抑制地

Numerical experiments show that the new method can restrain the "blocky effect" and preserve more edges and textures in the processing of noise removal.

数值试验表明，新方法能在有效抑制噪声的同时，能有效地抑制图像的"阶梯效应"，更好地保持图像的边缘、纹理细节信息。

Ovoalbumin-induced rat feet edema was significantly decreased by QLS with the doses of 35 and 70.0 mg·kg-1·d-1 (P.05).Cotton pellet-induced rat granuloma was significantly decreased by QLS with the doses of 35.0 and 70.0 mg·kg-1·d-1 (P.05 or P.01 ) and the granuloma inhibitory rates were 32.40 % and 35.65 %,respectively.

结果：QLS 3个剂量组均不同程度地抑制小鼠耳肿胀度，与空白对照组比较，50及100 mg·kg-1·d-1 QLS组小鼠耳肿胀度均明显减小（P.05或P.01），对小鼠耳肿胀的抑制率分别为53.97%和60.09%；QLS 35.0及70.0 mg·kg-1·d-1组均显著抑制大鼠足肿胀（P.05），且对棉球所致肉芽组织增生有明显的抑制作用（P.05或P.01），大鼠肉芽肿的抑制率分别为32.40 %和35.65 %。

The main job and contribution of this paper are as follows:Based on narrowband signal processing, we propose a new method for FH signal blind detection and blind parameter estimation. Firstly, compute the cross-correlation function of FH signals from multichannels, then we get the cross power spectral density matrixCSDM Secondly, select the hops of FH signals from the CSDM via processing and analyzing the CSDM, meanwhile reduce the influence coming from noise, fixed frequency signal and other interferences; Lastly, estimating the DOA of every hop and according to the DOA, we can separate the hops, get the number of FH signals and their parameters. Propose a new method for designing the threshold in FH signal detection. This method makes it easy to get the threshold by theoretic computing and it can extremely reduce the interference of non-FH signals.Based on broadband signal processing, we propose a new method for FH signal blind detection and blind parameter estimation. Firstly, get the DOA of the signals by wideband spatial spectrum estimation; secondly, separate the signals by spatial filtering using wideband beamformer; Lastly, we propose two methods for FH signal blind detection and blind parameter estimation, one is for several constant hop-rate FH signals whose dwell-time are different and another is for a single FH signal whose hop-rate is alterable.By employing spatial interpolation in broadband signal beamforming, it can reduce the number of antenna elements and corresponding RF modules, A/D converters, etc. It can also eliminate the mutual coupling between antenna elements while getting the narrower main beamwidth and lower sidelobe level.

本文的主要研究工作和贡献如下：●提出了一种基于窄带处理的FH信号盲检测和参数盲估计的实现方法，即首先求出多路信号之间的互相关，进而得到互功率谱密度矩阵，然后通过对CSDM进行对折处理来抑制噪声、定频信号及其他干扰的影响，并将所有属于FH信号的hop(每一跳的驻留时间在时频图上持续的线段)从CSDM中筛选出来，最后用阵列信号处理中的到达方向估计理论在频域中估计出各个hop的方向参数，利用方位信息完成FH信号分离并得到FH信号的数目及其各自的参数集；●提出了一种确定FH信号检测门限的策略，该策略不仅使得检测门限能够通过理论计算得到，而且还显著地抑制了非跳频信号的干扰，从而能有效地检测出FH信号；●提出了一种基于宽带处理的短波FH信号盲检测和参数盲估计的实现方法，即首先利用宽带空间谱测向技术得到接收信号的DOA，然后采用宽带波束形成技术对特定方向进行空间滤波，实现多个信号的分离，最后分别提出了两种FH信号的盲检测和参数盲估计方法，第一种方法适用于空间某个特定方向上存在着几个驻留时间不同的恒跳速FH信号和其他干扰信号的混合信号，第二种方法适用于空间某个特定方向上仅含一个可变跳速的FH信号和噪声的混合信号；●将空间插值的概念和宽带波束形成技术结合起来，提出了一种基于频域处理的宽带恒定束宽的波束形成方法。

Results: 1 The capacity of engulfment of A549 cells started enhancing at the 1h of preincubation with dexamethasone, reached the highest at the 4h of preincubation,At the 8h、24h of preincubation the capacity of engulfment evidently reduced. 2 Glucocorticoid cytosolic receptors antagonist RU38486 inhibited the improved engulfment ability by dexamethasone. 3Phagocytosing apoptotic eosinophils did not change the release of IL-6 and IL-8,but can inhibited the release of IL-6 and IL-8 from A549 cells stimulated by lipopolysaccharide. 4Increasely uptaking by dexamethasone also did not agitate the release of IL-6 and IL-8 from A549 cells. Conclusion: 1Dexamethasome improve the phagocyte capacity for apoptotic eosinophils of A549 cells by glucocorticoid cytosolic receptors, and the effect presents time-dependently. 2Phagocytosis of apoptotic eosinphils can degrade inflammatory reflection, so it is likely to be critical in the control of asthma inflammation.

结果： 1 A549细胞在地塞米松诱导后1小时吞噬能力开始升高，4小时吞噬能力达最高峰，8小时、24小时吞噬能力明显下降 2加入RU38486后抑制了地塞米松所增强的吞噬能力 3凋亡嗜酸粒细胞的吞噬并不能改变A549细胞释放IL-6和IL-8，但能抑制脂多糖刺激A549细胞后IL-6、IL-8的分泌 4地塞米松增加的吞噬也不激发A549细胞释放IL-6和IL-8 结论： 1地塞米松促进A549细胞吞噬凋亡的嗜酸粒细胞是通过A549细胞糖皮质激素胞浆受体发挥作用的，并且具有时间依赖性 2 A549细胞吞噬凋亡的嗜酸粒细胞能降低炎症反应，对哮喘炎症的控制具有重要意义

The experiment indicated that 1-MCP had significantly reduced Gala apple fruit respiration rate,delayed the climacteric,and inhibited fruit softening.1-MCP treatment significantly decreased starch degradation,inhibited the catabolism of fruit cell wall,delaying pectin transformation from insoluble to soluble.

结果表明，常温下，1-MCP可极显著地抑制嘎拉苹果果实的呼吸强度，推迟果实呼吸跃变期，而且很好地保持了果实原有硬度；对果实相关成分的分析表明，1-MCP显著地抑制了淀粉转化、降低细胞壁物质的降解速率，延缓了不溶性果胶向水溶性果胶转化，使其极显著地低于对照。

The in vitro effects of Gln49-PLA_2 were examined using the chick biventer cervicis nerve muscle preparation. Gln49-PLA_2 caused a dosedependent blockade of neuromuscular transmission. The time required to cause 50% blockade was about 150 min at the highest concentration 100μg/ml. This inhibition appeared to be presynaptic in origin as evidenced by the lack of effect of toxin on responses to exogenous acetylcholine in the non-stimulated BCp.

Gln49-PLA_2能够剂量依赖性地抑制颈二腹肌神经信号正常传导并影响肌肉收缩，在100μg/ml剂量下，肌肉收缩幅度在150分钟后达到50%的抑制率；同时，随着Gln49-PLA_2浓度的降低，肌肉收缩幅度的抑制效率也逐渐降低；实验证明Gln49-PLA_2对突触后膜乙酰胆碱受体敏感性无影响，对神经肌肉信号传导收缩的抑制作用位点在突触前膜。

These changes were prevented by genistein (a protein tyrosine kinase inhibitor) and antioxidant N-acetyl-L-cysteine, but promoted by sodium orthovanadate (a protein phosphatase inhibitor), which were administered to the SD rats 20 min before ischemia.

进一步的研究表明，缺血前20 min腹腔注射给药，然后缺血30 min，发现蛋白酪氨酸激酶抑制剂染料木黄酮和抗氧化剂N-乙酰半胱氨酸能显著地抑制核内STAT3的磷酸化水平及DNA结合活性的增加(磷酸化水平从2.3和2.5倍分别降为1.2和1.4倍， DNA结合活性则从2.8和3.7倍分别降为1.1和1.5倍)，而蛋白酪氨酸磷酸酶抑制剂矾酸钠则能明显地促进他们的增高(磷酸化水平从2.0倍增到3.4倍， DNA结合活性从3.1倍增为5.1倍)。

The colon temperatures were measured by a thermistor and the cytosolic free calcium concentration ([Ca2+]i) in dissociated brain cells was measured by Fura 2-AM. The results demonstrated that the pyretic action of egtazic acid (0.6 μmol, icv) was markedly inhibited by DEX (5 mg/kg,iv), but DEX (60-120 μmol/L) did′t affect [Ca2+]i in dissociated hypothalamus cells. Actinomycin D, which interferes with gene transcription (3 nmol, icv), completely abolished the antipyretic action of DEX on egtazic acid-induced fever.

结果发现：脑室灌注乙二醇双(2-氨基乙醚)四乙酸(0.6 nmol)引起家兔结肠温度明显升高，静脉注射地塞米松(5 mg/kg)显著抑制家兔乙二醇双(2-氨基乙醚)四乙酸性发热，地塞米松(60-120 μmol/L)并不影响下丘脑细胞内[Ca2+]i，而事先脑室灌注抑制基因转录的放线菌素D(3 nmol)则完全取消了地塞米松对乙二醇双(2-氨基乙醚)四乙酸性发热的解热作用。

But it in 10"8mol/L-10"mol/L inhibited gastrin secretion .whereas it promoted somatostatin secretion of duodenum in vitro. GnRH-A did not effect significantly gastrin and somatostatin secretion of stomach in vitro.These results suggested that GnRH-A may integrate with GnRH receptor and restrain directely gastric acid secretion in parietal cell.

综上所述，我们认为胃腔内的Gn尺H类似物可能首先和壁细胞上GnRE受体结合，直接抑制壁细胞胃酸的分泌，由于胃酸分泌减少反馈性地刺激胃泌素细胞，促进其合成及分泌胃泌素，胃酸分泌减少同时也反馈性的抑制生长抑素的释放，反馈性地抑制胃肠壁内EC细胞分泌5一HT。

The depressing ability of the macromolecule organic depressants can be improved by the increase of their D.S.. The high D.S. CPVA and CPEG show strong depressing ability to calcite, while CPEG indicates strong depressing ability to diaspore, and HPAM to pyrite.The depressing mechanism of organic depressants was confirmed by the measurements of zeta potential, contact angles, adsorption capacity and IR spectrum.

利用动电位、接触角、吸附量和红外光谱等测试方法和手段，探讨了抑制剂的作用机理：苯氧乙酸类小分子抑制剂与方解石之间可发生较强的化学作用，可以选择性地吸附在方解石表面，使捕收剂发生解吸或阻止捕收剂在矿物表面的吸附而使矿物受到抑制；大分子抑制剂则主要是依靠亲固基吸附在矿物表面，再利用其长链的强亲水性而使矿物受到抑制。

And Pharaoh spoke to Joseph, saying, Your father and your brothers have come to you.

47：5 法老对约瑟说，你父亲和你弟兄们到你这里来了。

Additionally, the approximate flattening of surface strip using lines linking midpoints on perpendicular lines between geodesic curves and the unconditional extreme value method are discussed.

提出了用测地线方程、曲面上两点间短程线来计算膜结构曲面测地线的方法，同时，采用测地线间垂线的中点连线和用无约束极值法进行空间条状曲面近似展开的分析。