无细胞的

The cancers studied here were throat cancer, aute non- lymphoblas He leukem ia and acut lymphoblastic leukemia. The result s indicated that,(1)the levels of Leu, Lys, Ser Asn,Ile,Gly,and Thr were obviously decreased whea lse the levels of orn, Trp evidently increased in cultural liquid of throat can cer cell line indicating that the growth of HepⅡ cell consumed 7 amino acids and releaced 2 amino acids into cultural liquid.(2)the levels of Glu, Gls, Leu, Phe, Tyr and Trp were evidently increased while the levels of Thr , His, Ala decreased in plasma from patient with ANLL. These r esults were consistent with the previous report.(3)the levels of Gly, Trp and Phe in plasma from patients with ANLL evidently de creased and the levels of Ala, His increased after therapy indicating that cance r cells were growing in an anaerobic catabolism state.

这些癌肿包括喉癌HepⅡ细胞，急性非淋巴细胞白血病和急性淋巴细胞白血病，结果表明：(1)喉癌细胞株培养过程中亮氨酸、赖氨酸、丝氨酸、天冬酰胺、异亮氨酸、甘氨酸以及苏氨酸等水平明显降低，而色氨酸水平明显增加，说明喉癌细胞的生长繁殖必须依赖以上7种氨基酸同时释放了色氨酸；(2)急性非淋巴细胞白血病患者血浆中的谷氨酸、甘氨酸、亮氨酸、苯丙氨酸、酪氨酸和色氨酸等水平明显升高，而苏氨酸、组氨酸、丙氨酸等水平明显降低，这些结果与国际报道相一致；(3)经治疗后，ANLL患者血浆中甘氨酸、色氨酸和苯丙氨酸等水平明显降低，而丙氨酸、组氨酸等水平明显升高，表明肿瘤细胞处在无氧代谢。

It can expedites proliferation and synthesizing active of cartilage cell and osteoblast too. Quicken ossification in the cartilagines and improving bone repair. But it has no influence to engendering blood vessel.

在骨愈合早期、中期，中药可加强巨核细胞的聚集及增强其活性，并可促进软骨细胞、成骨细胞的增殖及合成活性，加快软骨内骨化，促进骨愈合；而对血管形成无明显作用。

Thunberg Fritillary Bulb is one of the most popular Traditional Chinese herbs. Some trials in vivo and animals indicated that Peimine and Peiminine, extracted from Thunbery Fritillary Bulb, have the function of reversing MDR of two different kinds of mechanisms. They can improve the cytotoxic of ADR to cancer cells by 5～10 folds under concentration of 10% cancer cell growth inhibitory rate. Peimine and Peiminine not only feather typical character of modifier of MDR, but also differ from other moldifiers such as Calcium channel blocker, Cyclosporines in chemical structure, they belong to cevine groups alkaloid, a subgroup of C-Nor-D-homosteroid alkaloid.

浙贝母是临床常用中药，体外及动物试验证明，浙贝母中的有效活性成分贝母甲素和贝母乙素具有逆转肿瘤细胞MDR的作用，而且能逆转两种不同机制的多药耐药肿瘤细胞的耐药性，在无明显细胞毒剂量下，可使阿霉素对耐药肿瘤细胞的杀伤活性提高5-10倍，不但具备明确的多药耐药逆转剂的特征，而且在化学结构上属异甾类生物碱中的瑟文类生物碱，完全不同于钙离子拮抗剂、免疫抑制剂和其它现有的耐药逆转剂，具有作用靶点宽、化学结构独特的特点。

Results The inhibiting effect of gambogic acid on Bel-7402cell cycle particularity,but couldn't in-duce the cell apoptosis.

结果 藤黄酸对Bel-7402细胞的抑制作用表现为细胞增殖周期特异性，无明显诱导细胞凋亡的作用。

Results The inhibiting effect of gambogic acid on Bel-7402cell cycle particularity,but couldn't in-duce the cell apoptosis.

结果 藤黄酸对Bel-7402细胞的抑制作用表现细胞增殖周期特异性，无明显诱导细胞凋亡的作用。

The outward rectifier K+ current current was the dominant K+ current in the whole cell currents in Deiters' cells. In the presence of 200μ M linopirdine, the Ik current was not significantly effected. Our findings demonstrated that the KCNQ heteromeric or homomeric potassium channel may be the molecular basis for the peak outward K+ current and testified that the inward IKn current is mediated by KCNQ potassium channel.

在细胞外液中，加入100 μmol/L利诺吡啶后，OHCs中的四乙基二乙胺敏感的钾电流峰电流成分被抑制，稳态电流幅值减小，且电流的失活时间常数明显延长；在细胞外液中加入100 μmol/L和200 μmol/L利诺吡啶后，OHCs的内向性钾电流IKn被完全抑制；而细胞外液中利诺吡啶终浓度为200 μmol/L 时，Deiters细胞的外向整流性钾电流幅值无明显变化。

This effect was observed clearly at final concentration between 1 and 10μ mol/L after treating for 24 hours. We concluded that the possible mechanisms of hydroquinone bleaching hyperpigmented skin were through its inhibition of tyrosinase activity at lower concentration and its cytotoxicity at higher one.

提示18α-甘草酸双胺盐、熊果苷的皮肤脱色作用机制与氢醌可能不同，后者低浓度时以抑制酪酸酶活性为主，高浓度时主要是细胞毒，且细胞早期的损伤部位可能发生在树状突。18α-甘草酸双胺盐能否成为一种无细胞毒酪氨酸酶抑制型皮肤脱色剂有待进一步临床加以验证。

He experiment results indicate that (1) The extract of Rubia Cordifolia L has the effects of anti-proliferation and inducing APO;(2) Whenadministrating with 5-fu, the extract of Rubia Cordifolia L obviously enhanced the effects of anti-proliferation and inducing APO of 5-fu;(3) The basement of molecular biology of the extract of Rubia Cordifolia L for inducing APO of MGC-803 cells is correlated with inhibiting the expression of bcl-2 of anti-apoptosis gene.

UNEL法与PCNA的免疫细胞化学法的结果：0.2、0.4、0.8、1.0mg/ml浓度的茜草提取物与2、4、8、16μg/ml浓度的5-Fu，分别诱导细胞凋亡和抑制细胞增殖，表现出浓度、时间依赖性关系，除个别浓度与个别时间之间无显著性差异（P＞0.05）外，不同浓度与不同作用时间之间均有显著性差异（P＜0.05或P＜0.01），并且两种药物联合使用时，MGC-803细胞的APO%显著提高，降低PCNA的阳性表达率（P＜0.05或P＜0.01）。

The Ito inactivation curve of M cells shifted in the hyperpolarization direction, while the Ito inactivation curves of Epi and Endo shifted in the depolarization direction. There were no significant differences in the Ito recovery curves from inactivation among the three subtypical cells.

V曲线呈时间依赖性下移，Endo呈时间依赖性上移，M细胞先上移然后下移；M细胞的Ito失活曲线左移，而Epi和Endo右移；模拟缺血3层细胞的Ito失活后再恢复无明显差异。

The proliferation was determined by measuring -TdR incorporation in a β-scintillation counter. RESULTS: The in vivo administration of CCK-8 resulted in increase of B7.2 expression, but without any influence on B7.1 expression in peritoneal macrophages. CCK-8 also exhibited increased the -TdR incorporation in CD4(superscript +)T cells.

结果：整体应用CCK-8作用小鼠腹腔巨噬细胞，与对照组相比，CCK-8可上调静息小鼠腹腔巨噬细胞B7.2的表达，而对B7.1的表达则无影响；并且CCK-8使CD4T细胞[3H]-TdR的掺入率升高，即促进其增殖，增强巨噬细胞的协同刺激活性。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。