心肌的

At the same time, the contents of myocardial Ca2+ and MDA were decreased significantly as well as the activities of myocardial SOD and GSH-Px increased markedly. Conclusion: CASI is of protective effect on myocardial ischemia-reperfusion arrhythmia, which may be related to scavenging the oxygen free radicals and Ca2+ overload formed during reperfusion.

CASI对大鼠心肌缺血再灌注心律失常具有明显保护作用，其机制可能与提高心肌组织的抗氧化酶活性，减少自由基及Ca2+超负荷对心肌的氧化损伤有关。

Objective: Through establishing the acute myocardial ischemia rat model, we discuss the situation of myocardial apoptosis and expression of apoptosis- associated factors during the later period of acute myocardial ischemia, as well as Cornu Cervi s intervention function and the mechanism of its inhibitory effects, further to analyze the protective effect of Cornu Cervi on the myocardial ischemia.

目的：通过建立大鼠急性心肌缺血动物模型，探讨在急性心肌缺血损伤后期心肌细胞凋亡情况及凋亡相关因素的变化，以及鹿茸的干预作用及其可能机制，进一步分析鹿茸对损伤心肌的保护作用。

Objective To study the effects of transplanted allogenic cardiomyocytes on the repair and function of infarcted myocardium of rat.

目的 观察同种异体心肌细胞移植对心肌梗死大鼠心肌的修复作用和心功能的影响。

Transmembrane action potentials were recorded simultaneously from endocardium, epicardium, together with a transmural ECG, in arterially perfused left ventricular preparations by use of 2 separate intracellular floating microelectrodes. The stimulus-response- interval of the epicardium and the incidence of ventricular tachycardia were observed as well.

灌流全程同时采用浮置玻璃微电极法同步记录内膜下心肌、外膜下心肌跨膜动作电位和跨壁ECG，并观察心外膜下心肌的刺激反应间期(Stimulus-response-interval， SRI)和室性心动过速的诱发率。

Objective: To observe the effect of the herbicide butachlor on myocardial contracts dint and myocardial enzymogram of Bufo bufo gargarizans, and to study physiological toxicity of herbicide butachlor on myocardial of Bufo bufo gargarizans.

目的：观察除草剂丁草胺对蟾蜍心肌收缩力和心肌酶谱的影响，探讨除草剂丁草胺对蟾蜍心肌的生理毒性。

In recent animal studies intramyocardial transplantation of ESCs or their differentiated cardiac-like cells regenerated injured myocardium and improved heart function in infarcted animal models.

新近的研究表明，在心肌梗死动物模型中，心肌内移植胚胎干细胞或由其分化成的心肌样细胞，能导致已损伤心肌的再生，并改善心脏功能。

CaN Aβ gene silencing can reduce myocardial hypertrophy in cultured cells, si1280 (21bp) of CaN Aβ gene is the most effective target site for siRNA. The method of intrapericardial injection of plasmid, microbubbles and erzymes can improve transfection efficiency of non-viral plasmid with satisfying targeted transfection. But the scope of transfected myocytes is still limited. CaN Aβ shRNA expressing plasmid transfection in vivo by pericardial injection results in decreased CaN Aβ protein expression of small part of myocytes, and CaN Aβ mRNA only shows decreased trend. The dosage of non-viral vector and the parameters of ultrasound energy should be optimized in further study.

结论RNAi技术抑制CaN Aβ基因表达可有效减轻Ald诱导的心肌细胞肥大程度；CaN Aβ基因中si1280(21bp)片段为实现RNAi的更有效片段；微泡+酶类心包腔内注射超声导入的方法可有效改善非病毒载体在体心肌的转染效率，同时具有一定的靶向性，但总的转染范围仍然有限；采用这一方法进一步进行&一肾一夹&心肌肥大模型大鼠在体心肌细胞的CaNAβ的RNAi干预，发现心肌肥大大鼠心外膜下局部心肌细胞CaN Aβ蛋白水平降低，CaN AβmRNA水平虽有下降趋势，但无统计学差异，提示质粒的用量及超声导入的参数有待进一步研究使其优化。

Reduce the hoist of leucocytes of sinus coronaries blood of myocardial ischemia and reperfusion injury dogs;2. decrease the leakage of plasma CK, CK-MB and LDH;3. regulate the vaso-active substances such as ET, TXB〓 and PGI〓;4. decreased the inflammatory cell factors such as IL-1, IL-6 and TNF

综上，奥沙恩注射液可降低心肌缺血再灌注损伤模型犬冠状窦血白细胞总数，减少I／R后CK、CK-MB和LDH的释放，调节血管活性物质ET、〓和〓的产生，降低血清炎性细胞因子TNF-α、IL-1和IL-6的含量，抑制PMN的激活及对心肌的浸润，减轻心肌组织的病理改变，以及减轻心肌缺血及坏死程度、降低心肌耗氧量，从而显示出较强的对犬急性心肌缺血再灌注损伤的保护作用。

Measurirg indxes included:(1) Electrocardiograh;(2) Hemodynamics: cardiac output, pump output, aortic pressire, pulmo nary artery pressure, left atrium pressure, right atrium pressure, left ventricular end systolic pressure, left ventricular end diastolic pressure, LV-dp/dt, right ventricul ar end systolic pressure, RV-dp/dt, systolic pressure time index, diastolic pressure time index, endocardial viability ratio;(3) Myocardial metabolism: coronory sinus blood flow, myocardial oxygen consumption, myocardial lactate extractiorn;(4) Ischemic myocardial area;(5) Ultrastructure of myocardium.

测量指标包括：①心电图；②血流动力学：心输出量、泵输出量、主动脉压、肺动脉压、左房压、右房压、左室收缩末压、左室舒张末压、左室压的一阶导数、右室收缩末压、右室压的一阶导数、收缩压力时间指标、舒张压力时间指标、心内膜下心肌供血比率；③心肌代谢；冠状窦血流量、心肌氧耗量、心肌乳酸摄取量；④心肌缺血面积检查及缺血心肌的超微结构观察。

Cardiac hypertrophy is a common complication of clinical cardiovascular diseases, and one of fatal factors of cardiovascular diseases' incidences and mortalities. So it is very beneficial to prevent cardiac hypertrophy for the people.

心肌肥厚是临床上心血管疾病常见的并发症，是心血管病发生率和死亡率增高的重要危险因素，因此，防止心肌肥厚的发生具有重要的意义。L-甲状腺素诱发的大鼠心肌肥厚是研究缺血性肥厚心肌的适宜模型。

Breath, muscle contraction of the buttocks; arch body, as far as possible to hold his head, right leg straight towards the ceiling (peg-leg knee in order to avoid muscle tension).

呼气，收缩臀部肌肉；拱起身体，尽量抬起头来，右腿伸直朝向天花板（膝微屈，以避免肌肉紧张）。

The cost of moving grain food products was unchanged from May, but year over year are up 8%.

粮食产品的运输费用与5月份相比没有变化，但却比去年同期高8％。