收缩血管的

Methods: Fresh saphena rings were sudied in organ chamber after incubating with Krebs-Henseleit bicarbonate solutions, FWM, KH with 0.1mmol/L NCR, FWM with 0.1mmol/L NCR at 37℃ for 1 hour. Precontraction induced by prostaglandin F2 (U4661930nmol/L) and endothelium-dependent relaxation induced by calcium ionophore A23187 l0^(-10~l0^(-6) mol/L were measured in the presence of indomethacin (7μmol/L) and LNNA (300μmol/L). Result: No significant changes were found in the 4 groups under the light microscope.

采用器官槽法研究在37℃有氧条件下用重碳酸盐缓冲液、含0.1mmol/L NCR的KH液、FWM液、含0.1mmol/L NCR的FWM液中浸泡血管环1h后，再用7μmol/L吲哚美辛和300 μmol/L N-硝基－L-精氨酸作用后，检测30 nmol/L U46619及不同浓度A23187引发的血管收缩舒张反应。

Treatment group was treated with valsartan and control group was treated without drugs such as ACEI or ARBs. Pmax,Pd and LADd,LVDs,LVDd were measured and compared before treatment and 6 months' after treatment. Result: There was significant difference in Pd , Pmax and LADd between treatment group and control group .

治疗组给予缬沙坦治疗，对照组给予非血管紧张素转化酶抑制剂类及非血管紧张素受体拮抗剂类降压药治疗，观察药物治疗6个月前后心电图中的Pmax和Pd及超声心动图中的左房舒张末内径、左室收缩末内径、左室舒张末内径的变化。

Abnormal vasoconstriction of digital arteries and cutaneous arterioles due to a local defect in normal vascular responses is thought to underlie the disorder .

本病的基础是因正常血管反应的局部缺陷而导致的指动脉和皮肤小动脉的异常血管收缩。

These results suggest that the inhibitoryeffect of sevoflurane on Ang II-induced vasoconstriction is,at least in part, caused by the inhibition of the p44/42 MAPK-mediatedsignaling pathway.

这一结果表明：七氟醚对於血管紧张素 II 介导的血管收缩的抑制作用，至少部分是由於对 p44/42 MAPK 介导的信号通道的抑制引起。

Results: The total saponins of Achyranthes Bidentata Blume not only had significantly inhibitive effects on NE-induced vasoconstriction, but also had direct relaxation effect on vascular smooth muscle.

结果：怀牛膝总皂苷不仅对NE引起的血管收缩有显著的抑制作用，而且还有直接舒张血管平滑肌的作用。

Results Following hemorrhagic shock, the systemic and local vascular responsiveness was significantly decreased significantly and it was time dependent. Shed blood reinfusion alone did not restore the decreased vascular reactivity.

结果失血性休克后全身（血压对 NE的升压反应）和局部（SMA对NE的收缩反应）血管反应性显著降低，且呈一定的程度和时间依赖性，即休克程度越重，时间越长，血管反应性降低越多。

In forearm resistance arteries,ADM can abolish the vasoconstrictive effects of high dose norepinephrine or endothelin.

在前臂阻力血管低浓度的ADM可以逆转高浓度的NE或内皮素的血管收缩作用。

Excessive neural input does not mediate diminished vasoconstrictive responses in the ACL transected knee; inflammatory mediators may play a role in the deficient vascular responsiveness of the ACL transected knee.

过量的神经输入没有能够调节前交叉韧带切除了的膝关节的血管收缩应答反应；炎症调节因子可能在前交叉韧带切除的膝关节血管应答不良中期重要作用。

Lung Ang Ⅱ correlated positively with MIP-2 in BALF in all study rats. These results indicate that local angiotensin system is involved in the pathogenesis of VILI and suggest that blockade of Ang Ⅱ might have potential therapeutic implications in alleviating VILI.

这些结果表示局部血管紧缩素系统与机械通气导致肺伤害的病理生理机转有关，并且建议阻断血管收缩素可能有治疗机械通气导致肺伤害的潜在功效。

This contracting effect of NE on BA could be augmented by β-adrenoceptor antagonist propranolol (10〓mol/L)(there was no this augmenting effect in RA) and could be antagonised by α〓-adrenoceptor antagonist prazosin but couldn′t be antagonised by α〓-adrenoceptor antagonist yohimbin (10〓mol/L).

这说明AVP引起的收缩在BA对胞外钙的依赖性明显大于RA，提示与V〓受体耦联的胞内钙池在BA明显小于RA。因此，血管活动的个血管活动的个性现象绝不仅仅是受体分布的差异所致。

We have no common name for a mime of Sophron or Xenarchus and a Socratic Conversation; and we should still be without one even if the imitation in the two instances were in trimeters or elegiacs or some other kind of verse--though it is the way with people to tack on 'poet' to the name of a metre, and talk of elegiac-poets and epic-poets, thinking that they call them poets not by reason of the imitative nature of their work, but indiscriminately by reason of the metre they write in.

索夫农 、森那库斯和苏格拉底式的对话采用的模仿没有一个公共的名称；三音步诗、挽歌体或其他类型的诗的模仿也没有——人们把&诗人&这一名词和格律名称结合到一起，称之为挽歌体诗人或者史诗诗人，他们被称为诗人，似乎只是因为遵守格律写作，而非他们作品的模仿本质。

The relationship between communicative competence and grammar teaching should be that of the ends and the means.

交际能力和语法的关系应该是目标与途径的关系。