支气管原的

In early COPD, the CT findings are based on hyperplasia of glands and rupture of smooth muscles at bronchus and bronchiole.

早期表现为腺体增生，1级时即可出现细支气管壁平滑肌的断裂，2级时出现支气管壁平滑肌的断裂及胶原纤维的增多。

At present, the pathological change of MP is interstitial pneumonia, or mixed bronchopneumonia or lobar pneumonia.

目前，肺炎支原体的基本病变为间质性肺炎，也可为融合性支气管肺炎或大叶性肺炎。

Methods SD rat were divided into asthma group (A, n=8), CGRP(8-37) interfering group (B, n=8) and normal control group (C, n=8). The CGRP level of Right lobes from rats exposed to 1% ovalbumin for seven episodes (30 min each day) or filtered air Group B are Pre-treated with the antagonist CGRP (8-37 before peritoneal injection OVA, were detect with histochemistry.

方法雄性SD大鼠给予腹腔注射致敏原后连续激发7天，其中干预组于激发前30min腹腔注射CGRP(8-37)，观察大鼠肺部组织学变化并用免疫组化SABC法及ELISA法分别观察肺组织、静脉血和支气管肺泡灌洗液中CGRP水平的变化。

Methods Using inhalation allergen, the patients of bronchial asthma were intradermally tested, and desensitization treatment was carried on by the corresponding initial concentration according to the grades of inspection results.

应用吸入性变应原对支气管哮喘患者进行皮内试验明确过敏原，并根据检查结果的分级以相应的初始浓度进行脱敏治疗。

An improved procedure of bronchialprovocation test with ragweed pollen ex-tract is described.

本文叙述了支气管激发试验的改良方法，用豕草花粉提取液作为过敏原。

It is concluded that both inasie and noninasie pulmonary function tests are capable of detecting allergen-specific as well as non-allergic bronchoconstriction in intact mice or rats.

结论是：无论是侵入性还是非侵入性肺功能实验，在大鼠或小鼠体内，都能检测出特异性变应原和非特异性变应原引起的支气管收缩。

Results 6 patients with PAP presented with ground-glass opacification of the bilateral lungs on HRCT. Typical chest HRCT scan showed "map-like" lesions in 3 cases and "cobble stone-like" lesions in 3 cases. Multifocal consolidative opacities in the lungs were seen on HRCT in 3 cases. Lung biopsies from 6 patients with PAP revealed a prominent positive periodic acid-Schiff intra-alveolar exudates. There were normal alveolar spetasis in 3 patients while interstitial thickening, fibroblast hyperplasia, interstitial fibrosis in the others.

结果 6例患者在不同层面可见磨玻璃影，其中2例可见小结节影；3例表现为两肺斑片状磨玻璃影与周围肺组织分界清楚，呈地图样改变；3例因磨玻璃影与小叶间隔增厚交织成铺路石样改变；3例可见肺泡实变融合成密度较高的斑片状阴影，1例在肺泡实变区可见&空气支气管征&。6例患者活检肺组织在光镜下显示肺泡腔内充满大量块状或颗粒状嗜伊红物质，PAS染色阳性，AB染色阴性，其中3例肺泡间隔正常，3例肺泡间隔增宽，可见慢性炎性细胞浸润、成纤维细胞增生和胶原沉积。

Lavement of pulmonary alveolus of right lung bronchus, undertake left lung of; of cellular classification, computation organizes divide evenly oar to determine group of immunity of collagen content; changes a law to determine sex of STAT1, plaque source grows factor the expression of albumen of 1(PAI-1 of depressor of content of activation of PDGF and fine dissolve zymogen.

右肺支气管肺泡灌洗，进行细胞分类、计数；左肺组织匀浆测定胶原含量；免疫组化法测定STAT1、血小板源性生长因子和纤溶酶原激活物抑制剂1(PAI-1)蛋白的表达。

Cells in the bron choalveolar lavage fluid were counted and differentiated, as well as the amounts of total protein, albumin and the activities of alkaline phos phatase, lactate dehydrogenase and type Ⅳ collagenase (matrix metallco proteinases, MMP-2, MMP-9) were detected.

计数支气管肺泡灌洗液中的细胞数并行分类，测定总蛋白、白蛋白含量及碱性磷酸酶、乳酸脱氢酶及Ⅳ型胶原酶(MMP-2、MMP-9)活性。

Drug intervention groups received either daily inhalation of budesonide, ipratropine or heparin respectively, starting on the 8th day or TGF-β1 monoclonal antibody(TB21)0.5 mg twice (6th and 19th day) via the tail veinous injection.

其它各药物干预组于制作模型第8 d起分别雾化吸入布地奈德（布地奈德组，12只）、溴化异丙托品（溴化异丙托品组，12只）和肝素（肝素组，6只）溶液。4周后检测小支气管平滑肌及胶原厚度，用免疫组化法及原位杂交法观察各生长因子在支气管肺内的表达，用放免法检测血清和BALF中细胞外基质成分Ⅲ型前胶原、层粘连蛋白及透明质酸。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。