无胸腺的
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In addition, the result of CT-DNA photodamage experiments suggested that the photodamage ability of p-HPcZn had increased 1.3 or 2.1 fold approximately compared with hematoporphyrin, in the presence or absence of oxygen.
7p-HPcZn以嵌插的方式与小牛胸腺脱氧核糖核酸相互作用,在有氧和无氧的条件下,p-HPcZn对CT-DNA的光敏损伤能力分别是血卟啉光敏损伤能力的1.3和2.1倍。
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The EAE rats were induced by injecting subcutaneously an emulsion of GPSCH and CFA. In addition, each rat received an intracutaneous injection of pertussis. Albumin in serum and CSF samples was examined on the days of 4,6,8,10,12,14,16,18 and 20 p.i.. The integrity of BBB was assessed by caculating CSF to serum albumin quotient. At the same time the brains, spinal cords, thymus and inguinal lymph nodes were removed for paraffin sections cutting. These sections were used for HE staining and ICAM-1 semiquantitative immunohistochemical staining. The results showed as follows:1. None of EAE rats on days 4,6 and 8 p.i. showed clinical signs whichbecame apparent on day 10 p.i.(2.33±0.76; n=6). On days 12 and 14 p.i.
检测p.i。第4天、6天、8天、10天、12天、14天、16天、18天、20天血清和CSF中ALB含量,其CSF/S ALB比值作为评价BBB损害的指标;同时段取动物脑、脊髓、胸腺和腹股沟淋巴结制成石蜡切片行HE染色和ICAM-1半定量免疫组化分析,结果显示如下: 1、EAE大鼠p.i.4-8天无任何临床表现;p.i.10天症状开始明显,症状评分为2.33±0.76(n=6);p.i.12-14天为发病高峰期,症状评分为3.75±0.54(n=12),病情由最初的活动减少,体重减轻逐渐发展为肢体无力、瘫痪,共济失调,甚至濒死状态;p.i.16天大部分动物症状开始缓解,评分为2.08±0.66(n=6);p.i.20天疾病症状完全消失。
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In this paper the thymus DNA of calf and the pBs plasmid are the main objects of our studies in ultraweak luminescence. By means of PIAS(Photon-counting image acquisition), ultraweak luminescence detector and Lambda Bio 40 of UV/VIS spectrophotometer, we studied the UL of DNA induced by the three oxygen free radicals: singlet oxygen Oa, hydroxy radical and superoxide anion(O2 ). We found that the OH can greatly increase the UL intensity of DNA.
本论文以小牛胸腺DNA和质粒pBs为实验材料,利用光子图像探测系统(Photon-counting image acquistion,PIAS)、微弱发光测量仪和UV/VIS分光光度计和生物统计学的成组比较法原理对单线态氧(~1O_2)、羟自由基、超氧阴离子(O_2~-)三种氧自由基诱导DNA超弱发光的变化过程进行了研究,结果发现这三种氧自由基中·OH能诱导DNA的超弱发光发生显著的增强变化,而其他两种氧自由基对DNA的超弱发光并无显著诱导作用。
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The results of this study indicated that there were changes on the distribution of T-lymphocyte subsets in patients with asthenia gravis.
MG患者存在有明显的T细胞亚群的改变,符合自身免疫性疾病的特点:胸腺切除手术近期对患者周围血中各淋巴亚群无明显影响;胸腺切除围手术期使用TP-5治疗对其发挥免疫调节作用的主要机理之一。
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To find methylated genes, researchers usually turn to a technique called bisulphite sequencing, which chemically changes normal cytosine to thymidine (T; another DNA base), while leaving methylated cytosines unchanged.
研究者通常借助于亚硫酸氢盐测序技术检测甲基化的基因,它通过化学反应使正常胞嘧啶转变为胸腺嘧啶,而对甲基化的胞嘧啶无作用。
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Each group of rats was kept in a separate cage. All of the rats drank water freely and were fed on stand food for rats.The experiment lasted 4 weeks. Indexes to be examined are as follows:⑴spleen T lymphocyte transform value,⑵spleen NK cell activation alternation value,⑶abdominal cavity macrophagocyte IL-1 and IL-2 activation alternation value.
所有动物分笼饲养,各组均自由饮水,喂食标准鼠类饲料,实验时间为4周。4周后,ME组和FE组作一次性力竭游泳运动后和其他各组大鼠股动脉放血处死,留取血清,无菌取胸腺和脾脏,行以下指标检测:⑴脾脏T淋巴细胞转化值;⑵脾脏NK细胞毒活性的变化值;⑶腹腔巨噬细胞IL-1的活性变化值和脾脏分泌的IL-2的活性变化值。
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There is no effective treatment protocol for patients with advance stage thymic carcinoma.
目前对晚期胸腺癌病患的治疗方法仍无有效的整合。
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There were no difference between ZS rats and ZA〓 rats.
补锌后,p56〓蛋白的表达下降,ZS组大鼠胸腺和脾脏组织的阳性细胞率和ZA〓组无显著差异。
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In vivo gene therapy of ovarian cancer by adenovirus-mediated thymidine kinase gene transduction and ganciclovir administration.
羟甲基无环鸟苷对携有单纯疱疹病毒胸腺嘧啶核苷激酶基因人卵巢癌裸鼠皮下移植瘤生长的抑制作用。
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Objective:To study the relationships between the expression of thymidine phosphorylase,the density of microvesselsof the tumor,the clinical and pathological features and tumor-free survival rate in endometrial carcinoma.
目的:探讨胸腺嘧啶核苷磷酸化酶与子宫内膜癌的临床病理特征、无瘤生存率、微血管密度的关系。
- 推荐网络例句
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This one mode pays close attention to network credence foundation of the businessman very much.
这一模式非常关注商人的网络信用基础。
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Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.
扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。
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There is no differences of cell proliferation vitality between labeled and unlabeled NSCs.
双标记神经干细胞的增殖、分化活力与未标记神经干细胞相比无改变。