机体
- 与 机体 相关的网络例句 [注:此内容来源于网络,仅供参考]
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Antitrypsin (α1-AT) is the major proteinase inhibitor and takes part in many metabolic processes in human bo...
1抗胰蛋白酶(α1-AT)是人体内主要的蛋白酶抑制剂,它参与机体的多种代谢过程。
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Methods After treating 147 teeth which diagnosed as aschronic apicitis were suitable for root canal therapy,divided at random into three groups:0.5% Anerdian treatment group,FC group and dry cotton spill to seal the root canal of them.
结果 安尔碘作为根管消毒药物治疗根尖周疾病效果与FC相比差异无显著性,而FC对机体组织有一定刺激性和细胞毒性,且部分患者会过敏。
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In short, their common purposes are diminishing inflammation and apocatastasis, relieving pain, strengthening organic integral ability and improving joint function.
共同目的是消炎、止痛、消肿、增强机体整体能力和关节功能改善。
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The center of adjusting the appetite in the body activities lies in the nervous system,the root cause of apositia is controlling disorder of the central nervous system,this sdudy intends to establish an animal model of apositia with the function of central inhibition according to the cause of disease to give some reference for pharmacological research of apositia.
因机体调节食欲的中枢在神经系统,厌食的根本原因在于中枢神经系统调控失常,故本研究拟建立符合致病原因的中枢抑制性厌食症动物模型,为厌食症的药理学研究提供参考依据。瘦素是肥胖基因的产物,是白色脂肪细胞分泌的一种由167个氨基酸残基组成的蛋白质。
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The good regulating effect of acupuncture on the function of arcuate nucleus of hypothalamus might be one of mechanisms in the antiobesity of acupuncture.
针刺对肥胖机体下丘脑弓状核功能的良性作用可能是针灸减肥的作用机制之一。
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Traditionally, it was divided into acute and chronic arseniasis, which were decided mainly by exposing dose and time from exposure to attack.
传统砷中毒的概念分急性砷中毒和慢性砷中毒,它们主要是由于机体单位时间接触砷的量和从接触到发病的时间长短来决定。
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Small artery and arteriole are the main position of organism to regulate the organ blood flow and the redistribution of blood among apparatus.
小动脉和微动脉是机体调节器官血流量和器官之间血液重新分配的主要部位,其血管的口径受神经体液因素的调节。
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"This may mean you could develop therapeutic strategies to manipulate fat metabolism (the rate at which food is turned into energy) independently of what you eat," says study co-author Kaveh Ashrafi,
加州大学的科学家在《细胞代谢》杂志上报道,这种神经信使即食欲抑制因子,它不仅控制着你能吃下多少,而且还相应地在机体如何消耗卡路里中发挥作用。
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Results: The changes following hydrocortisone intervention of rats could be identified by PCA, and the kidney deficiency syndrome rats entered a state of "hyperfunction", involving a series of changes in metabolic pathway and metabolic network. The response integral area of lactic acid (δ 1.37) increased, indicating the accumulation of metabolites of lactic acid. The amount of dimethylamine (δ 2.72) also increased, indicating that the disorder of kidney function was related to kidney-deficiency syndrome. The response of creatinine (δ 3.87), asparaginic acid (δ 2.83), taurine (δ 3.44, 3.28), and hippurate (δ 7.84, 7.56, 7.64, 3.97) all decreased significantly, indicating that the hydrocortisone might cause adrenal cortex excretion injury. The response integral area of succinate (δ 2.41) and citric acid (δ 2.53, 2.68) decreased, which was usually caused by disorder of the mitochondrial function.
结果:PCA方法处理肾虚证和空白对照大鼠尿液数据显示,两组大鼠的数据可以在得分图实现分类,与对照组比较肾虚组大鼠由于氢化可的松诱导,大鼠机体进入一个过消耗后衰弱的状态,相关代谢发生显著变化,乳酸(δ 1.37)代谢发生堆积;二甲胺(δ 2.72)的含量增加,提示肾虚与肾功能异常是密切相关的;天冬氨酸(δ 2.83),牛磺酸(δ 3.44, 3.25),马尿酸(δ 7.84, 7.56, 7.64, 3.97),肌氨酸(δ 3.87)等的相对积分面积明显下降,这预示糖皮质激素可能引起了肾上腺皮质分泌功能的损害;琥珀酸(δ 2.41)和柠檬酸(δ 2.53, 2.68)是三羧酸循环能量代谢及糖酵解的中间产物,其含量的降低,通常是由于线粒体功能紊乱所引起。
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Recently,numerous studies proved that each member of secreted aspartyl proteinase have different requirements in temperature and pH,with develop of gene mutation technology further cleared relation between gene expression regulation and organism′s infection sites and state,as well as transcription activation factor effect on expression of each member of secreted aspartyl proteinase.
近年来,大量研究证实了分泌型天冬氨酸蛋白酶各成员对温度、pH值等培养方面有不同的要求,基因突变技术的发展,进一步明确了基因表达调控与机体感染部位、感染状态之间的关系及转录活化因子在蛋白酶各成员表达中的作用。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。