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Lobular artery and bronchiole diameters are 1.0 mm.

小叶动脉和细支气管直径为1.0mm。

RESULTS: The expression of VCAM1 in the lung tissues of normal rats was weak, and distributed mainly in alveolar epithelial cells, bronchiole epithelial cells, alveolar macrophage and some pulmonary interstitium.

结果: 在正常对照组,VCAM1大鼠肺组织中呈弱表达,分布于肺泡上皮细胞、细支气管上皮细胞、肺泡巨噬细胞及少量的肺内间质。

At the ends of each bronchiole are clusters of air sacs. Adult lungs contain about 600 million of these spongy sacs, with enough combined surface area to cover a tennis court.

肺泡在每个支气管末端是一束束气泡,成人的肺包含约有6亿个这种海绵样的气泡,它们的表面紧密连接在一话可以覆盖一个网球场。

Twenty-six of 27 SARS patients and 5 of 18 healthy controls had strongly stained columnar epithelia of the bronchiole, especially the lumen border of the epithelia, the difference between two groups was significant.

在2 7例SARS患者血清中有2 6例在肺组织细小支气管柱状上皮细胞的腔面尖端呈现强阳性荧光信号,献血员中有5例,统计分析表明两者阳性率差异有统计学意义。

The bronchiole region and the alveolar well fibrosis caused the complaisance of the lung degraded the volume of lung decrease and the dysfunction ventilation.

由于细支气管领域和肺泡壁纤维化使肺的顺应性降低导致肺容量减少和限制性通气障碍。

Due to ozone can cause upper respiratory tract inflammation, injury terminal bronchiole epithelial cilia, thus weakening the defense of the upper respiratory tract, so long-term exposure to a certain concentration of ozone is also easy to secondary upper respiratory tract infection.

由于臭氧能引起上呼吸道炎症、损伤终末细支气管上皮纤毛,从而削弱了上呼吸道的防御功能,因此长期接触一定浓度的臭氧还易于继发上呼吸道感染。

Severe nonspecific airway inflammation is demonstrated in asthmatic patients with sudden death. Lesions of the left ventricle and mucous plug in bronchiole may contribute to the main cause leading sudden death.

结论哮喘猝死患者气道存在明显的非特异性炎症反应,心肌受损和细支气管粘液栓形成为其主要致死原因。

QTY06 significantly decreased MUC5ac expression in trachea and bronchiole epithelium, and reduced the optical density and mucins area as detected by image analysis in rats with chronic airway inflammation.

QTY06使气管和细支气管上皮内黏蛋白MUC5ac表达的阳性染色区域和程度明显减轻,通过图像分析系统测得的光密度值和黏蛋白面积%均明显低于模型组。

There was no significant difference in epithelial destruction, intra-luminal obstruction and BO formation between allografts from HO-1 transgenic pigs or non-transgenic pigs. However, in day 28 and 56 allografts, we did see some of the grafts still showed bronchiole structure with normal epithelial cells which hasn'tbeen found before.

使用HO-1 基因猪的异体移植肺块,在气管表皮破坏、气管内径阻塞、及阻塞性细支气管炎形成的程度,与使用非基因猪并无显著差异,惟在第28 天及56 天的肺块中,有部份的异体移植肺块中可见正常的支气管型态及表皮细胞。

The antibodies were purified and the purity and specificity were identified by SDS-PAGE and Western-blot,respectively.Lung tissues from five pigs with PRRS were tested with the established method,which clearly demonstrated the abundant presence of viral antigens in the cytoplasm of alveolar macrophages,and to less extent in alveolar and bronchiole epithelial cells.

SDS-PAGE和Western-blotting检测结果表明,制得的兔抗PRRSV IgG纯度较高,具有良好的特异性,可用于原位检测病猪肺组织中PRRSV抗原的分布;Envision法的检测结果显示,PRRSV抗原的阳性表达产物主要出现在肺巨噬细胞胞浆内,其次为肺泡上皮细胞和细支气管黏膜上皮细胞;通过对5份经RT-PCR检测确认的PRRS病猪肺组织进行检测,均有较高的阳性细胞表达,平均阳性细胞率为46.5%。

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