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Actinomycin D is the potent inhibitor of RNA synthesis, and is demonstrated to be able to induce Sf-9 cell, K562 cell, aleolar macroages cell CTLL-2 and Jurkat cell apoptosis.

放线菌素D是RNA合成抑制剂,具较强的诱导细胞凋亡的能力,实验证明放线菌素D可诱导Sf-9细胞,人白血病细胞株K562,肺泡巨噬细胞,CTLL-2和Jurkat细胞等凋亡。

Knamycin and Hygromycin were taken as screening reagents for gerbera resistive plantlets, and Cefotaxime and Carbenicillin as bacteriostats. Under different concentrations, their effects on differentiation of adventive buds were different.

以卡那霉素和潮霉素作为非洲菊抗性植株的选择剂,头孢霉素和羧苄青霉素为抑菌剂,分别采取不同浓度,分析其对非洲菊试管苗不定芽分化的影响。

This paper reaseched the interaction of Dihydromyricetin and lysozyme by using spectrophotography and antiblastic test to measured the activity of lysozyme indirectly,and compared with the activity of the lysozyme which is not acted by the Dihydromyricetin.

本文通过研究二氢杨梅素与溶菌酶的相互作用,用分光光度法和抑菌试验间接测定溶菌酶活性,并与未被二氢杨梅素处理的溶菌酶活性比较。

This study aims at the expression of Wnt signaling pathway inhibitor FrpHE and DKK-1 induced by ectogenous factor.

本课题的目的在于研究外源性因素对Wnt通路抑制剂FrpHE和DKK-1的诱导表达,我们选择抗癌药物p53及化疗药阿霉素、羟喜树碱、顺铂作为外源性诱导剂,以期弄清Wnt通路及其抑制剂FrpHE和DKK-1是否能够被p53和化疗药这两类药物所调节。

Conclusion; Myricetin and quercetin are inhibitors of PI3K, The inhibitory effects against p110β PI3K of myricetin and quercetin are dose-dependent.

杨梅素和槲皮素是PI3K的抑制剂,杨梅素和槲皮素对PI3K p110β催化亚基的抑制作用与浓度成正比关系。

Andres, and its mechanism against rot-causing fungi, the antifungal activity and period of pyrolin against Physalospora piricola and Alternaria alternate was evaluated by using agar dilution method, and ultrastructure changes was observed used by scanning electron microscope.

采用中药抑菌实验方法测定了鹿蹄草素对苹果轮纹病菌和梨链格孢菌的抑菌活性和时期,并在扫描电镜下观察菌体超微结构的变化。

PONI skin health research centre, with advanced skin care technology and causes skin pigment, successful research and development of a "'8 'black-suppression, the corresponding treatment and conditioning" technology, not undermine, not suppressed, does not damage the ecology of the skin Under the circumstances, the skin pigment formation of a smart decomposition, hypnosis pigment cells, skin in a relatively short time, reaching Suji elegant, Whitening Source lucent effect, lasting effect, the relative safety of similar products increased 68 percent.

&8&重抑黑,对应疗理,彰显透亮白嫩!柏宁皮肤健康研究中心,结合先进的护肤科技和皮肤色素成因,成功推出&8重抑黑,对应疗理&科技,在不破坏、不压制、不损伤肌肤原生态的情况下,对皮肤色素形成过程进行智能分解,催眠色素母细胞,使肌肤在较短时间内,达到素肌雅致,净白透亮的效果,效果持久、安全性相对同类产品提高了68%以上。

Methods After treated with a specific demethylating agent,Aza and acetylating agent, TSA, the status of 5'CpC island methylation of ING1b gene in HT29 human colon cancer cell line was analyzed using methylation specific polymerase chain reaction,and the level of histone acetylation was analyzed by chromatin immunoprecipitation,and reverse transcription polymerase chain reactionwas used to examine ING1b mRNA expression.

应用特异性DNA甲基转移酶抑制剂5-氮-2'-脱氧胞苷(5-Aza-2'-dc,以下简称Aza)及组蛋白去乙酰化酶抑制剂曲古抑菌素A作用人结肠癌细胞株HT29后,用甲基化特异性PCR检测该ING1b基因核心启动子区域CpG岛甲基化情况,用染色质免疫沉淀检测其乙酰化组蛋白绑定的DNA情况,并用逆转录聚合酶链反应检测ING1bmRNA表达。

Our experiments confirmed that two known inhibitors DRB and A3 Inhibited the activity of purified recombinant human CK2 holoenzyme, and that quercetin, its derivatives and some tyrphostins exerted stronger inhibitory effect on CK2 than DRB and A3. These results provided important experimental basis and a simple screening method for the development of more effective inhibitors of CK2 and their clinical application in the future.

本结果实验证实纯化的重组人CK2全酶可受到已知抑制剂DRB和A3的抑制,但槲皮素及其衍生物和某些Tyrphostins抑制CK2的作用较DRB和A3更强,这对于开发更有效的CK2抑制剂,为今后应用于临床提供了重要的实验依据和一种较为简便的筛选方法。

V increased (other than Ara-C) and the inhibitory effect against 〓 improved significantly, especially for ADM. The empty pro-liposomes may undergo three process when hydrated with drug solution: swelling of phospholipid film, fracture of film and resealing to form vesicles.

以空白脂质体前体为载体,对几种临床上常用的抗癌药物的抑瘤活性及毒性进行了研究,研究结果得出:与游离药物相比,5-Fu,阿霉素等药物的抑瘤活性都有不同程度的提高;急性毒性实验表明:除Ara-C外,几种药物的毒性均有所降低,以顺铂最明显。

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