抑制...

The pathological findings showed that ZBT could alliviate the synovial tissues inflammatory congestion, edema, remit the inflammatory cell infiltration and inhibit the proliferation of synovial cells, therefore reduce the degree of destruction of the articular cartilage and subcartilaginous bone tissue. Inhibited the disablement of the woulded limbs.

结果：止痹痛药酒能轻模型鼠关节肿胀度，对免疫注射后继发病变具有良好的治疗作用，说明该药具有抗炎免疫作用，病理学观察证实该药能减轻关节滑膜的充血水肿和炎细胞浸润、抑制滑膜细胞增生，减轻关节软骨和软骨下骨组织的破坏程度，抑制模型鼠患肢的致残。

Only small numbers of shrunken cells were observed in Cyclin B1 over-expressing cells, while markedly morphological changes were observed in the control subline.

Cyclin B1蛋白高表达抑制细胞凋亡的可能机制是，正常表达的Cyclin B1蛋白存在于细胞质中，促进细胞增殖、抑制细胞凋亡；药物诱导的Cyclin B1蛋白存在于细胞核中，促进细胞凋亡。

It has been also demonstrated that lots of some chemical compounds not only have the utility of cancer therapy, but also can reduce the side effects of currently used drugs. Thus, to search for a succedaneum much more effective and easily absorbed by human-body seems to be an appropriate approach to substitute the presently used clinical remedy against cancer.

而在相关研究当中显示，许多抑制血管增生的化合物不仅有抑制肿瘤生长的作用且相对有著较低的副作用，因此寻找有效且容易被人体吸收的化合物用以取代目前临床对於癌症的治疗药物，似乎是比较妥当的方式。

Some new sulfonamide derivatives possessing potent CA inhibitory properties act as effective in vitro tumor cell growth and metastasis inhibitors.

一些新型的磺胺类碳酸酐酶抑制剂可有效抑制体外多种肿瘤细胞系的生长和转移。

Among the pesticides detected, sulfureous organophosphates had lower inhibitory ability, but phosphorothionate pesticides could be detected after direct oxidation with N-bromosuccinimide.

针对抑制能力差的硫代型有机磷农药，应用NBS进行氧化前处理，提高酶抑制的灵敏度。

Intracellular pH change did not influence the actions of ketamine while superfusion of NMDA .

在pH 6.81的人工脑脊液中，ketamine抑制放电频率增快的药效减弱了35 %，在pH 8.1的人工脑脊液中，ketamine抑制去极化的药效减少了29 %，而胞内酸化或碱化则不影响ketamine拮抗NMDA的药效。

Ethanol inhibition was not affected by 5 nM and 20 nM calyculin A (a phosphatase 1/2A inhibitor). Prior superfusion of 5 nM and 20 nM KT5720 also attenuated ethanol inhibition of NMDA-mediated depolarization. 1 μM and 3 μM chelerythrine, a selective protein kinase C inhibitor or 20 nM calyculin A did not affect ethanol inhibition of NMDA-induced response.

当以表面灌流方式，事先给予5 nM或20 nM KT5720时，同样可以减少乙醇抑制NMDA引发的去极化作用；分别给予1 μM或3 μM chelerythrine或20 nM calyculin A则不会影响乙醇抑制NMDA所引发的反应。

The inward current in OHCs was totally inhibited after the superfusion of 100μmolIL and 200μmo1/L linopirdine respectively. The outward rectifier K(superscript +) current I(subscript K was the dominant K(superscript +) current in the whole cell currents in Deiters' cells. In the presence of 200μmol/L linopirdine, the current was not significantly affected.

在细胞外液中，加入100μmol/L利诺吡啶后，OHCs中的四乙基二乙胺敏感的钾电流峰电流成分被抑制，稳态电流幅值减小，且电流的失活时间常数明显延长；在细胞外液中加入100μmol/L利诺吡吮后，OHCs的内向性钾电流I被完全抑制；而细胞外液中利诺吡啶终浓度为200μmol/L时，Deiters 细胞的外向整流性钾电流幅值无明显变化。

Moreover, RT-PCR results revealed that the expression of AtSPX3 in phrl was reduced, suggesting PHR1 may function at the upstream of AtSPX3. Transgenic lines with RNAi of AtSPX3 showed supersensitive to Pi-starvation compared with Col.

另外，缺磷条件下AtSPX3基因启动子驱动的GUS活性在phr1突变体中受到抑制，定量RT-PCR的结果也显示，缺磷条件下该基因在phr1突变体中的表达受到抑制，说明在磷信号的传递途径中，PHR1在AtSPX3的上游。

Therefore, NAG7 gene was a nice candidate of tumor suppresser gene associated with NPC. The mechanism might be that the re-expression of NAG7 gene contributed to the change of gene expression profile and following the change of protein expression profile, then involved in the cell cycle and apoptotic regulation changed, and finally suppressed the NPC growth.

因此，NAG7基因作为鼻咽癌相关的抑瘤基因候选者，其作用机理可能是通过诱发基因表达谱的改变而引起蛋白质表达谱发生相应的改变，在一致性改变的功能分子的作用下，抑制细胞增殖，同时诱发细胞凋亡增加，从而发挥抑制鼻咽癌细胞生长的作用。

The labia have now been sutured together almost completely.The drains and the Foley catheter come out at the top.

此刻阴唇已经几乎完全的缝在一起了，排除多余淤血体液的管子和Foley导管从顶端冒出来。

To get the business done, I suggest we split the difference in price.

为了做成这笔生意，我建议我们在价格上大家各让一半。