有肠的

objective:to investigate the therapeutic action of somatostatin in pancreatic leakage after pancreaticoduodenectomy.methods: to collect 100 patients with pancreaticoduodenectomy during 2003～2006,contrast 44 patients use somatostatin after pancreaticoduodenectomy as experimental group,and 56 patients with conventional therapy as control group.results:there were no pancreatic leakage case in the modified group of 44 cases.3 cases complicating pancreatic leakage occurd in control group,and the proportion of pancreatic leakage are different examined by statistic mathord.conclusions:use somatostatin after pd can restrain organism excrete diastase vera,and help anastomotic stoma to concrescent and decrease pancreatic leakage;pancreatic leakage should be treated by different grades,grade 1 can be cured by persistent drainage with somatostatin and nutritional support;grade 2 should be pay more energetic intervention.

作者单位：潍坊医学院，潍坊 261042；潍坊市人民医院肝胆外科，潍坊 261042 目的：探讨生长抑素在胰十二指肠切除术后减少胰漏的作用。方法：收集2003年1月～2006年12月施行pd患者100例，术后应用生长抑素持续泵入病人44例，并与同期施行的pd术后常规应用全肠外营养及抗生素治疗病人56例，结果进行了对比观察。结果：实验组病人44例无1例发生胰漏，对照组56例病人，3例发生不同程度的胰漏，发生率5.3%，统计学比较有显著差异（p＜0.05）。结论： pd后应用生长抑素持续泵入可以抑制胰酶的作用，使胰肠吻合口较好地愈合，从而有效地减少胰漏的发生；胰漏可进行分级治疗，ⅰ级胰漏采用通畅引流为主，同时使用生长抑素和营养支持等治疗多可治愈，ⅱ级胰漏需要积极治疗。

The effect on gastric secretion in rats was studied with pylorus ligation. The analgesic effect was tested by the hot - plate test and writhing method.

结果：舒肝丸能抑制正常小鼠胃排空及新斯的明所致的胃排空和肠推进亢进，对阿托品所致的胃排空、肠推进迟缓有协同作用，但不明显；能抑制大鼠胃酸及胃蛋白酶的分泌，促进胃粘液的分泌；可提高小鼠的热痛阈，减少小鼠的扭体次数。

The results of experiment indicate, 1 OOmg/kg GLJ have no notable influence on normal or sthenic ileum of anesthetic rabbits.1.3 Observe the action of GLJ on the sthenic vermiculation of ileum of SD rats due to Neostigmine, using the pressure sensor attached to the ileum.

1.3采用贴壁压敏传感器的方法观察GLJ对麻醉SD系大鼠回肠蠕动的影响，结果表明，100mg╱kgGLJ对新斯的明所致大鼠回肠蠕动亢进有显著抑制作用。

Previous studies have revealed that aganglionic or morphological and numerical abnormalities of the intestinal intramural ganglia may contribute to the pathogenesis of the intestine motility obstacle of them.

目前认为HD和HAD肠动力障碍的发病机制主要是病变肠段粘膜下神经丛和肌间神经丛的神经节细胞的缺失和减少，病变肠壁内的副交感、交感及第三类神经系统均有改变。

In recent years, to bowel as people is the thorough knowledge of the central organ that should stimulate response and importance of function of alvine path protective screen, the nutrition inside bowel especially the attention that inchoate EN caused people again. The feasibility of 1 EEN and opportunity tradition idea think, the meeting after abdominal surgery has continuously the bowel that 3 D controls is paralytic, block up small intestine is absorbed to what nurture pledges.

概要： 近年来，随着人们对肠道是应激反应的中心器官以及肠道屏障功能重要性的深入熟悉，肠内营养尤其是早期EN又引起了人们的重视。1 EEN的可行性和时机传统观念认为，腹部手术后会有持续3 d左右的肠麻痹，阻碍小肠对营养物质的吸收。

The result shows, in 3 kinds of endocrine cells, divide outside discovering the cell that secretes 5- hydroxide lubricious amine exists, secrete gastric secrete element and grow the cell of curb element all has in the whole bowel path of piscine body distributing, and its distributing density once upon a time bowel backward alvine direction is reduced gradually.

结果显示，3种内分泌细胞中，除未发现分泌5-羟色胺的细胞存在外，分泌胃泌素和生长抑素的细胞在鱼体的整个肠道皆有分布，且其分布密度从前肠向后肠方向逐渐降低。

And more, ERK contents between group A and B, group C and D were not dramatically different. The pathological lesions indicated that intestinal tissue necrosis was severe in group C and D, which were graded 3 points, but obviously lessened in group A and B, which was graded 1 point, with ITF interfering. Conclusion: Intestinal inflammation was ameliorated after ITF was injected hypodermically or intraperitoneally.

结果：A、B组组织匀浆中ERK的面密度值各为87.68±19.24、82.65±21.35，较C、D组(30.23±7.79、30.74±8.19)明显升高(P＜0.01)，C、D组与E组(5.41±1.46)比较也有升高(P＜0.05)；并且随著ERK的激活伴随著明显的核转位。A、B组间及C、D组间ERK含量无显著差异(P＞0.05)；病理切片显示C、D组HE染色切片见肠壁损伤轻重不一，可见全肠粘膜绒毛坏死，病理评分的中位积分为3分；A、B组肠上皮细胞少量脱落，顶端绒毛坏死，病理评分的中位积分为1分。

Monodon severely infected by MBV was different from those of healthy ones. The contents of fatty acids in mid-gut glands of P. japonicus suffering from BMNV diseases also displayed similar changes.

MBV重度感染斑节对虾中肠腺各种脂肪酸的含量均有异于健康虾，患病日本对虾中肠腺各种脂肪酸的含量也出现明显的变化。

In recent years, ENK-IR neurons in myenteric plexus have been discovered. ENK is one of the important neurotransmitters in ENS. It can reduce the amplitude of cholinergic fEPSP and the release of Ach so as to inhibit contraction of smooth muscles.

近年发现在肠肌间丛神经元中有ENK-IR神经元，脑啡肽是肠神经系统中重要的神经递质之一，可降低胆碱能快速兴奋性突触后电位的幅度，减少乙酰胆碱的释放，抑制平滑肌的收缩。

Feed additives, including mannnanoligosaccharides, will reduce intestinal colonisation and shedding as salmonella with type-1 fimbria are agglutinated by the mannose component within the lumen and are excreted without adherence to enterocytes.

由于携有1型菌毛的沙门氏菌易与动物肠腔内的甘露糖分结合而脱离肠上皮细胞被排出体外，因此，选择和使用合适的饲料添加剂尤其是甘露寡糖等，将有助于降低沙门氏菌在家禽肠道内的增殖速度和减少排毒量。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。