抑制神经

Prolonged exposure to agonistic neuroinhibitors can result in long-term damage to metabolic systems.

长时间接触竞争性神经抑制剂会导致神经系统的长期损害。

When the treated group were injected Safflor after ICH 6,12,24 hours,The number of caspase-3 positive cell around hematoma was gradually increased and the number of bcl-2/bax positive cell was gradually degraded and the number of TUNEL positive cell of Safflor group treated after ICH 6 hours were less than that of Safflor group treated after ICH 24 hours.

在不同时间给予红花注射液治疗脑出血大鼠，均能不同程度地促进脑出血大鼠神经功能的恢复，其中术后6h给药组治疗效果最好，其可能通过促进Bcl-2蛋白的表达，抑制Bax和Caspase-3蛋白的表达，从而抑制血肿周围的细胞凋亡。

Results Compared with ICH model group,Safflor injection could significantly alleviate neurological system damage symptoms,heighten the ratio of bcl-2/bax,and reduce the number of caspase-3 positive cell and the number of TUNEL posit

在不同时间给予红花注射液治疗脑出血大鼠，均能不同程度地促进脑出血大鼠神经功能的恢复，其中术后6h给药组治疗效果最好，其可能通过促进Bcl-2蛋白的表达，抑制Bax和Caspase-3蛋白的表达，从而抑制血肿周围的细胞凋亡。

The identification of protein were performed by searching the mass spectrometry data from the NCBInr database using the software of Sequest Bioworks3. 2. The results indicated that over 25 protein components including acid and basis phospholipase A2, nerve growth factor, presynaptic neurotoxin, hemorrhagic factor, thrombin-like enzyme, serine protease, glycogen synthase, metalloproteinase, maturase, L-amino acid oxidase, plasminogen activator, adhesion, cysteine-rich secretory protein, disintegrin, fibronectin-binding protein, fibrinogen clotting inhibitor A chain, vascular apoptosis-inducing protein 1, pyranose 2-oxidase, carbamoyl-phosphate synthase, as well as the homology proteins of snake venom ablomin, ablomin, catrin, mucrofirase, pallabin, salmosin and triflin homologues were identified from the GSSV.

从GSSV中鉴定出超过25种蛋白组分，主要包括：酸性磷脂酶A1、碱性磷脂酶A2、神经生长因子、突触前神经毒素、出血因子、类凝血酶、丝氨酸蛋白酶、糖原合酶、金属蛋白酶、成熟酶、L-氨基酸氧化酶、纤溶酶源激活物、外源凝集素、富含半胱氨酸分泌酶、去整合素、纤连蛋白结合蛋白、纤维蛋白原凝固抑制因子A、血管凋亡诱导蛋白1、氨甲酰基磷酸合成酶、吡喃糖－2-氧化酶以及ablomin， catrin， mucrofirase， pallabin， salmosin和triflin的同源蛋白。

In fact, cells with NSCs-like properties are likely to occur throughout the adult CNS, but they are able to give rise to neurons in only a few restricted areas in vivo, i.e., the subventricular zone in the wall of the lateral ventricle and the subgranular zone of the hippocampal dentate gyrus. Very poor regenerative capability of the adult mammalian CNS could be explained as follows: first, theneuronal differentiation of endogenous NSCs may be inhibited by microenvironmental factors present in most areas of the adult CNS.

成年哺乳动物中枢神经系统可怜的再生能力可能主要归结于两方面的原因：第一，成年动物中枢神经系统内的大多数区域存在一些特殊的微环境因素，它们抑制了内源性神经干细胞向神经元方向的分化；第二，内源性神经干细胞的数量太少，不足以形成有效的自我修复。

The results indicated that GABAmay be the important inhibitory neuropeptides; VIP,β-EP, SP also involved in theregulation of the GnRH. Optical information may control the chicken productionperformance by adjusting the distribution of GABA, VIP andβ-EP neuropeptides withinthe nuclei related to the tectal layerⅠ.

结果表明，GABA可能是峡核和GLv核主要的抑制性神经肽，VIP、SP和β-EP也参与GnRH分泌的调控，光信息可能通过调节峡核-顶盖通路和顶盖-GLv通路中的GABA、VIP、SP和β-EP几种神经肽的分布来调控GnRH的分泌，进而调节鸡的生产性能。

To understand the basic properties, underlying mechanisms and functions of the short-term synaptic plasticity of thalamocortical visual pathway, we combined in vivo field potential recording, focal drug application technique and neuron models, to explore the short-tem synaptic plasticity of rat major thalamocortical visual pathway (from lateral geniculate nucleus to primary visual cortex) and extrageniculate pathway (from lateral posterior nucleus to primary visual cortex), as well as the role of short-term depression in the neuronal correlated firing detection.

我们结合在位场电位记录、局部加药和神经模型等方法，对不同刺激模式下大鼠丘脑皮层主要视觉通路（外侧膝状体到初级视皮层）和非主要视觉通路（膝体外通路：丘脑侧后核到初级视皮层）的短时程突触可塑性，以及短时程抑制作用对神经元同步活动检测的作用做了研究，试图了解视觉丘脑皮层突触传递短时程可塑性的基本特性、神经机制和功能。

IEPA was combined with neural network by the new algorithm, multi-layer feed-forward neural network structure was replaced with RBF neural network structure, and back propagation algorithm was replaced with IEPA, then, expected side-lobe suppression quota was rapidly waisted.

该算法将进化规划算法和神经网络结合起来，用径向基RBF神经网络结构取代多层前馈网络MFNN（Multilayer Feedforward Neural Nerworks）结构，用改进进化规划取代反向传播算法BP，可以以较大概率快速的收敛到要求的旁瓣抑制指标。

We show gain of function of excitatory neurotransmission due to increased action-potential-evoked Ca(2+) influx and increased probability of glutamate release at pyramidal cell synapses but unaltered inhibitory neurotransmission at fast-spiking interneuron synapses.

我们发现兴奋性神经传递的功能增益来自于诱发Ca(2+)内流动作电位的增加，并可能与谷氨酸盐在椎体细胞突触中的释放增加有关，但与快速成峰中间神经元突触的抑制性神经传递无关。

Hypoxia inhibits a variety of K+ channels in the type Ⅰ cells of the carotid body, in a seemingly species-dependent manner, and the resultant membrane depolarisation is sufficient to activate voltage-gated Ca2+ entry leading to neurosecretion and afferent discharge.

低氧可抑制颈动脉体Ⅰ型细胞中的多种K+通道，这种作用可能有种属差异；K+通道的抑制使膜电位去极化，启动电压依赖性Ca2+内流，最后导致神经分泌和传入放电。

This one mode pays close attention to network credence foundation of the businessman very much.

这一模式非常关注商人的网络信用基础。

Cell morphology of bacterial ghost of Pasteurella multocida was observed by scanning electron microscopy and inactivation ratio was estimated by CFU analysi.

扫描电镜观察多杀性巴氏杆菌细菌幽灵和菌落形成单位评价遗传灭活率。