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When the transfusion bottles / bags hanging on to the hanging, the infusion of liquid Disu window Disu window gradually dropped to the bottom of this maglev ball on the absorption of Disu window at the bottom of the dry reed pipe, alarm circuits , Issued a call buzzer sound, had informed the infusion of water, the need to stop the transfusion and played a prevent patients in blood transfusion occurred when back to the role.

当输液瓶/袋内的吊液吊尽时,输液滴速窗的液面逐渐下降至滴速窗的底部,此时磁浮球就吸合位于滴速窗底部的干簧管,报警回路工作,蜂鸣器发出鸣叫声,告知输液水已无,需要停止输液,起到了防止病人在输液时发生回血的作用。

The improvement of preparation of the compound after the capsule in the treatment of bone fractures with the traditional technique of compound osteosynthesis capsules compared with obvious effect, safety, no significant side effects, most of the patients in the application of a drug the first course of treatment that is swelling, pain effect a treatment for various parts of the fractures, dislocation, as well as various parts of the body's soft tissue injury of effective drugs.

改进制备工艺后的复方接骨胶囊治疗骨折方面与传统工艺的复方接骨胶囊比较具有疗效明显、安全、无明显毒副作用,多数病人在应用药物第1个疗程即有消肿、止痛效果,是一种治疗各种部位的骨折,脱位以及身体各部位的软组织损伤的有效药物。

At least half of patients with X-ALD are adults with somewhat milder manifestations, and women who are carriers may become symptomatic. X-ALD is often misdiagnosed as attention-deficit/hyperactivity disorder in boys and as multiple sclerosis in men and women, and is not an uncommon cause of Addison disease;(2) the incidence of X-ALD, estimated to be 1:17 000 in all ethnic groups, approximates that of phenylketonuria;(3) noninvasive and presymptomatic diagnosis and prenatal diagnosis are available; family screening and genetic counseling are key to disease prevention; and (4) new therapies, applied early, show promise.

至少1半的X-ALD为成年病人,表现较轻,而作为携带者的女性也可发病。X-ALD在男孩常误诊为注意缺陷/多动综合征,男女可误诊为多发性硬化,并且也是Addison病的病因;(2)据估计X-ALD发病率为1:17 000,类似于苯丙酮尿症;(3)可进行无创性、症状前诊断和产前诊断;家系筛查和遗传咨询是疾病预防的关键;(4)早期采用新的治疗方法非常具有前景。

At least half of patients with X-ALD are adults with somewhat milder manifestations, and women who are carriers may become symptomatic. X-ALD is often misdiagnosed as attention-deficit/hyperactiity disorder in boys and as multiple sclerosis in men and women, and is not an uncommon cause of Addison disease;(2) the incidence of X-ALD, estimated to be 1:17 000 in all ethnic groups, approximates that of phenylketonuria;(3) noninasie and presymptomatic diagnosis and prenatal diagnosis are aailable; family screening and genetic counseling are key to disease preention; and (4) new therapies, applied early, show promise.

至少1半的X-ALD为成年病人,表现较轻,而作为携带者的女性也可发病。X-ALD在男孩常误诊为注意缺陷/多动综合征,男女可误诊为多发性硬化,并且也是Addison病的病因;(2)据估计X-ALD发病率为1:17 000,类似于苯丙酮尿症;(3)可进行无创性、症状前诊断和产前诊断;家系筛查和遗传咨询是疾病预防的关键;(4)早期采用新的治疗方法非常具有前景。

Results Serum EDLS increased within 24 h after brain trauma of different degree in all patients with brain injuries of various intensities.The group of slight injury was the most typal,EDLS demonstrated no change or decrease in a few patients with the injury intensity growing;In most cases EDLS decreased after brain operation or accompaning the wounded recovery; Contents of EDLS at 24 h among different pathological types of brain injuries showed no difference, but in the successful recovery patients with severe or ultra-severe brain injuries, EDLS contents at 72 h posttrauma were lower than in the patients of death or disability. The above change characteristics of EDLS related to injury locations and the degree of brain injury.

结果 不同程度脑外伤后24小时内血清EDLS均升高,以轻伤组最为典型;随着损伤程度的加剧,少数病例EDLS不变甚至下降;术后或随伤情的恢复,EDLS多同步下降;不同病理类型的脑损伤24小时内EDLS无明显差异,在成功康复的重型、特重型脑损伤病人,伤后72小时EDLS含量低于死亡或伤残组;EDLS 的上述变化特点与损伤部位和程度不同有关。

Itamin D from the skin and diet is metabolized in the lier to 25-hydroxyitamin D (Figure 1), which is used to determine a patient's itamin D status1,2,3,4; 25-hydroxyitamin D is metabolized in the kidneys by the enzyme 25-hydroxyitamin D-1-hydroxylase (CYP27B1) to its actie form, 1,25-dihydroxyitamin D.1,2,3,4 The renal production of 1,25-dihydroxyitamin D is tightly regulated by plasma parathyroid hormone leels and serum calcium and phosphorus leels.1,2,3,4 Fibroblast growth factor 23, secreted from the bone, causes the sodium–phosphate cotransporter to be internalized by the cells of the kidney and small intestine and also suppresses 1,25-dihydroxyitamin D synthesis.5 The efficiency of the absorption of renal calcium and of intestinal calcium and phosphorus is increased in the presence of 1,25-dihydroxyitamin D (Figure 1).2,3,6 It also induces the expression of the enzyme 25-hydroxyitamin D-24-hydroxylase (CYP24), which catabolizes both 25-hydroxyitamin D and 1,25-dihydroxyitamin D into biologically inactie, water-soluble calcitroic acid.2,3,4

从皮肤和食物来的维生素D在肝中代谢为25-羟基维生素D(图1),被用来决定病人体内维生素D情况的1,2,3,4;25-羟基维生素D在肾中被25-羟基维生素D1羟化酶(CYP27B1)转变为有活性的1,25-二羟基维生素D 。1,2,3,4由肾产生1,25-二羟基维生素D是被血浆甲状旁腺激素和血清钙,磷水平紧密调节。1,2,3,4由骨分泌的成纤维细胞生长因子23使钠磷协同转运蛋白被肾和小肠细胞内化及抑制1,25-二羟维生素D合成。5 在1,25-二羟基维生素D作用下肾和小肠吸收钙及磷的效率增高(图1)。2,3,6 它也包括25-羟四- 24 -羟化酶的表达(CYP24),且将1,25二羟基维生素D和25羟基维生素D异化成无生物活性,水溶性的维生素D3-23羧酸。2,3,4

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