case.

尽管对孕期LBP的病因学及病理生理学了解甚少，有一个似乎可信的假说宣称：孕期LBP是多因素引起的（施加在韧带和关节的生物力学上的应力、肌肉疲劳及松弛肽的关节松弛作用），对单个对象来说，有不止一个因素交互作用。

Pregnancy, oral contraceptie use, and use of estrogens during menopause may result in higher estrogen leels, resulting in an increased laxity of joints and ligaments through increased relaxin, resulting in chronic LBP, but not chronic UEP.

既往怀孕史、使用口服避孕药及绝经期使用雌激素等因素可使雌激素水平升高，进一步提高松弛肽水平，使关节和韧带松弛度增加，产生慢性LBP症状，但不会产生慢性UEP症状。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。