抑制神经

A - V compound has greater effect of analgesia and inhabiting nerve action potential transmission than its components Asarum and Verapamil .

复方制剂的镇痛及抑制神经动作电位传导作用均大于其组分细辛和维拉帕米。

Antigene peptide nucleic acid specifically inhibits MYCN expression in human neuroblastoma cells leading to cell growth inhibition and apoptosis Roberto Tonelli,Stefania Purgato,Consuelo Camerin,Raffaele Fronza,Fabrizio Bologna,Simone Alboresi,Monica Franzoni,Roberto Corradini,Stefano Sforza,Andrea Faccini,Jason M. Shohet,Rosangela Marchelli,and Andrea Pession Department of Pediatrics, University of Bologna,Sant'Orsola-Malpighi Hospital, Bologna, Italy; Department of Organic and Industrial Chemistry, University of Parma, Parma,Italy; and 3Center for Cell and Gene Therapy, Texas Children's Cancer Center, Baylor College of Medicine, Houston, Texas Abstract We developed an antigene peptide nucleic acid for selective inhibition of MYCN transcription in neuroblastoma cells, targeted against a unique sequence in the antisense DNA strand of exon 2 of MYCN and linked at its NH2 terminus to a nuclear localization signal peptide.

抗原肽核酸抑制MYCN具体表达的神经母细胞瘤细胞，导致细胞生长的抑制和凋亡罗伯托利，斯特凡Purgato ，苏埃洛卡默琳，拉菲尔Fronza ，梅奥尼博洛尼亚，西蒙娜Alboresi ，莫妮卡弗兰索尼，罗伯托Corradini ，斯特凡诺斯福尔扎，安德烈Faccini ，杰森先生Shohet ， Rosangela Marchelli ，和安德烈Pession儿科，博洛尼亚大学，圣' Orsola -马尔皮基医院，博洛尼亚，意大利；有机部和工业大学化学帕尔马，帕尔马，意大利和3Center的细胞和基因治疗，德州儿童癌症中心， Baylor医学院，得克萨斯州休斯敦摘要我们开发了一种抗原肽核酸的选择性抑制神经母细胞瘤MYCN转录细胞，针对一个独特的序列反义DNA链的第2外显子，并与在MYCN 其氨基端为核定位信号肽。

In this method, we use the pooled correlogram based on human auditory model to extract the pitch of speech. An unsupervised lateral inhibitory network is used to get the peak position, which simulates the lateral inhibitory phenomenon in human auditory system.

采用基于人耳听觉模型的相关图来提取语音信号的基频，运用无监督的侧抑制神经网络来模拟人耳侧抑制属性进行基频检测，为了克服在低信噪比情况下侧抑制神经网络的误判问题，引入了相邻语音帧的语音基频的帧间约束。

Although both xGATa-1a and xGATA-1b are able to stimulate erythropoiesis, only xGATA-1b is capable of inhibiting neurogenesis in Xenopus embryos.

二种亚型在造血方面具有同样的作用，但只有xGATA-1b具有抑制神经发生的功能。

We proposed a hypothetic model with lateral inhibitions and give corresponding mathematical deduction to illustrate the internal biological mechanism of neuronal temporal RF.

提出了一个基于侧抑制神经网络的假设性模型来解释视皮层神经元时间感受野形成的内部机制，并给出了数学上的推导。

He angiogenesis and tumor growth of human neuroblastoma can also be effectively inhibited by TrkA gene.

rkA基因能降低NB细胞的致瘤性及抑制神经母细胞瘤的血管生成和肿瘤生长。

An amino acid that is found in the central nervous system; acts as an inhibitory neurotransmitter.

在中枢神经系统中发现的氨基酸，作为抑制神经传递素。

It was demonstrated that toosendanin was a selective presynaptic blocker, which blocked the neurotransmitter release both in peripheral and central nervous system.

已有的研究表明，它是一种作用于突触前的特异性传递阻断剂，通过抑制神经递质释放阻断神经肌肉接头和中枢神经系统的兴奋传递。

The ability of melanoblasts in the Silkie embryos to migrate ventrally is correlated with a lack of peanut agglutinin binding barrier tissues, which are present in the Lightbrown Leghorn embryos.

丝羽乌骨鸡的黑色素母细胞之所以可以移动至腹部是由於其缺少了神经阻碍物分子，而此神经阻碍物分子在棕色来航鸡的胚胎发育过程中，会明显地形成一道障壁以抑制神经嵴细胞和黑色素母细胞入侵胚胎腹部。

objective to observe the protective effect of qiliqiangxin capsule in rabbit heart ventricle reconstitution of chronic heart failure.methods using the method of giving rabbit abdomen aorta half ligations to build the chronic heart failure model to observe qiliqiangxin capsule the effect to blood flow mechanics index,room wall thickness,heart exponent and heart ventricle reconstitution biochemistry index.results qiliqiangxin capsule could reduce effectively bp,lvsp,lvedp,t wave,improve room wall thickness and heart exponent,reduce angii and alt,all the paramenters ahd notable difference,composed to those of model group (p＜0.05,p＜0.01).conclusion qiliqiangxin capsule can improve the blood flow mechanics change of chronic heart failure rabbit,which has notable inhibitory action on heart ventricle reconstitution which originate from chronic heart failure.inhibition of ang ii,alt of immune nerve endocrine system may be a mechanism of qiliqiangxin capsule.

观察血流动力学、标志心室重构的室壁厚度及心脏指数及肾素―血管紧张素―醛固酮系统相关生化指标的变化。结果芪苈强心胶囊可有效降低血压、左室压、左室末期舒张压、t波，改善室壁厚度和心脏指数，降低血清血管紧张素ii及醛固酮水平，与模型组比较均有显著性差异(p＜0.05，p＜0.01)。结论芪苈强心胶囊可改善慢性心力衰竭兔血流动力学的变化，对慢性心力衰竭引起的心室重构具有显著抑制作用，其机制可能与该药发挥抑制神经内分泌免疫系统rass中angii、ald的水平有关。

The split between the two groups can hardly be papered over.

这两个团体间的分歧难以掩饰。

This approach not only encourages a greater number of responses, but minimizes the likelihood of stale groupthink.

这种做法不仅鼓励了更多的反应，而且减少跟风的可能性。