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Objective To study the effective treatment in patients with oliguresis and anuresis after renal transplantation.

目的 探讨同种异体肾移植术后发生少尿或无尿患者的有效诊治。

Objective To study the effective treatment in patients with oliguresis and anuresis after renal transplantation.Methods Active waiting therapy was performed including carefully detection of allograft blood sup-ply,hemodialysis,prevention and treatment of rejection,etc.

目的 探讨同异体肾移植术后发生少尿或无尿患者的有效诊治方法彩色普勒超声严密观察移植肾脏的血供,仔细防治排斥反应等的"积极等待"疗法。

Sudden anuresis was the main feature of arterial thrombosis, while venous thrombosis was characterized by sudden anuresis and renal graft pain even graft rupture.

动脉血栓形成主要表现为突发的无尿,静脉血栓表现为少尿伴移植肾区胀痛甚至移植肾破裂。

The result indicated that the animal occurred respiratory acidosis when IAP got 15mmHg,and occurred hypotension and ologuresis or anuresis..

结果表明,当腹腔压力达到15mmHg时机体病理生理改变即可出现酸中毒,当腹腔压力达到20mmHg时继而出现低血压、少尿、无尿等症状。

Results: The ratio of male patients to female in the group was about 1.8∶1. Frequent clinical symptoms and signs: back and flank pain(69.57%),hydronephrosis(75.65%),deficiency of renal function(52.17%),dilatation of superior segment ureter(39.13%),edema of lower extremities(25.22%), hypertension(22.62%),and oliguria or anuresis(20.87%).

结果: (1)该病患者男∶女为1.8∶1,腰背部疼痛80例(69.57%),肾积水87例(75.65%),肾功能损害60例(52.17%),上段输尿管扩张49例(42.61%),下肢水肿29例(25.22%),高血压26例(22.61%),少尿、无尿24例(20.87%)。

Its most obvious symptom is the reduction in urine volume, and even anuria.

其最明显的症状就是尿量减少,甚至无尿

Urine of these patients gradually reduced, even anuria. Twenty-one patients were dead during hospitalization and eight patients were discharged by themselves.

其尿量逐渐减少,甚至无尿。29例患者中21例在住院期间死亡,8例自动出院。

Results: 32 patients with PNS suffered from acute oliguria or anuria,acute deterioration of renal function and rising of serum BUN and SCr, excluding secondly glomerular disease and acute tubular necrosis?

结果 :原发性肾病综合征在病情中突然发生少尿或无尿,肾功能急骤恶化,血尿素氮及肌酐上升,排除继发性肾小球疾病及其他原因引起的急性肾小管坏死、急性间质性肾炎和双肾静脉主干血栓形成,经甲基强的松龙和环磷酰胺或吗替麦考酚酯及标准疗程泼尼松、血液透析等对症治疗,32例患者肾功能均改善。

Indicatrix of the model parameter is concise and dirct-viewing, such as 20mmHg abdominal pressure, oliguresis oranuresis, dyspneic respiration,and so on.④Basing on the model, it is the next step to study hemodynamic change, pathological change of hepar,kidney and lung, digestive canal dynamia change, resuscitation strategies, reperfusion injury and survival after decompression,and so on.

如腹腔压力为20mmHg时,即出现少尿或无尿、呼吸困难等病理生理改变;(4)可利用该模型获得血液动力学、肝、肾、肺组织病理学、消化道动力学等改变的客观数据;并进行优化选择复苏液体方案及减压后脏器再灌注损伤、减压后成活率等研究;(5)为ACS的研究提供了良好的病理生理基础。

Methods 60 patients with acute cerebral infarction with uroschesis or not at the same time were retrospectively and contrastively analysed.

回顾分析60例急性脑梗死患者合并尿潴留的病例并与同期无尿潴留的急性脑梗死患者对比。

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