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By immunohistochemistry, porcine parvovirus signals were mainly distributed in interstitial and necrotic lesions, and the positive signals could be found in macrophages-like cells and epithelial cells of terminal bronchiole and alveoli.

另外,以免疫组织化学染色检测猪小病毒,其阳性讯号主要分布於间质及坏死病灶区,此阳性讯号存於巨噬细胞与肺泡及终末细支气管上皮细胞。

Inflammatory cell infiltration and bronchiolitis severity were significantly reduced ofter administration of E. globulus oil. Especially in 300 mg·kg -1 treated rats,there were significant decreases of mucin content in BALF and MUC5ac expression in trachea and bronchiole epithelium. Optical density and mucins area% detected by image analysis system were apparently lower than those in model group. Conclusion: E.

各给药组经不同浓度蓝桉油灌胃后能减少中性粒细胞及淋巴细胞的浸润,降低炎症程度;尤其是蓝桉油高剂量组(300 mg·kg-1),能明显减少灌洗液中黏蛋白含量,使气管和细支气管上皮内黏蛋白MUC5ac表达的阳性染色区域和程度明显减轻,通过图像分析系统测得的吸光度值和黏蛋白面积%均明显低于模型组。

Parents should pay special attention to whether the baby bronchiole issues, or whether there痰多caused by respiratory obstruction, and that leads to the causes of neonatal respiratory distress include: low birth weight, the mother has a history of diabetes, premature rupture of membranes, meconium pollution, birth mother irregular fetal heart tone, prolapsed umbilical cord, the fetus had cooked, prematurity, multiple births, chest, heart or respiratory abnormalities.

家长要特别留意宝宝是否有细支气管方面的问题,或是否有痰多造成呼吸阻塞的现象,导致新生儿呼吸窘迫的原因包括:出生时体重过低、母亲有糖尿病史、胎膜过早破裂、胎便污染、妈妈分娩时胎心音不规则、脐带脱垂、胎儿过熟、早产、多胞胎、胸腔、心脏或呼吸道异常。

The IHC results showed the signals of PPV were present in the necrotic and interstitial lesions, the signals could occasionally be found in suppurative lesions of the terminal bronchiole.

PPV常於坏死及间质病灶区内见有阳性讯号,但亦能於终末细支气管之化脓病灶区见有阳性讯号。

Patients and Other Participants There are 25 patients with hronchial asthma, 21 patients with asthmaticbronchitis and 25 patients with bronchiolitis .

患者和其他参与者支气管哮喘患者25例,哮喘性支气管炎21例,毛细支气管炎25例。

One, build chronic bronchitic , bronchus is asthmatic animal model because the hair interpretation of the cause of CB, BA is made complex, imitate its pathology change and clinical feature become unusually difficult.

一、建慢性支气管炎、支气管哮喘动物模型的标准由于CB、BA的发病机制复杂,模拟其病理变化和临床特征变得异常困难。

Objective To investigate the changes of the levels of serum NO/NOS and TNF after treatment in patients with children broncho pneumonia.

目的探讨小儿支气管肺炎治疗前后血清NO/NOS和TNF-2水平的变化。

Comparison between lesions displayed by airway post processing and the results of fiberoptic broncho...

由专家对不同扫描参数的气管后处理所显示的病变与支气管镜检结果进行比较,按Ⅰ、Ⅱ和Ⅲ级图像等级评判图像质量,并进行统计学处理。

Methods Serum TNF-2 and NO/NOSlevels were determined in 42 patients with children broncho pneumonia both before and after treatment as well as in 35 controls.

方法分别应用放免法和化学法测定了42例小儿支气管肺炎患者血清NO/NOS和TNF-2含量,并与35名正常儿童作比较。

The expression of ICAM 1 and HO 1 on broncho epithelial cells were detected through immuno histochemistry method.

采用免疫组化法检测各组大鼠支气管上皮细胞ICAM 1和HO 1表达。

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